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ETS1 encodes a member of the ETS family of transcription factors, which are defined by the presence of a conserved ETS DNA-binding domain that recognizes the core consensus DNA sequence GGAA/T in target genes. Additionally we are shipping V-Ets erythroblastosis Virus E26 Oncogene Homolog 1 (Avian) Kits (14) and V-Ets erythroblastosis Virus E26 Oncogene Homolog 1 (Avian) Proteins (10) and many more products for this protein.
Showing 10 out of 177 products:
Human Polyclonal ETS1 Primary Antibody for EIA, FACS - ABIN952130
Harris, Yamakuchi, Kondo, Oettgen, Lowenstein: Ets-1 and Ets-2 regulate the expression of microRNA-126 in endothelial cells. in Arteriosclerosis, thrombosis, and vascular biology 2010
Show all 4 references for ABIN952130
Human Monoclonal ETS1 Primary Antibody for ICC, FACS - ABIN1724735
Chen, Shin, Lin: Ets1 was significantly activated by ERK1/2 in mutant K-ras stably transfected human adrenocortical cells. in DNA and cell biology 2005
Show all 2 references for ABIN1724735
Human Monoclonal ETS1 Primary Antibody for IHC, ELISA - ABIN1724736
Kessler, Schroeder, Brar, Handwerger: Transcription factor ETS1 is critical for human uterine decidualization. in Molecular human reproduction 2006
Show all 2 references for ABIN1724736
Human Polyclonal ETS1 Primary Antibody for IF, IHC - ABIN1531315
Gao, Peng, Liang, Shahbaz, Liu, Wang, Sun, Niu, Niu, Liu, Wang, Lin, Wang, Niu: β6 integrin induces the expression of metalloproteinase-3 and metalloproteinase-9 in colon cancer cells via ERK-ETS1 pathway. in Cancer letters 2014
Human Monoclonal ETS1 Primary Antibody for ELISA, IHC - ABIN4309665
Meerson, Traurig, Ossowski, Fleming, Mullins, Baier: Human adipose microRNA-221 is upregulated in obesity and affects fat metabolism downstream of leptin and TNF-α. in Diabetologia 2013
Chicken Polyclonal ETS1 Primary Antibody for IHC, WB - ABIN2792530
Yordy, Li, Sementchenko, Pei, Muise-Helmericks, Watson: SP100 expression modulates ETS1 transcriptional activity and inhibits cell invasion. in Oncogene 2004
the affinities of two sequence-divergent ETS homologs, PU.1 and Ets-1, to DNA sites harboring a hemi- and fully methylated CpG dinucleotide, were measured.
data support a general model in which Ets-1 interacts with nonspecific DNA via dynamic electrostatic interactions, whereas hydrogen bonding drives the formation of well-ordered complexes with specific DNA
Ets-1 links glucotoxicity to pancreatic beta cell dysfunction through inhibiting PDX-1 (show PDX1 Antibodies) expression in type 2 diabetes.
Akt2 (show AKT2 Antibodies) upregulates dendritic cell migration at least in part by ETS1-dependent stimulation of IP3R2 (show ITPR2 Antibodies) transcription.
The short nose, otitis media, and hearing impairment in Jacobsen syndrome are likely because of hemizygosity for ETS1 and FLI1 (show FLI1 Antibodies).
Ets1 and Sox10 (show SOX10 Antibodies) interact to promote proper melanocyte and enteric ganglia development from the neural crest.
our data suggested that ESE1/ELF3 (show ELF3 Antibodies) may promote the UC progression via accelerating NF-kappaB (show NFKB1 Antibodies) activation and thus facilitating IEC apoptosis.
maintenance of appropriate c-ets-1 levels is essential to prevent loss of self-tolerance in the B cell compartment.
Data indicate interactions between Ets1 transcription factor and two of its important regulators: Lyn (show LYN Antibodies), which maintains Ets1 expression to limit the differentiation of autoreactive plasma cells (PCs), and Btk (show BTK Antibodies).
our results demonstrate that both Ets1 and miR (show MLXIP Antibodies)-155 can directly regulate the expression of CD1d (show CD1D Antibodies) on B-cells
Ets-1 p27 (show PAK2 Antibodies) isoform is cleaved in the same manner as Ets-1 p51 (show ZNF398 Antibodies) isoform within the exon VII (show TH Antibodies)-encoded region, thus generating a stable C-terminal fragment that induces cell death by initiating apoptosis.
