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Protects actin filaments from depolymerization (By similarity). Additionally we are shipping XIRP1 Kits (2) and many more products for this protein.
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the porcine CMYA1 gene has effects on porcine back fat deposition
The results thus suggest that the CMYA1 promoter could be an effective muscle-specific (show EIF3K Antibodies) promoter, which may be useful in further studies of cardiomyopathy treatment and transgenic animal research
Aciculin (show PGM5 Antibodies) interacts with filamin C (show FLNC Antibodies) and Xin and is essential for myofibril assembly.
The strong correlation between the degree of muscle damage and Xin immunoreactivity suggests that Xin may be a suitable outcome measure to evaluate disease progression and treatment effects in clinical trials.
We identify the SH3 domains of nebulin (show NEB Antibodies) and nebulette (show NEBL Antibodies) as novel ligands of proline-rich regions of Xin and XIRP2 (show XIRP2 Antibodies).
Data provide further support that ALS2CL (show ALS2CL Antibodies), EPHA3 (show EPHA3 Antibodies), and CMYA1 are bona-fide tumor-suppressor genes and contribute to the tumorigenesis of HNSCC.
Xin and XIRP2 define a novel actin-binding motif
Here, we identify Xin, the protein encoded by the human gene 'cardiomyopathy associated 1'(CMYA1) as filamin c (show FLNC Antibodies) binding partner at these specialized structures where the ends of myofibrils are attached to the sarcolemma
Data show that in myofibrillar myopathies Xin protein exhibites significant alterations in their localization.
Xin deficiency leads to a myopathic condition characterized by increased muscle fatigability
The results suggest that mXinalpha play a role in myotendinous (show TNC Antibodies) junction conductance of contractile and stretching forces.
mXinalpha controls the dynamics of cortical actin cytoskeleton.
Data show that mXinbeta (CMYA3/XIRP2 (show XIRP2 Antibodies)) but not mXinalpha ((CMYA1/XIRP1) was uniquely up-regulated during the redistribution of intercellular junction from the lateral membrane of cardiomyocytes to their termini.
Total Xin deficiency leads to topographical intercalated disc alterations, premature fibrosis and subtle changes in contractile behaviour; this is a milder cardiac phenotype than that observed in XinAB(-/-) mice, which still can express XinC
Xin has a role in the formation of cell-cell contacts and possibly in myofibrillogenesis
Xin is expressed within muscle satellite cells during skeletal muscle regeneration and is involved in the regulation of myoblast function.
The binding of mXin alpha to beta-catenin at the adherens junction would then facilitate actin binding.
Protects actin filaments from depolymerization (By similarity).
xin actin-binding repeat containing 1
, cardiomyopathy associated 1
, xin actin-binding repeat-containing protein 1
, cardiomyopathy-associated 1
, xin actin-binding repeat-containing protein 1-like
, Xin alpha 2
, Xin repeat-containing protein
, cardiomyopathy-associated protein 1
, cardiomyopathy associated protein 1
, Xin alpha
, mXin alpha
, cardiac morphogenesis
, cardiomyopathy associated 3
, xin actin-binding repeat containing 2