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ECM1 encodes a soluble protein that is involved in endochondral bone formation, angiogenesis, and tumor biology. Additionally we are shipping ECM1 Antibodies (83) and ECM1 Proteins (7) and many more products for this protein.
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Lipoid proteinosis is a rare autosomal recessive disorder caused by mutations in ECM1, encoding extracellular matrix protein 1, a glycoprotein expressed in many organs and which has important protein-protein interactions in tissue homeostasis
MMP-2 (show MMP2 ELISA Kits) protein and ECM1 gene are useful preoperative markers for defining malignancy in suspicious thyroid nodules
association between beta-catenin (show CTNNB1 ELISA Kits) and the MUC1 (show MUC1 ELISA Kits) cytoplasmic tail was increased by ECM1
Lipoidproteinosis results from a large homozygous deletion of ECM1 gene in a Chinese family.
High extracellular matrix protein-1 expression is associated with the growth, metastasis and angiogenesis of laryngeal carcinoma
This large cohort revealed extensive phenotypic variability in individuals with the same mutation in ECM1.
Lipoid proteinosis (LP) is a rare autosomal recessive genodermatosis caused by mutations in extracellular matrix protein 1 (ECM1) that involves deposition of basement membrane-like material in the skin and other organs.
ECM1 induced the expression of genes that promote the Warburg effect, such as glucose transporter 1 (GLUT1 (show SLC2A1 ELISA Kits)), lactate dehydrogenase A (LDHA (show LDHA ELISA Kits)), and hypoxia-inducible factor 1 alpha (HIF-1alpha (show HIF1A ELISA Kits)).
Report a global loss of 5hmC identified three new genes (ECM1, ATF5 (show ATF5 ELISA Kits), and EOMES (show EOMES ELISA Kits)) with potential anti-cancer functions that may promote the understanding of the molecular mechanisms of hepatocellular carcinoma development and progression.
High extracellular matrix protein 1 expression is correlated to carcinogenesis and lymphatic metastasis of gastric cancer
HA accumulation primes the vasculature for atherosclerosis by crosslinking and reorganizing the extracellular matrix (ECM (show MMRN1 ELISA Kits)) and by pushing VSMC differentiation towards a less mature phenotype.
Retinoid signaling in the stroma activates expression of Ecm1, which in turn down-regulates Ret (show RET ELISA Kits) expression in the ureteric bud cleft, where bifurcation normally occurs and normal branching progresses.
This gene encodes a soluble protein that is involved in endochondral bone formation, angiogenesis, and tumor biology. It also interacts with a variety of extracellular and structural proteins, contributing to the maintenance of skin integrity and homeostasis. Mutations in this gene are associated with lipoid proteinosis disorder (also known as hyalinosis cutis et mucosae or Urbach-Wiethe disease) that is characterized by generalized thickening of skin, mucosae and certain viscera. Alternatively spliced transcript variants encoding distinct isoforms have been described for this gene.
extracellular matrix protein 1
, secretory component p85