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The protein encoded by SELE is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. Additionally we are shipping Selectin E + CD62e Kits (172) and Selectin E + CD62e Proteins (53) and many more products for this protein.
Showing 10 out of 389 products:
Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN111607
Goda, Tanaka, Monden, Miyasaka: Characterization of an apparently conserved epitope in E- and P-selectin identified by dual-specific monoclonal antibodies. in European journal of immunology 1999
Show all 11 references for ABIN111607
Human Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN319449
Thornhill, Haskard: IL-4 regulates endothelial cell activation by IL-1, tumor necrosis factor, or IFN-gamma. in Journal of immunology (Baltimore, Md. : 1950) 1990
Show all 7 references for ABIN319449
Mouse (Murine) Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN112381
Hallmann, Zimmermann, Sorokin, Needham, Von der Mark: Adhesion of leukocytes to the inflammed endothelium. in Scandinavian journal of rheumatology. Supplement 1995
Show all 3 references for ABIN112381
Human Polyclonal Selectin E + CD62e Primary Antibody for IP, IHC - ABIN223312
He, Horuk, Moochhala, Bhatia: Treatment with BX471, a CC chemokine receptor 1 antagonist, attenuates systemic inflammatory response during sepsis. in American journal of physiology. Gastrointestinal and liver physiology 2007
Show all 3 references for ABIN223312
Human Monoclonal Selectin E + CD62e Primary Antibody for EIA, FACS - ABIN319451
Wellicome, Thornhill, Pitzalis, Thomas, Lanchbury, Panayi, Haskard: A monoclonal antibody that detects a novel antigen on endothelial cells that is induced by tumor necrosis factor, IL-1, or lipopolysaccharide. in Journal of immunology (Baltimore, Md. : 1950) 1990
Show all 3 references for ABIN319451
Mouse (Murine) Monoclonal Selectin E + CD62e Primary Antibody for FACS, IHC (fro) - ABIN112380
Hammel, Weitz-Schmidt, Krause, Moll, Vestweber, Zerwes, Hallmann: Species-specific and conserved epitopes on mouse and human E-selectin important for leukocyte adhesion. in Experimental cell research 2001
Show all 3 references for ABIN112380
Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN781710
Goebeler, Roth, Meinardus-Hager, Sorg: The contact allergens nickel chloride and cobalt chloride directly induce expression of endothelial adhesion molecules. in Behring Institute Mitteilungen 1994
Show all 2 references for ABIN781710
Human Polyclonal Selectin E + CD62e Primary Antibody for IF (p), IHC (p) - ABIN674244
Kong, Luo, Li, Zhou, He: The anti-inflammatory effect of kaempferol on early atherosclerosis in high cholesterol fed rabbits. in Lipids in health and disease 2013
Show all 2 references for ABIN674244
Human Monoclonal Selectin E + CD62e Primary Antibody for FACS - ABIN1106491
Vermot-Desroches, Marchand, Roy, Wijdenes: Heterogeneity of antigen expression among human umbilical cord vascular endothelial cells: identification of cell subsets by co-expression of haemopoietic antigens. in Immunology letters 1996
The E-selectin S149R polymorphisms is associated with the oncogenesis of breast cancer.
Neisseria meningitidis caused a high level of E-selectin expression elicited by the activity of phosphorylated ATF2 (show ATF2 Antibodies) transcription factor on the E-selectin promoter.
E-selectin is more effective than cTnI in diagnosing myocardial injury in children.
Inhibition of E-selectin expression allows an anti-tumoral effect on sLex-expressing hepatocellular carcinoma tumors in vivo
Glycosphingolipids likely contribute to human myeloid cell adhesion to E-selectin under fluid shear, particularly the transition of rolling cells to firm arrest.
Data show that both mucin16 (MUC16 (show MUC16 Antibodies)) and podocalyxin (PODXL (show PODXL Antibodies))-E-selectin-mediated interactions are mechanically stronger than like L-selectin (show SELL Antibodies) interactions at the single-molecule level.
These findings provide consistent evidence that A516C and G98T polymorphisms of the SELE gene may be associated with increased susceptibility of coronary artery disease.
Glaucomatous MYOC (show MYOC Antibodies) mutations activate the IL-1beta (show IL1B Antibodies)/NF-kappaB (show NFKB1 Antibodies) inflammatory stress response and the glaucoma marker SELE in trabecular meshwork cells.
P. gingivalis was shown to activate NOD1 (show NOD1 Antibodies), NOD2 (show NOD2 Antibodies), and TLR2 (show TLR2 Antibodies) expression, resulting in increased E-selectin expression in endothelial cells; direct inhibition of NF-kappaB (show NFKB1 Antibodies) and P38 MAPK (show MAPK14 Antibodies) significantly attenuated E-selectin expression induced by P. gingivalis in endothelial cells
The number of E-selectin-positive cells was significantly higher in the tuberculoid form than in the lepromatous form of leprosy skin lesions.
Chitosan oligosaccharides downregulate the expression of E-selectin and ICAM-1 (show ICAM1 Antibodies) by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Antibodies) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Increased E-selectin mRNA at end of 20 hours of cold ischemia time might indicate a preactivated state of endothelial cells potentially triggered by bacterial translocation or products.
In a model of corneal transplantation, P- and E-selectin mediate T cell recruitment to the graft, E-selectin mediates APC (show APC Antibodies) trafficking to lymphoid tissue, and blockade of E-selectin has a modest effect on improving long-term graft survival.
Social defeat induced an exposure-dependent increase in mRNA levels of E-selectin, CXCL1 (show CXCL1 Antibodies), and CXCL2 (show CXCL2 Antibodies) that increased with additional days of social defeat.
The histopathological modification of lupus nephritis by non-nephritogenic bystander IgM (show CD40LG Antibodies) antibodies is associated in part with glomerular E-selectin expression.
Data show that actin cytoskeleton regulated CD44 (show CD44 Antibodies) membrane mobility and interactions with E-selectin.
Regulatory T cells dynamically regulate P and E selectin ligand expression during multiple challenge contact hypersensitivity.
Systemic inflammation may increase the expression of E-selectin which mediated the lung metastasis of breast cancer in mouse model.
In the absence of either PSGL-1 (show SELPLG Antibodies) or E- and P-selectin (show SELP Antibodies).
Elevated E-selectin plays an important role in hepatic neutrophil infiltration and injury induced by chronic-binge feeding in mice and may also contribute to the pathogenesis of early stages of human alcoholic liver disease.
expression of E-selectin and ICAM-1 (show ICAM1 Antibodies) are suppressed by paeoniflorin in a mouse model of cutaneous Arthus reaction
Expression of E-selectin is sufficient to support rescue & hematopoietic reconstitution of lethally irradiated recipients forllowing bone marrow transplant.
The protein encoded by this gene is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. It exhibits structural features such as the presence of lectin- and EGF-like domains followed by short consensus repeat (SCR) domains that contain 6 conserved cysteine residues. These proteins are part of the selectin family of cell adhesion molecules. Adhesion molecules participate in the interaction between leukocytes and the endothelium and appear to be involved in the pathogenesis of atherosclerosis.
, endothelial adhesion molecule 1
, selectin E (endothelial adhesion molecule 1)
, CD62 antigen-like family member E
, endothelial leukocyte adhesion molecule 1
, leukocyte endothelial cell adhesion molecule 2
, leukocyte-endothelial cell adhesion molecule 2
, selectin, endothelial cell