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Gap Junction Protein, beta 2, 26kDa (GJB2) ELISA Kits

GJB2 encodes a member of the gap junction protein family. Additionally we are shipping GJB2 Antibodies (89) and GJB2 Proteins (4) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
GJB2 14619 Q00977
GJB2 394266 P21994
GJB2 2706 P29033
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Top GJB2 ELISA Kits at antibodies-online.com

Showing 10 out of 36 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Supplier Delivery Price Details
Human 0.66 ng/mL 1.56-100 ng/mL 96 Tests Log in to see 9 to 11 Days
$700.00
Details
Rat 9.38 pg/mL 15.63-1000 pg/mL Diagramm of the ELISA kit to detect Rat CX26with the optical density on the x-axis and the concentration on the y-axis. 96 Tests Log in to see 8 to 9 Days
$680.63
Details
Mouse 11.72 pg/mL 19.53-1250 pg/mL Diagramm of the ELISA kit to detect Mouse CX26with the optical density on the x-axis and the concentration on the y-axis. 96 Tests Log in to see 8 to 9 Days
$680.63
Details
Pig 0.1 ng/mL 5-100 ng/mL   96 Tests Log in to see 11 to 13 Days
$801.43
Details
Human 0.1 ng/mL   96 Tests Log in to see 11 to 13 Days
$785.71
Details
Monkey
  96 Tests Log in to see 8 to 9 Days
$770.00
Details
Pig
  96 Tests Log in to see 8 to 9 Days
$770.00
Details
Rabbit
  96 Tests Log in to see 8 to 9 Days
$770.00
Details
Chicken
  96 Tests Log in to see 8 to 9 Days
$770.00
Details
Guinea Pig
  96 Tests Log in to see 11 to 13 Days
$801.43
Details

More ELISA Kits for GJB2 Interaction Partners

Xenopus laevis Gap Junction Protein, beta 2, 26kDa (GJB2) interaction partners

  1. Using reconstituted hemichannels in a liposome-based transport-specific fractionation assay, we confirmed that homomeric Cx26 and Cx32 (show GJB1 ELISA Kits) and heteromeric Cx26/Cx32 (show GJB1 ELISA Kits) are permeable to GSH and other endogenous reductants.

Mouse (Murine) Gap Junction Protein, beta 2, 26kDa (GJB2) interaction partners

  1. mir (show MLXIP ELISA Kits)-27a was identified as an apoptotic molecule that participates in Cx26 knockout-induced apoptosis in the cochlear sensory epithelium of mice by downregulating sgk1 (show SGK1 ELISA Kits) expression

  2. Cx26 knockout predisposes the mammary gland to primary mammary tumors in a DMBA-induced mouse model of breast cancer.

  3. Cx26-mediated intercellular communication is required for cochlear development and that deficiency of Cx26 can impair miRNA-mediated intercellular genetic communication in the cochlea, which may lead to cochlear developmental disorders

  4. presence of Cx30 (show GJB6 ELISA Kits) in the cochlea does not compensate for Cx26 loss, and the absence of both connexins from vestibular sensory epithelia is no more injurious than the absence of one of them

  5. Reciprocal positive regulation between Cx26 and PI3K/Akt (show AKT1 ELISA Kits) pathway confers acquired gefitinib resistance in non-small-cell lung carcinoma cells by promoting epithelial mesenchymal transition via a gap-junctional communication-independent manner.

  6. The inserting reconstituted gap junction Cx26 liposomes into the oocytes allowed the demonstration of intracellular/extracellular Ca(2 (show CA2 ELISA Kits)+)-regulated hemi-channel activities.

  7. Study used an inducible gene knockout technique to delete Cx26 expression in the cochlea after birth, results suggest that Cx26 deficiency may impair active cochlear amplification leading to late-onset hearing loss

  8. These results suggest that low levels of Cx26 expression throughout pregnancy and lactation, and not the physiological surge in Cx26, is sufficient for normal gland development and function.

  9. These results suggest that successful treatment of Cx26 deafness requires early intervention before the cochlea fully matures.

