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mouse homolog is a transcription factor that is an important regulator of the glucose transporter GLUT4. Additionally we are shipping and and many more products for this protein.
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KLF15 activates SOX9 (show SOX9 ELISA Kits) expression directly. SOX9 (show SOX9 ELISA Kits) is involved in KLF15 function during chondrogenic differentiation.
study identifies novel genes associated with insulin (show INS ELISA Kits) sensitivity in adipocytes in women independently of obesity. KFL15 and SLC25A10 (show SLC25A10 ELISA Kits) are inhibitors of insulin (show INS ELISA Kits)-stimulated lipogenesis under conditions when glucose transport is the rate limiting step
In general, our study revealed that KLF15 is dramatically down-regulated in GC tissues and cell lines and that KLF15 is negatively associated with aggressive clinical characteristics, such as tumor stage, lymph node metastasis, and DFS (show FST ELISA Kits). Importantly, up-regulation of KLF15 inhibits cell proliferation by regulating CDKN1A/p21 (show CDKN1A ELISA Kits) and CDKN1C/p57 (show CDKN1C ELISA Kits).
KLF15 is directly induced by glucocorticoids in primary human airway smooth muscle and it represses ASM (show SMPD1 ELISA Kits) hypertrophy.
the expression of KLF15 increased in the RA-treated cells regardless of fluidic condition
In breast cancer cells, KLF6 (show KLF6 ELISA Kits) and KLF15 are suppressed via miR (show MLXIP ELISA Kits)-4262.
Findings suggest that, in obese status, the lower expression level of A2bAR (show ADORA2B ELISA Kits), KLF4 (show KLF4 ELISA Kits), and KLF15 of visceral adipose tissue may correlate with obese-dyslipidemia induced inflammation in Uygur population.
Case Reports: 2 girls with primary renal myoepithelial carcinomas with a novel EWSR1 (show EWSR1 ELISA Kits)-KLF15 fusion.
Our results indicate that nuclear KLF15 expression suppresses breast cancer cell proliferation at least partially through p21 (show CDKN1A ELISA Kits) up-regulation and subsequent cell cycle arrest
GR and KLF15 physically interact via low affinity GR binding sites within glucocorticoid response elements (GREs) for PRODH (show PRODH ELISA Kits) and AASS (show AASS ELISA Kits) that contribute to combinatorial regulation with KLF15.
Functional KLF15 significantly enhanced the promoter activity of fad24.
The expression of Klf15 is increased during neuropathic pain. TNF-alpha (show TNF ELISA Kits) regulates the expression of Klf15. KLF15 regulates the expression of dopamine D2 receptor (show DRD2 ELISA Kits). KLF15 regulates neuropathic pain in mouse models.
Taken together, our findings imply that KLF15 possesses potential anti-hypertrophic and anti-fibrotic functions, possibly via regulation of cell death pathways and the inhibition of Akt (show AKT1 ELISA Kits)/mTOR (show FRAP1 ELISA Kits) axis. KLF15 may constitute an efficient candidate drug for the treatment of heart failure and other cardiovascular diseases.
Kruppel-like factor 15 (KLF15) governs a biphasic transcriptomic oscillation in the heart with a maximum ATP production phase and a remodeling and repair phase corresponding to the active and resting phase of a rodent.
deletion of Klf15 interferes with nuclear control of mitochondrial fission, whereas fusion appears to be unaffected.
Our results demonstrate that c-Jun (show JUN ELISA Kits) can suppress adipocyte differentiation through the down-regulation of KLF15 at the transcriptional level.
KLF15 regulates bile acid synthesis via negative regulation of circadian Fgf15 expression.
Adipolin is transcriptionally regulated by KLF15 in adipocytes.
KLF15 upregulated slow myosin heavy chain expression through NFATc1 (show NFATC1 ELISA Kits).
This study establishes KLF15 as an important molecular link between ER stress and insulin (show INS ELISA Kits) action.
KLF15 as a key regulator of myocardial lipid utilization and is the first to implicate the KLF transcription factor family in cardiac metabolism.
mouse homolog is a transcription factor that is an important regulator of the glucose transporter GLUT4
Krueppel-like factor 15
, kidney-enriched Kruppel-like factor
, kidney-enriched krueppel-like factor
, cardiovascular Krueppel-like factor