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The protein encoded by SELE is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. Additionally we are shipping Selectin E + CD62e Kits (121) and Selectin E + CD62e Proteins (52) and many more products for this protein.
Showing 10 out of 391 products:
Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN111607
Goda, Tanaka, Monden, Miyasaka: Characterization of an apparently conserved epitope in E- and P-selectin identified by dual-specific monoclonal antibodies. in European journal of immunology 1999
Show all 11 references for ABIN111607
Human Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN319449
Thornhill, Haskard: IL-4 regulates endothelial cell activation by IL-1, tumor necrosis factor, or IFN-gamma. in Journal of immunology (Baltimore, Md. : 1950) 1990
Show all 7 references for ABIN319449
Mouse (Murine) Monoclonal Selectin E + CD62e Primary Antibody for FACS, IHC (fro) - ABIN112380
Hallmann, Zimmermann, Sorokin, Needham, Von der Mark: Adhesion of leukocytes to the inflammed endothelium. in Scandinavian journal of rheumatology. Supplement 1995
Show all 3 references for ABIN112380
Mouse (Murine) Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN112381
Hammel, Weitz-Schmidt, Krause, Moll, Vestweber, Zerwes, Hallmann: Species-specific and conserved epitopes on mouse and human E-selectin important for leukocyte adhesion. in Experimental cell research 2001
Show all 3 references for ABIN112381
Human Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN319451
Wellicome, Thornhill, Pitzalis, Thomas, Lanchbury, Panayi, Haskard: A monoclonal antibody that detects a novel antigen on endothelial cells that is induced by tumor necrosis factor, IL-1, or lipopolysaccharide. in Journal of immunology (Baltimore, Md. : 1950) 1990
Show all 3 references for ABIN319451
Human Polyclonal Selectin E + CD62e Primary Antibody for IP, IHC - ABIN223312
He, Horuk, Moochhala, Bhatia: Treatment with BX471, a CC chemokine receptor 1 antagonist, attenuates systemic inflammatory response during sepsis. in American journal of physiology. Gastrointestinal and liver physiology 2007
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Human Polyclonal Selectin E + CD62e Primary Antibody for IF (p), IHC (p) - ABIN674244
Kong, Luo, Li, Zhou, He: The anti-inflammatory effect of kaempferol on early atherosclerosis in high cholesterol fed rabbits. in Lipids in health and disease 2013
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Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN781710
Goebeler, Roth, Meinardus-Hager, Sorg: The contact allergens nickel chloride and cobalt chloride directly induce expression of endothelial adhesion molecules. in Behring Institute Mitteilungen 1994
Show all 2 references for ABIN781710
Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN1106491
Vermot-Desroches, Marchand, Roy, Wijdenes: Heterogeneity of antigen expression among human umbilical cord vascular endothelial cells: identification of cell subsets by co-expression of haemopoietic antigens. in Immunology letters 1996
SELE expression is significantly upregulated in human masticatory mucosa during wound healing
E-selectin concentrations in this study were associated with hyperglycemia, possibly reflecting early endothelial damage. E-selectin was not useful to assess carotid intima-media thickness, a marker of subclinical atherosclerosis, which appeared to be determined by aging and male gender.
Plasma E-selectin is overexpressed in pediatric patients with mycoplasma pneumoniae pneumonia and myocardial injury.
The significant presence of CLA (show SELPLG Antibodies)+ T cells and E-selectin (CD62E) expressions in the OLPG suggests their involvement in the etiopathogenesis of oral lichen planus ; however, only a weak correlation between CLA (show SELPLG Antibodies)+ T cells and E-selectin was observed.
BD patients have a lower expression of ICAM-1 (show ICAM1 Antibodies), VCAM-1 (show VCAM1 Antibodies), and E-selectin in their tumor tissues. BD-HDL (show HSD11B1 Antibodies) facilitates the adhesion of tumor cells to vascular endothelium by upregulating the expression of ICAM-1 (show ICAM1 Antibodies) and VCAM-1 (show VCAM1 Antibodies), thereby promoting the initial progression of breast cancer metastasis
The E-selectin S149R polymorphisms is associated with the oncogenesis of breast cancer.
Neisseria meningitidis caused a high level of E-selectin expression elicited by the activity of phosphorylated ATF2 (show ATF2 Antibodies) transcription factor on the E-selectin promoter.
E-selectin is more effective than cTnI in diagnosing myocardial injury in children.
Inhibition of E-selectin expression allows an anti-tumoral effect on sLex-expressing hepatocellular carcinoma tumors in vivo
Glycosphingolipids likely contribute to human myeloid cell adhesion to E-selectin under fluid shear, particularly the transition of rolling cells to firm arrest.
Chitosan oligosaccharides downregulate the expression of E-selectin and ICAM-1 (show ICAM1 Antibodies) by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Antibodies) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Increased E-selectin mRNA at end of 20 hours of cold ischemia time might indicate a preactivated state of endothelial cells potentially triggered by bacterial translocation or products.
In a model of corneal transplantation, P- and E-selectin mediate T cell recruitment to the graft, E-selectin mediates APC (show APC Antibodies) trafficking to lymphoid tissue, and blockade of E-selectin has a modest effect on improving long-term graft survival.
Social defeat induced an exposure-dependent increase in mRNA levels of E-selectin, CXCL1 (show CXCL1 Antibodies), and CXCL2 (show CXCL2 Antibodies) that increased with additional days of social defeat.
The histopathological modification of lupus nephritis by non-nephritogenic bystander IgM (show CD40LG Antibodies) antibodies is associated in part with glomerular E-selectin expression.
Data show that actin cytoskeleton regulated CD44 (show CD44 Antibodies) membrane mobility and interactions with E-selectin.
Regulatory T cells dynamically regulate P and E selectin ligand expression during multiple challenge contact hypersensitivity.
Systemic inflammation may increase the expression of E-selectin which mediated the lung metastasis of breast cancer in mouse model.
In the absence of either PSGL-1 (show SELPLG Antibodies) or E- and P-selectin (show SELP Antibodies).
Elevated E-selectin plays an important role in hepatic neutrophil infiltration and injury induced by chronic-binge feeding in mice and may also contribute to the pathogenesis of early stages of human alcoholic liver disease.
expression of E-selectin and ICAM-1 (show ICAM1 Antibodies) are suppressed by paeoniflorin in a mouse model of cutaneous Arthus reaction
Expression of E-selectin is sufficient to support rescue & hematopoietic reconstitution of lethally irradiated recipients forllowing bone marrow transplant.
The protein encoded by this gene is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. It exhibits structural features such as the presence of lectin- and EGF-like domains followed by short consensus repeat (SCR) domains that contain 6 conserved cysteine residues. These proteins are part of the selectin family of cell adhesion molecules. Adhesion molecules participate in the interaction between leukocytes and the endothelium and appear to be involved in the pathogenesis of atherosclerosis.
, endothelial adhesion molecule 1
, selectin E (endothelial adhesion molecule 1)
, CD62 antigen-like family member E
, endothelial leukocyte adhesion molecule 1
, leukocyte endothelial cell adhesion molecule 2
, leukocyte-endothelial cell adhesion molecule 2
, selectin, endothelial cell