  10. Over-expression of BDNF (show BDNF ELISA Kits) beginning around 1 month of age resulted in a significant rescue of spiral ganglion degeneration in connexin 26 knockout mice.

Human Gap Junction Protein, beta 2, 26kDa (GJB2) interaction partners

  1. Connexin26 I30N and D50Y mutations resulted in the retention of Cx26 protein in the Golgi apparatus.

  2. The results show a very high proportion of GJB2-positive individuals in Lithuanian patients affected with sensorineural hearing loss.

  3. Carrier frequency of the GJB2 mutations that cause hereditary hearing loss in the Japanese population.

  4. Cx26 overexpression by transfection of Cx26 plasmid DNA enhanced OXA cytotoxicity.

  5. We have found a 148G-A transition in the GJB2 gene, resulting in an asp50-to-asn (D50N) substitution in a girl with congenital deafness. This finding allowed us to diagnose small a, Cyrillic KID syndrome.

  6. The detection rate of the single GJB2 mutation among the 160 SNHL subjects in the present study (6.25%) was higher than 2.58% in normal hearing controls in Korean. The DFNB1 was clearly excluded as a molecular etiology in four (40%) subjects

  7. We found a novel mutation in the GJB2 gene and two trimutations with SNHL not previously reported.

  8. ER-alpha (show ESR1 ELISA Kits) status of endometrioid adenocarcinomas could restrict eventual proapoptotic or anti-apoptotic impact of aberrantly expressed Cx43 (show GJA1 ELISA Kits) and Cx26 in these tumors.

  9. GJB2 mutation is associated with hereditary deafness in Yakutiya.

  10. GJB2 and SLC26A4 (show SLC26A4 ELISA Kits) mutations are associated with good post-implant outcomes.

Cow (Bovine) Gap Junction Protein, beta 2, 26kDa (GJB2) interaction partners

  1. intermediate invasive status of bovine trophoblast is supported by the fact that trophoblast giant cells coexpress connexins (Cx)26, Cx32 (show GJB1 ELISA Kits), and Cx43 (show GJA1 ELISA Kits)

GJB2 Antigen Profile

Antigen Summary

This gene encodes a member of the gap junction protein family. The gap junctions were first characterized by electron microscopy as regionally specialized structures on plasma membranes of contacting adherent cells. These structures were shown to consist of cell-to-cell channels that facilitate the transfer of ions and small molecules between cells. The gap junction proteins, also known as connexins, purified from fractions of enriched gap junctions from different tissues differ. According to sequence similarities at the nucleotide and amino acid levels, the gap junction proteins are divided into two categories, alpha and beta. Mutations in this gene are responsible for as much as 50% of pre-lingual, recessive deafness.

Gene names and symbols associated with GJB2

  • gap junction protein, beta 2, 26kDa (gjb2) antibody
  • gap junction protein, beta 2, 26kDa (GJB2) antibody
  • gap junction protein, beta 2 (Gjb2) antibody
  • AI325222 antibody
  • Cnx26 antibody
  • connexin-26 antibody
  • CX26 antibody
  • CXN-26 antibody
  • DFNA3 antibody
  • DFNA3A antibody
  • DFNB1 antibody
  • DFNB1A antibody
  • Gjb-2 antibody
  • GJB2 antibody
  • HID antibody
  • KID antibody
  • MGC53062 antibody
  • NSRD1 antibody
  • PPK antibody

Protein level used designations for GJB2

gap junction protein, beta 2, 26kDa , connexin 26 , connexin 29 , gap junction membrane channel protein beta 6 , gap junction protein, beta 2, 26kDa (connexin 26) , connexin-26 , gap junction beta-2 protein , gap junction membrane channel protein beta 2 , gap junction channel protein connexin 26 , connexin26 , gap junction protein beta 2 , connexin 26 protein , cx26

GENE ID SPECIES
380170 Xenopus laevis
467220 Pan troglodytes
14619 Mus musculus
394266 Rattus norvegicus
100379528 Cavia porcellus
2706 Homo sapiens
403570 Canis lupus familiaris
407154 Bos taurus
443345 Ovis aries
704224 Macaca mulatta
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