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Glutamate receptors are the predominant excitatory neurotransmitter receptors in the mammalian brain and are activated in a variety of normal neurophysiologic processes.
These results provide direct evidence for cortical AMPA (show GRIA3 ELISA Kits) receptors to contribute to zymosan-induced visceral and spontaneous pain.
Animals trained in the trace fear conditioning protocol had GluA2 RNA editing levels were nearly 100% in amygdala and hippocampus.
These results provide evidence for VPS35 (show vps35 ELISA Kits)'s function in promoting spine maturation, which is likely through increasing AMPA (show GRIA3 ELISA Kits) receptor targeting to the postsynaptic membrane.
Bacopa monnieri extract (CDRI-08) upregulates the expression of the GluR2 subunit in the CA3 (show CA3 ELISA Kits) area of the hippocampus.
subcellular redistribution of GRIP1 (show NCOA2 ELISA Kits) and a change in the binding of GRIP1 (show NCOA2 ELISA Kits) to GluA2 during synaptic scaling, was observed.
Data indicate that the AMPA (show GRIA3 ELISA Kits) receptor subunits abundance is hippocampus, GluA2 > GluA1 (show GRIA1 ELISA Kits) > GluA3 (show GRIA3 ELISA Kits) >> GluA4 (show GRIA4 ELISA Kits); cortex, GluA2 > GluA3 (show GRIA3 ELISA Kits) >/= GluA1 (show GRIA1 ELISA Kits) >> GluA4 (show GRIA4 ELISA Kits); and cerebellum, GluA2 > GluA3 (show GRIA3 ELISA Kits) >/= GluA1 (show GRIA1 ELISA Kits) > GluA4 (show GRIA4 ELISA Kits).
Loss of long-term potentiation was linked to reduced N-methyl-D-aspartate (NMDA) to alpha-amino-3-hydroxy- 5-methyl-4-isoxazolepropionic acid (AMPA (show GRIA3 ELISA Kits)) current ratio.
Removal of FXR1P (show FXR1 ELISA Kits) from the forebrain of postnatal mice selectively enhances long-term storage of spatial memories, hippocampal late-phase long-term potentiation (L-LTP (show SCP2 ELISA Kits)), and de novo GluA2 synthesis.
The data of this study support a model in which GluA2 is translated in the cell body and subsequently transported to neuronal dendrites and synapses, and synaptic GluA2 concentrations are modified primarily by regulated protein trafficking.
DNA methylation (show HELLS ELISA Kits) of such genes as gria2 mediates persistent epileptiform activity and inter-individual differences in the epileptic response to neuronal insult.
Glutamate receptors are the predominant excitatory neurotransmitter receptors in the mammalian brain and are activated in a variety of normal neurophysiologic processes. This gene product belongs to a family of glutamate receptors that are sensitive to alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA), and function as ligand-activated cation channels. These channels are assembled from 4 related subunits, Gria1-4. The subunit encoded by this gene (Gria2) is subject to RNA editing (CAG->CGG\; Q->R) within the second transmembrane domain, which is thought to render the channel impermeable to Ca(2+). Alternative splicing, resulting in transcript variants encoding different isoforms, (including the flip and flop isoforms that vary in their signal transduction properties), has been noted for this gene.
AMPA-selective glutamate receptor 2
, glutamate receptor 2
, glutamate receptor B
, AMPA glutamate receptor 2
, AMPA receptor GluR2/B
, AMPA selective glutamate receptor
, glutamate receptor ionotropic, AMPA 2