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Metallothioneins have a high content of cysteine residues that bind various heavy metals\; these proteins are transcriptionally regulated by both heavy metals and glucocorticoids.. Additionally we are shipping Metallothionein 2A Antibodies (19) and Metallothionein 2A Proteins (6) and many more products for this protein.
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Metallothioneins may have a protective role against post-burn inflammation and inflammatory organ damage, at least partly through inhibiting the p38 MAPK (show MAPK14 ELISA Kits) signaling.
Enhanced senescence due to absence of metallothionein (show MT ELISA Kits) genes may shorten lifespan.
The MT2 (show MTNR1B ELISA Kits) receptor activates Akt (show AKT1 ELISA Kits)/GSK-3beta/CRMP-2 (show DPYSL2 ELISA Kits) signaling and is necessary and sufficient to mediate functional axonogenesis and synaptic formation in central neurons.
MT1 (show MT1 ELISA Kits)/2 deficiency aggravated depleted uruanium-induced nephrotoxicity, and the molecular mechanisms appeared to be related to the increased oxidative stress and apoptosis, and decreased SGLT expression.
Zinc-binding MT1 (show MT1 ELISA Kits) and MT2 (show MTNR1B ELISA Kits) are key modulators of Fc epsilonRI-induced basophil production of IL-4 (show IL4 ELISA Kits).
Metallothionein (show MT ELISA Kits) blocks oxidative DNA damage induced by acute inorganic arsenic exposure
the metal binding abilities of mouse MT2 (show MTNR1B ELISA Kits) (mMT2) with divalent (Zn(II) and Cd(II)) and monovalent (Cu(I)) ions were analyzed and compared to those of the mouse MT1 (show MT1 ELISA Kits) (mMT1) isoform.
The results of this study suggest that metallothioneins may play a key role in protecting the kidney against high glucose-induced ROS (show ROS1 ELISA Kits) and subsequent inflammation in diabetic nephropathy.
these results demonstrate that MT-I (show MT1 ELISA Kits)/II knockout mice are more vulnerable to cadmium-induced fetal growth retardation.
Metallothioneins act as negative regulators for IL-27-induced Tr1 cells.
The present study was undertaken to explore further the interrelationship between p53 (show TP53 ELISA Kits) and metallothioneins.
HMBOX1 regulates intracellular free zinc level by interacting with MT2A (show MT2 ELISA Kits) to inhibit apoptosis and promote autophagy in vascular endothelial cells.
Polymorphism of Metallothionein 2A (show MT2 ELISA Kits) modifies Lead Body Burden in workers chronically exposed to the metal.
The findings suggest that MT2A (show MT2 ELISA Kits) gene variation rs28366003 may be implicated in the etiology of sinonasal inverted papilloma in a Polish population
Metallothionein 2A (show MT2 ELISA Kits) affects the cell respiration by suppressing the expression of mitochondrial protein (show COX6B2 ELISA Kits) cytochrome c oxidase subunit II (show COX2 ELISA Kits).
MT2A (show MT2 ELISA Kits) was down-regulated in renal cell carcinogenesis.
In the Han population of China, the gene polymorphism of MT2A (show MT2 ELISA Kits)-838G/C is correlated to coronary heart disease.
Electrospray ionization ion mobility mass spectrometry and molecular dynamics simulations reveal new insights into metal-induced conformational changes and the mechanism for metalation of human metallothionein-2A (show MT2 ELISA Kits).
MT2A (show MT2 ELISA Kits) mRNA expression has a significant inverse correlation with Cd and Cu levels in dry inverted papilloma tissue. Inverted papilloma cases with lower average cadmium and copper content were observed to be the most frequent positive MT2A (show MT2 ELISA Kits) mRNA expression.
A genetic variation of MT2A (show MT2 ELISA Kits) may be a risk factor in laryngeal cancer in a Polish population and the presence of -5 A/G (rs28366003) MT2A (show MT2 ELISA Kits) gene polymorphism could be considered as a new potential biomarker of the invasive phenotype of laryngeal cancer.
Expression of MT-1a (show MT1A ELISA Kits), MT-2b and MT-3 (show MT3 ELISA Kits) were significantly higher in high zinc fed pigs.
Metallothioneins have a high content of cysteine residues that bind various heavy metals\; these proteins are transcriptionally regulated by both heavy metals and glucocorticoids.
, metallothionein 2A