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Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 Proteins (KCNJ9)

This receptor is controlled by G proteins. Additionally we are shipping Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 Antibodies (40) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
KCNJ9 3765 Q92806
KCNJ9 16524 P48543
KCNJ9 116560 Q63511
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Top Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 Proteins at antibodies-online.com

Showing 6 out of 6 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
HOST_Escherichia coli (E. coli) Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
Insect Cells Human rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.5 mg Log in to see 49 to 54 Days
$6,041.49
Details
Insect Cells Mouse rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.25 mg Log in to see 49 to 54 Days
$4,244.78
Details
HOST_Escherichia coli (E. coli) Mouse His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
HOST_Escherichia coli (E. coli) Human Un-conjugated   5 applications Log in to see 1 to 2 Days
$312.71
Details
HOST_Human Human Un-conjugated   20 μg Log in to see 9 to 11 Days
$785.40
Details

KCNJ9 Proteins by Origin and Source

Origin Expressed in Conjugate
Human , ,
,
Mouse (Murine) ,
,
Rat (Rattus)

More Proteins for Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 (KCNJ9) Interaction Partners

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 (KCNJ9) interaction partners

  1. This study is the first to identify the expression of GIRK2 (show KCNJ6 Proteins)-4 subunits in human esophageal smooth muscle cells.

  2. a decisive role for the neuronal K(+) channel (show KCNC4 Proteins) in regulating NCAM (show NCAM1 Proteins)-dependent neurite outgrowth and attribute a physiologically meaningful role to the functional interplay of Kir3.3, NCAM (show NCAM1 Proteins), and TrkB (show NTRK2 Proteins) in ontogeny

  3. The frequencies of GRIK3 (show GRIK3 Proteins) (T928G) genotype distributions in the patients with schizophrenia were similar to those of their relatives.

  4. GIRK1 (show KCNJ3 Proteins) and GIRK2 (show KCNJ6 Proteins) channels, but not GIRK3 or GIRK4 (show KCNJ5 Proteins), may may activate signaling pathways in development of lung cancer

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 (KCNJ9) interaction partners

  1. GIRK3 appears to be a critical gatekeeper of ethanol incentive salience and a potential target for the treatment of excessive ethanol consumption.

  2. a decisive role for the neuronal K(+) channel (show KCNC4 Proteins) in regulating NCAM (show NCAM1 Proteins)-dependent neurite outgrowth and attribute a physiologically meaningful role to the functional interplay of Kir3.3, NCAM (show NCAM1 Proteins), and TrkB (show NTRK2 Proteins) in ontogeny

  3. GIRK3 exhibited in Purkinje neurons, basket cells, stellate cells and unipolar brush cells.

  4. A single locus is partially responsible for the genetic mediation of pain inhibition, and genetic variation associated with the potassium channel (show KCNAB2 Proteins) gene, Kcnj9, is a prime candidate for explaining the variable response to these analgesic drugs.

  5. Inhibition of adrenergic tone is required for the induction of dependence, and channels containing GIRK2 (show KCNJ6 Proteins) and GIRK3 serve as inhibitory gate.

  6. Results describe opioid-induced postsynaptic inhibition in locus coeruleus neurons from wild-type and GIRK2 (show KCNJ6 Proteins)/GIRK3(-/-) mice at baseline and following chronic morphine treatment.

  7. Kcnj9 as a particularly promising candidate and report the development of a Kcnj9-null mutant model that exhibits significantly less severe withdrawal from pentobarbital as well as other sedative-hypnotics.

Potassium Inwardly-Rectifying Channel, Subfamily J, Member 9 (KCNJ9) Protein Profile

Protein Summary

This receptor is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium\; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium (By similarity).

Gene names and symbols associated with KCNJ9

  • potassium inwardly-rectifying channel, subfamily J, member 9 (KCNJ9)
  • potassium inwardly-rectifying channel, subfamily J, member 9 (Kcnj9)
  • 1700085N21Rik protein
  • Girk3 protein
  • Kir3.3 protein
  • mbGIRK3 protein

Protein level used designations for KCNJ9

potassium inwardly-rectifying channel subfamily J9 , G protein-activated inward rectifier potassium channel 3 , potassium inwardly-rectifying channel, subfamily J, member 9 , G protein-coupled inward rectifier potassium channel , GIRK-3 , inward rectifier K(+) channel Kir3.3 , inwardly rectifier K+ channel KIR3.3 , potassium channel, inwardly rectifying subfamily J member 9 , inwardly rectifier K(+) channel Kir3.3

GENE ID SPECIES
469548 Pan troglodytes
614969 Bos taurus
719597 Macaca mulatta
3765 Homo sapiens
16524 Mus musculus
116560 Rattus norvegicus
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