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Growth arrest-specific 1 plays a role in growth suppression. Additionally we are shipping GAS1 Antibodies (55) and GAS1 Proteins (2) and many more products for this protein.
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Studies establish Gas1 as a negative regulator in colorectal cancer.
These results demonstrated that miR (show MLXIP ELISA Kits)-184 and the GAS1/Akt (show AKT1 ELISA Kits) pathway may be a potential therapeutic target for intervertebral disc degeneration.
WT1 (show WT1 ELISA Kits) modulates Akt (show AKT1 ELISA Kits)/JNK (show MAPK8 ELISA Kits) signaling pathway mediated autophagy by controlling the expression of growth arrest-specific 1 (Gas1).
Downregulation of GAS1 is associated with clear cell renal cell carcinoma.
the synergistic effect of Gas1 inhibition and GDNF against glutamate (show GRIN1 ELISA Kits)-induced cell injury in human SH-SY5Y neuroblastoma (show ARHGEF16 ELISA Kits) cells, is reported.
solutions structure and biophysical characterization of the multifaceted signalling effector protein growth arrest specific-1
This review summarizes the structure of Gas1 and molecular mechanism of action during embryonic development and cellular functions.
GAS1 is a novel endogenous inhibitor of glomerular mesangial cell proliferation.
Growth arrest-specific gene 1 is downregulated and inhibits tumor growth in gastric cancer.
GAS1 induced the activity of caspase-3 (show CASP3 ELISA Kits) and resulting in apoptosis of the cells.
Up-regulation of inducible Gas1 contributed to the apoptosis of reactive astrocytes in injured substantia nigra.
The C/EBP (show CEBPA ELISA Kits) target Gas1 is induced in granulosa cells and is associated with ovulation and luteinization.
the overexpression of Gas1 (growth arrest specific 1) gene reduces the size, proliferating activity and malignancy of liver tumors.
WT1 (show WT1 ELISA Kits) modulates receptor tyrosine kinase (show ERBB3 ELISA Kits) signaling in nephron progenitor cell by directing the expression of Gas1.
When Shh (show SHH ELISA Kits) is presented as a point source, enteric axons turn away from it in a Gas1-dependent manner.
widespread distribution and cellular localization of the GAS1 native protein in adult mammalian CNS
finding that Gas1 is transcribed at the G(1)/S transition suggests that the gene may exert a novel function during cell proliferation.
Gas1 promotes myogenic differentiation through regulation of cell cycle arrest and is critical to activate p38MAPK (show MAPK14 ELISA Kits)
Genetic loss-of-function of GAS1, CDO (show CDO1 ELISA Kits), and BOC (show BOC ELISA Kits) in SHH (show SHH ELISA Kits) is associated with defective neural patterning.
Gas1 acts to maintain high levels of FGF10 (show FGF10 ELISA Kits) at the tip mesenchyme and support the proposal that Fgf10 (show FGF10 ELISA Kits) expression in this region is crucial for maintaining Fgf8 (show FGF8 ELISA Kits) expression in the AER
Growth arrest-specific 1 plays a role in growth suppression. GAS1 blocks entry to S phase and prevents cycling of normal and transformed cells. Gas1 is a putative tumor suppressor gene.
, Growth arrest-specific gene-1
, growth arrest-specific protein 1
, growth arrest-specific 1
, growth arrest-specific protein 1-like