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FOXD3 belongs to the forkhead family of transcription factors which is characterized by a distinct forkhead domain. Additionally we are shipping FOXD3 Kits (23) and FOXD3 Proteins (4) and many more products for this protein.
Showing 10 out of 74 products:
Human Monoclonal FOXD3 Primary Antibody for IHC, ELISA - ABIN969153
Gregory, Barlow, McLay, Kaul, Swarbreck, Dunham, Scott, Howe, Woodfine, Spencer, Jones, Gillson, Searle, Zhou, Kokocinski, McDonald, Evans, Phillips, Atkinson, Cooper, Jones, Hall, Andrews, Lloyd et al.: The DNA sequence and biological annotation of human chromosome 1. ... in Nature 2006
Show all 2 references for ABIN969153
Human Monoclonal FOXD3 Primary Antibody for EIA, WB - ABIN1107262
Saleem, Banerjee-Basu, Berry, Baxevanis, Walter: Analyses of the effects that disease-causing missense mutations have on the structure and function of the winged-helix protein FOXC1. in American journal of human genetics 2001
Cow (Bovine) Polyclonal FOXD3 Primary Antibody for WB - ABIN2779723
Hromas, Moore, Johnston, Socha, Klemsz: Drosophila forkhead homologues are expressed in a lineage-restricted manner in human hematopoietic cells. in Blood 1993
Foxd3 rescues the prdm1a (show PRDM1 Antibodies) loss-of-function neural crest phenotype.
These results reveal dynamic and differential regulation of FoxD3 in distinct neural crest subpopulations, suggesting that heterogeneity is encrypted at the regulatory level
analysis of how a FoxD3 gene trap line reveals neural crest precursor movement and a role for FoxD3 in their specification
tfap2a (show TFAP2A Antibodies) and foxd3 are expressed during gastrulation prior to neural crest induction in distinct, complementary, domains; tfap2a (show TFAP2A Antibodies) is expressed in the ventral non-neural ectoderm and foxd3 in the dorsal mesendoderm and ectoderm
analysis of a Foxd3/mitfa (show MITF Antibodies) transcriptional switch that governs whether a bi-potent pigment precursor will attain either an iridophore or a melanophore fate
foxd3 is an essential Nodal-dependent regulator of zebrafish mesoderm development.
Decrement of function of foxd3 and/or sox10, two genes important for the development and specification of neural crest, resulted in a reduction and/or loss of GnRH cells of the midbrain, as well as a reduction in the number of terminal nerve GnRH cells.
zebrafish Foxd3 is necessary for the differentiation of a subset of neural crest cell fates, perhaps in precursors of particular neural crest lineages.
Foxd3, a well-known regulator in neural crest development, is also involved in myf5 (show MYF5 Antibodies) regulation
Foxd3 selectively specifies premigratory neural crest cells for a neuronal, glial or cartilage fate by inducing the expression of lineage-associated transcription factors in these cells and regulating their subsequent migration.
FOXD3 might serve as an independent prognostic biomarker and a potential therapeutic target for high-grade gliomas, which warrant further investigation.
Down-regulation of FOXD3 is associated with metastasis in hepatocellular carcinoma.
Data show that transcription factors PAX3 (show PAX3 Antibodies) and FOXD3-mediated melanoma cell migration is dependent on promoting the expression of chemokine receptor CXCR4 (show CXCR4 Antibodies).
In addition to a possible association of rs78645479 in FoxD3 with vitiligo (show MITF Antibodies), our data on the association of this FoxD3 variant with thyroid autoantibodies suggest a potential involvement of FoxD3 in thyroid immunoregulation.
FOXD3 overexpression significantly inhibits cell growth and results in G1 cell cycle arrest in NSCLC A549 and H1299 cells.
FOXD3 and TWIST1 (show TWIST1 Antibodies) define distinct subgroups of cells within a heterogeneous tumor.
Findings indicate that miR (show MLXIP Antibodies)-137 is a valuable biomarker for hepatocellular carcinoma (HCC (show FAM126A Antibodies)) prognosis and the forkhead box D3 (FoxD3)/miR (show MLXIP Antibodies)-137/AKT2 (show AKT2 Antibodies) regulatory network plays an important role in HCC (show FAM126A Antibodies) progression.
Results suggest an inverse relationship between FoxD3 expression and tumor metastasis in invasive ductal carcinomas of the breast.
Results indicate that FOXD3 exhibits tumor suppressive activity that affects the growth, aggressiveness and angiogenesis of NB through transcriptional regulation of NDRG1.
Our results indicate FOXD3 exhibits tumor suppressive activity and may be useful for breast therapy.
Foxd3 poises enhancers in pluripotent stem cells by recruiting multiple epigenetic enzymes that together simultaneously initiate and repress enhancer activity.
Foxd3 suppresses NFAT (show NFATC1 Antibodies)-mediated differentiation to maintain self-renewal of embryonic stem cells
In a xenograft tumor model, FOXD3 overexpression inhibits tumor growth and angiogenesis.
Data indicate that homeobox b5 (Hoxb5 (show HOXB5 Antibodies)) regulated the neural crest (NC)development by directly inducing Forkhead box D3 gene (Foxd3).
Foxd3 induced mutant ESCs (show NR2E3 Antibodies) precociously express genes required for mesoderm induction, but they are likely unable to differentiate into skeletal muscle.
Data indicate that growth factor receptor (show RYK Antibodies) protein binding protein 2 (Grb2 (show GRB2 Antibodies)) is upregulated and regulated by Forkhead Box D3 (Foxd3), and pregulated Grb2 (show GRB2 Antibodies) interacts with huntingtin (Htt (show HTT Antibodies)).
Foxd3 deficiency completely abolishes the sphere-forming potential of skin embryonic stem cells.
Foxd3 is part of a dynamically expressed gene network that is necessary and sufficient to regulate fate decisions in premigratory neural crest cells.
Methylation profile analyses identified the promoter of FOXD3 as the only genomic region with increased methylation in mice and humans during progression of Helicobacter pylori-associated gastric tumors.
The findings highlighted the crucial roles played by Foxd3 during enteric nervous system development including progenitor proliferation, neural patterning, and glial differentiation.
This gene belongs to the forkhead family of transcription factors which is characterized by a distinct forkhead domain. Mutations in this gene cause autoimmune susceptibility 1.
fork head domain protein 6
, forkhead box protein D3
, mother superior
, forkhead box D3
, HNF3/FH transcription factor genesis
, HNF-3/forkhead homolog 2
, hepatocyte nuclear factor 3 forkhead homolog 2
, winged helix protein CWH-3
, winged-helix protein CWH-3