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MIF encodes a lymphokine involved in cell-mediated immunity, immunoregulation, and inflammation. Additionally we are shipping MIF Kits (70) and MIF Proteins (57) and many more products for this protein.
Showing 10 out of 206 products:
Human Polyclonal MIF Primary Antibody for EIA, WB - ABIN118028
Bernhagen, Calandra, Mitchell, Martin, Tracey, Voelter, Manogue, Cerami, Bucala: MIF is a pituitary-derived cytokine that potentiates lethal endotoxaemia. in Nature 1993
Show all 11 references for ABIN118028
Human Monoclonal MIF Primary Antibody for EIA, IHC (p) - ABIN121160
Weiser, Temple, Witek-Giannotti, Remold, Clark, David: Molecular cloning of a cDNA encoding a human macrophage migration inhibitory factor. in Proceedings of the National Academy of Sciences of the United States of America 1989
Show all 3 references for ABIN121160
Human Polyclonal MIF Primary Antibody for EIA, ELISA - ABIN250498
Hsieh, Su, Wang, Tsai, Huang, Chang, Lai, Lei, Huang: Hepatitis B virus pre-S2 mutant surface antigen induces degradation of cyclin-dependent kinase inhibitor p27Kip1 through c-Jun activation domain-binding protein 1. in Molecular cancer research : MCR 2007
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Human Polyclonal MIF Primary Antibody for ELISA, WB - ABIN185406
Church, Willis, Priest, Lamonte, Earnest, Wilkinson, Wilson, Giroir: Obesity, macrophage migration inhibitory factor, and weight loss. in International journal of obesity (2005) 2005
MIF was found to be essential for axis formation and neural development of Xenopus embryos.
Data show that the mif pathway is required for both sensory hair cell (HC) and sensory neuronal cell survival in the ear, for HC differentiation, semicircular canal formation, statoacoustic ganglion (SAG (show SAG Antibodies)) development, and lateral line HC differentiation.
MIF inhibits the myoblast differentiation by affecting the cell cycle progression, but does not affect proliferation.
this paper shows that the detrimental effect of MIF knockout was associated with accentuated loss in cardiac autophagy with aging
our results suggest that MIF promotes mCSC (show SLC25A25 Antibodies) survival, proliferation and endothelial differentiation through the activation of the PI3K/Akt (show AKT1 Antibodies)/mTOR (show FRAP1 Antibodies) and AMPK (show PRKAA1 Antibodies) signaling pathways.
Posttranslational modification of MIF by S-nitrosation results in intracellular accumulation and protection from myocardial ischemia reperfusion injury.
Data show that the siRNA-induced macrophage migration inhibitory factor (MIF) reduction in murine mammary cancer line 4T1 and human breast cancer line MDA-MB-231 resulted in significant reduction of cell proliferation and increase of apoptosis.
High expression levels of macrophage migration inhibitory factor sustain the innate immune responses of neonates.
The deletion of the MIF gene led to reduced behavioural despair in mice of both sexes and IFN-gamma (show IFNG Antibodies) mRNA levels were reduced in the hippocampus of the MIF KO mice.
In D-galactosamine-sensitized mice CP+Cu(II) increased the LPS (show TLR4 Antibodies)-induced lethality from 54 to 100%, while administration of antibodies against MIF prevented the lethal effect. The enhancement by CP+Cu(II) of the pro-inflammatory signal of MIF is discussed.
data suggest that the MIF-Notch (show NOTCH1 Antibodies) axis may play an important role in the pathogenesis of experimental autoimmune uveitis
The functional role of MIF in cell recruitment was investigated by a chemotaxis assay and by flow cytometry of labeled macrophages that were injected into Mif-/-and wildtype mice
'low' serum Lipoprotein(a), mediating autoimmune activation, is a major determinant of metabolic disease risk and death. Damaged MIF (show AMH Antibodies) protein and more complex autoimmune activation in women may be responsible from lack of relationship to Metbolic syndrome/mortality.
Study revealed that TNF (show TNF Antibodies) gene -308G[A polymorphism is strongly associated with a susceptibility to dysmenorrhea in Turkish population; however, no relation was found for MIF (show AMH Antibodies) gene -173G[C polymorphism.
structural modeling predicts a new MIF (show AMH Antibodies)(CD74 (show CD74 Antibodies))3 dodecamer that may provide the basis for increased MIF (show AMH Antibodies) potency and the requirement for ~3-fold excess RTL1000 to achieve full antagonism
High MIF (show AMH Antibodies) levels and MIF (show AMH Antibodies) -173G/C gene polymorphism are powerful predictors of the severity of sepsis and its outcome.
miR (show MLXIP Antibodies)-608 overexpression inhibits the malignant behavior of glioma stem cells by downregulating MIF (show AMH Antibodies).
macrophage migration inhibitory factor (MIF) was regulated directly by miR451 and was a critical mediator of the biological effects of miR451 in neuroblastoma (show ARHGEF16 Antibodies).
MIF (show AMH Antibodies) gene -173 G/C single nucleotide polymorphisms implicate in genetic susceptibility to TB, and GC+CC of MIF (show AMH Antibodies) -173 site increases the risk of TB. We also found that no correlation between -173 G/C single nucleotide polymorphism and retreatment cases of TB in Yunnan Province population of China.
Significant differences exist in plasma MIF (show AMH Antibodies) levels between obese and non-obese women with and without polycystic ovary syndrome.
Genetic polymorphism of rs755622 in MIF is associated with increased risk of gestational diabetes and insulin resistance in Han Chinese women.
miR (show MLXIP Antibodies)-451 was down-regulated in renal cell carcinoma (show MOK Antibodies) and correlated with histological grade and lymphatic metastasis. MIF (show AMH Antibodies) was identified as a target of miR (show MLXIP Antibodies)-451, and down-regulation of MIF (show AMH Antibodies) could mimic the suppressive functions of miR (show MLXIP Antibodies)-451.
The objective of the present study was to determine if SNPs in 5' region of bovine MIF affects its promoter activity.
MIF plays a role in early embryo development, and further characterization of MIF expression and its regulation in the endometrium will add significantly to our understanding of early embryo-uterine interactions
The diverse actions of MIF within the immuno-neuroendocrine system may be a result of its occurrence in different isoforms and oligomerization states.
The purification of macrophage migration inhibitory factor (MIF) from bovine brain cytosol and its partial characterization are reported.
Transcription of MIF is induced by activation of PPARgamma2 (show PPARG Antibodies) and inhibited by excessive resistin (show RETN Antibodies).
The high activity of MIF in the maternal and fetal tissues throughout placentation and its expression in the nonpregnant uterus indicate a regulatory role for MIF during embryo receptivity and epitheliochorial placentation
This gene encodes a lymphokine involved in cell-mediated immunity, immunoregulation, and inflammation. It plays a role in the regulation of macrophage function in host defense through the suppression of anti-inflammatory effects of glucocorticoids. This lymphokine and the JAB1 protein form a complex in the cytosol near the peripheral plasma membrane, which may indicate an additional role in integrin signaling pathways.
, L-dopachrome tautomerase
, Phenylpyruvate tautomerase
, macrophage migration inhibitory factor
, phenylpyruvate tautomerase
, Macrophage migration inhibitory factor
, delayed early response protein 6
, glycosylation-inhibiting factor
, glutathione-binding 13 kDa protein