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The protein encoded by KLF7 is a member of the Kruppel-like transcriptional regulator family. Additionally we are shipping KLF7 Antibodies (55) and KLF7 Kits (1) and many more products for this protein.
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In the spinal cord, klf7- and klf6a (show KLF6 Proteins)-expressing cells are found in both the dorsal and ventral horns. Numerous sensory structures (e.g. auditory, lateral line, olfactory and visual) and several motor nuclei (e.g. oculomotor, trigeminal, and vagal motor nuclei) contain klf7- and/or klf6a (show KLF6 Proteins)-expressing cells
Data revealed that expression of DNA binding inhibitor 3, early B cell factor 2, Ebf3, Iroquois related homeobox 1, Kruppel-like factor 7 , mab-21-like 1 , fatty acid binding protein 7 and stathmin-like 4,were enriched in the diencephalon of zebrafish.
Higher expression of TLR4 (show TLR4 Proteins) and KLF7 may play a vital role in the process of inflammation induced by obesity in visceral adipose tissue
a dominant mutation in FBXO38 (show FBXO38 Proteins) causes adult-onset, progressive spinal muscular atrophy with calf predominance
KLF7 was upregulated by morphine at both the transcriptional and the translational levels, promoting cell growth up to day 4
The gene encoding KLF7 is a novel candidate for conferring genetic susceptibility to type 2 diabetes.
KLF7 may contribute to the pathogenesis of type 2 diabetes through an impairment of insulin (show INS Proteins) biosynthesis and secretion in pancreatic beta-cells and a reduction of insulin (show INS Proteins) sensitivity in peripheral tissues.
We identified a novel association between the minor A-allele of KLF7 and protection against obesity in the Danish population
Data show that adult corticospinal tract (CST (show CORT Proteins)) neurons failed to up-regulate KLF7 in response to axon injury, and overexpression of VP16-KLF7 in vivo promoted both sprouting and regenerative axon growth in the CST (show CORT Proteins) of adult mice.
KLF7 inactivation in Klf7-null mice decreases the expression of the neurogenic marker brain lipid-binding protein/fatty acid-binding protein 7 (show FABP7 Proteins) (BLBP/FABP7 (show FABP7 Proteins)) in neural stem cells (NSCs).
Klf7 gene ablation reduces TrkA (show NTRK1 Proteins) expression in sensory neurons due to effect on cell death
Klf7 is important for neuronal morphogenesis in selected regions of the central nervous system.
Cooperation between Brn3a (show POU4F1 Proteins) and Klf7 is required for endogenous TrkA (show NTRK1 Proteins) gene expression and the survival of nociceptive sensory neurons.
Results advance knowledge of transcriptional regulation of olfactory neurogenesis and KLF7 action.
Upregulation of adiponectin (show ADIPOQ Proteins) protein by (-)-catechin may involve, at least in part, suppression of KLF7 in 3T3-L1 cells.
The protein encoded by this gene is a member of the Kruppel-like transcriptional regulator family. Members in this family regulate cell proliferation, differentiation and survival and contain three C2H2 zinc fingers at the C-terminus that mediate binding to GC-rich sites. This protein may contribute to the progression of type 2 diabetes by inhibiting insulin expression and secretion in pancreatic beta-cells and by deregulating adipocytokine secretion in adipocytes. A pseudogene of this gene is located on the long arm of chromosome 3. Alternative splicing results in multiple transcript variants.
Kruppel-like factor 7 (ubiquitous)
, Krueppel-like factor 7
, ubiquitous Kruppel-like transcription factor