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LIPC encodes hepatic triglyceride lipase, which is expressed in liver. Additionally we are shipping Lipase, Hepatic Kits (48) and Lipase, Hepatic Proteins (16) and many more products for this protein.
Showing 10 out of 99 products:
Human Polyclonal LIPC Primary Antibody for EIA, FACS - ABIN953179
Zhang, Wang, Zhao, Wu: Preparation of mesoporous silica with pelagic clay and its optical property. in Journal of nanoscience and nanotechnology 2010
Show all 3 references for ABIN953179
Human Polyclonal LIPC Primary Antibody for EIA, FACS - ABIN953180
Jablonski, McAteer, de Bakker, Franks, Pollin, Hanson, Saxena, Fowler, Shuldiner, Knowler, Altshuler, Florez: Common variants in 40 genes assessed for diabetes incidence and response to metformin and lifestyle intervention in the diabetes prevention program. in Diabetes 2010
Show all 3 references for ABIN953180
Human Polyclonal LIPC Primary Antibody for IHC, ELISA - ABIN1451283
Stahnke, Sprengel, Augustin, Will: Human hepatic triglyceride lipase: cDNA cloning, amino acid sequence and expression in a cultured cell line. in Differentiation; research in biological diversity 1988
Human Polyclonal LIPC Primary Antibody for FACS, WB - ABIN655196
Hu, Lui, Mak, Chu, Lee, Poon, Tsui, Ko, Baum, Tam, Li, Tomlinson: Pharmacogenetic analysis of lipid responses to rosuvastatin in Chinese patients. in Pharmacogenetics and genomics 2010
We found inverse independent correlations between metabolic clearance rate of insulin (show INS Antibodies) (MCRI) and hepatic lipase (P = 0.0004), insulin (show INS Antibodies) secretion (P = 0.0002), alanine aminotransferase (show ALT Antibodies) (P = 0.0045), total fat mass (P = 0.014), and diabetes (P = 0.03). MCRI and apolipoprotein A-I (show APOA1 Antibodies) exhibited a positive independent correlation (P = 0.035).
Hepatic lipase activity increases after levothyroxine therapy in subclincal hypothyroidism and can be interpreted as a possible explanation for the decrease in remnant-like lipoprotein cholesterol.
The hepatic lipase (LIPC) rs10468017 variant was associated with a significantly decreased risk of AMD (show AMD1 Antibodies)
the association between LIPC C-514T polymorphism, obesity and plasma lipid profile in Chinese children and adolescents, is reported.
results show that SM is a physiological inhibitor of HL activity in lipoproteins and that the specificity of the enzyme towards HDL (show HSD11B1 Antibodies) is at least partly due to its low SM content.
The role of hepatic triglyceride lipase (show LIPG Antibodies) in remnant lipoprotein metabolism.
Dietary fat intake significantly modified the LIPC genetic effect on changes in serum triglyceride, LDL cholesterol, and HDL (show HSD11B1 Antibodies) cholesterol concentrations during a 2-y randomized weight-loss intervention trial.
Hepatic lipase C-514T gene polymorphism is associated with cardiometabolic parameters and cardiovascular risk factors but not with fatty liver
there was no strong evidence to support an association between LIPC and HDCP in Han Chinese women. However, LIPC variants were associated with BMI, SBP (show SHBG Antibodies), and lipid profiles in patients.
The present study suggested that the LIPC-514 C/T polymorphism of the HL gene has no significant association with the risk of endometriosis in the studied Iranian women.
hepatic lipase KO mice had dyslipidemia including hypercholesterolemia, hypertriglyceridemia and increased non-esterified fatty acid levels. Also glucose intolerance, pancreatic and hepatic inflammation and steatosis.
Bone marrow (BM)-derived hepatic lipase mitigates the HDL (show HSD11B1 Antibodies)-lowering, HDL (show HSD11B1 Antibodies)-modulating, and cholesterol-raising effects of BM-derived cholesteryl ester transfer protein (show CETP Antibodies).
HL expressed by osteoblasts has an impact on osteoblast OPG (show TNFSF11 Antibodies) expression and that lack of HL leads to increased bone mass.
Hepatic lipase and endothelial lipase (show LIPG Antibodies) influence molecular species of several classes of plasma lipids.
interaction of LIGHT with LTbetaR on hepatocytes, but not Kupffer cells, is sufficient to down regulate hepatic lipase expression and that this effect can be independent of LIGHT's costimulatory function.
Hepatic lipase- and endothelial lipase (show LIPG Antibodies)-deficiency in mice promotes macrophage-to-feces RCT (show FOXE3 Antibodies) and HDL (show HSD11B1 Antibodies) antioxidant properties.
Data show a novel pathway involving HL hydrolysis of VLDL that activates PPARdelta (show PPARD Antibodies) through generation of specific monounsaturated fatty acids.
These results demonstrated that fatty acid synthase (show FASN Antibodies) and hepatic lipase might be FXR (show NR1H4 Antibodies)-regulated genes in liver cells.
The additive effect of hepatic lipase and endothelial lipase (show LIPG Antibodies) on HDL (show HSD11B1 Antibodies) metabolism but not macrophage reverse cholesterol transport in mice.
Data show that shows the arrangement of an evolutionarily conserved domain within LMF1 (show LMF1 Antibodies) (DUF1222) that is essential to lipase (show LIPG Antibodies) maturation.
LIPC encodes hepatic triglyceride lipase, which is expressed in liver. LIPC has the dual functions of triglyceride hydrolase and ligand/bridging factor for receptor-mediated lipoprotein uptake.
hepatic triacylglycerol lipase
, hepatic triglyceride lipase
, lipase, hepatic
, lipase C
, hepatic lipase
, hepatic triacylglycerol lipase-like
, lipase member C