Our results indicated that ETS1 induced autophagy after inhibition of glycolysis, and thus led to comparative decrease of cell viability. These results implied that ETS1 could be a potential target for tumor metabolic therapy.
Furthermore, mir106a transfection resulted in decreased expression of MMP-2 (show MMP2 Antibodies) and diminished binding activity of transcription factor Ets (show ETV7 Antibodies)-1 in EJ cells.
Integrin b6 markedly promoted the proliferation and invasion of pancreatic carcinoma cells and induced ETS1 phosphorylation in an ERK (show EPHB2 Antibodies)-dependent manner, leading to the upregulation of matrix metalloprotease (show ADAM8 Antibodies)-9, which is essential for b6-mediated invasiveness of pancreatic carcinoma cells
SSRP1 (show SSRP1 Antibodies)/Ets-1/Pim-3 (show PIM3 Antibodies) signalling is tightly associated with the proliferation, apoptosis, autophagy, invasion and clonogenicity of nasopharyngeal carcinoma cells, and blockage of this signalling facilitates chemosensitivity of the cells to docetaxel.
MiR (show MLXIP Antibodies)-155 regulates the Th17 immune response by targeting Ets-1 in Behcet's disease.
results provide insight into the regulation of beta1 integrin through miR (show MLXIP Antibodies)-199a-5p-mediated Ets-1 silence and will help in designing new therapeutic strategies to inhibit signal pathways induced by miR (show MLXIP Antibodies)-199a-5p in breast cancer invasion
the markers Ets-1 and APAF-1 (show APAF1 Antibodies) relative to p53 (show TP53 Antibodies), Ki-67 (show MKI67 Antibodies) and PTEN (show PTEN Antibodies) expression in colon adenomas/polyps, were investigated.
NR4As regulate gene transcription primarily through interaction with distal enhancers that are co-enriched for NR4A1 (show NR4A1 Antibodies) and ETS transcription factor (show FEV Antibodies) motifs
A significant positive correlation was seen between ETS-1 rs73013527 and RA susceptibility. Carriers of the haplotype CCT (show FLVCR2 Antibodies) or TCT (show TRPM7 Antibodies) for rs4937333, rs11221332 and rs73013527 had decreased risk. No effect was seen for rs10893872, rs4937333 and rs11221332.
The activity of Gata4 (show GATA4 Antibodies) cardiac enhancer in transgenic embryos and in cultured aortic endothelial cells is dependent on four ETS sites.
Ets-1 up-regulation is involved in the development of retinal neovascularization.
This gene encodes a member of the ETS family of transcription factors, which are defined by the presence of a conserved ETS DNA-binding domain that recognizes the core consensus DNA sequence GGAA/T in target genes. These proteins function either as transcriptional activators or repressors of numerous genes, and are involved in stem cell development, cell senescence and death, and tumorigenesis. Alternatively spliced transcript variants encoding different isoforms have been described for this gene.
v-ets erythroblastosis virus E26 oncogene homolog 1
, Ets protein
, v-ets erythroblastosis virus E26 oncogene homolog 1 (avian)
, protein c-ets-1-B
, C-ets-1 protein (p54)
, avian erythroblastosis virus E26 oncogene 1
, protein C-ets-1
, tumor progression locus 1
, ELK channel 3
, ether-a-go-go-like potassium channel 1
, ether-a-go-go-like potassium channel 3
, potassium voltage-gated channel subfamily H member 8
, potassium voltage-gated channel, subfamily H, member 8
, voltage-gated potassium channel subunit Kv12.1
, Avian erythroblastosis virus E26 (v-ets) oncogene homolog-1
, ets protein
, v-ets avian erythroblastosis virus E2 oncogene homolog 1
, Ets avian erythroblastosis virus E2 oncogene homolog 1 (tumor progression locus 1)
, TNFAIP3 interacting protein 1
, c-ets-1 oncogene
, p54 transcription factor
, transforming protein p54/c-ets-1
, transforming protein p68/c-ets-1
, virion-associated nuclear-shuttling protein
, v-ets erythroblastosis virus E26 oncogene-like protein 1