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ZBTB16 is a member of the Krueppel C2H2-type zinc-finger protein family and encodes a zinc finger transcription factor that contains nine Kruppel-type zinc finger domains at the carboxyl terminus. Additionally we are shipping ZBTB16 Proteins (3) and many more products for this protein.
Showing 10 out of 84 products:
Human Monoclonal ZBTB16 Primary Antibody for ELISA - ABIN393658
Bailey, Xie, Do, Montpetit, Diaz, Mohan, Keavney, Yusuf, Gerstein, Engert, Anand: Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study. in Diabetes Care 2010
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Mouse (Murine) Monoclonal ZBTB16 Primary Antibody for FACS - ABIN2662834
Chen, Brand, Chen, Chen, Tong, Wang, Waxman, Zelent: Fusion between a novel Krüppel-like zinc finger gene and the retinoic acid receptor-alpha locus due to a variant t(11;17) translocation associated with acute promyelocytic leukaemia. in The EMBO journal 1993
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Human Polyclonal ZBTB16 Primary Antibody for EIA - ABIN358501
Labbaye, Spinello, Quaranta, Pelosi, Pasquini, Petrucci, Biffoni, Nuzzolo, Billi, Foà, Brunetti, Grignani, Testa, Peschle: A three-step pathway comprising PLZF/miR-146a/CXCR4 controls megakaryopoiesis. in Nature cell biology 2008
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Chicken Polyclonal ZBTB16 Primary Antibody for WB - ABIN2780737
Deng, Zhang, Kang, Liu, Zhao, Gao, Li, Pan, Tang, Wang, Niu, Yang, Zeng: An unusual haplotype structure on human chromosome 8p23 derived from the inversion polymorphism. in Human mutation 2008
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Human Polyclonal ZBTB16 Primary Antibody for EIA, WB - ABIN453283
Shi, Vogt: Posttranslational regulation of Myc by promyelocytic leukemia zinc finger protein. in International journal of cancer. Journal international du cancer 2009
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Human Monoclonal ZBTB16 Primary Antibody for ICC, ELISA - ABIN969465
Felicetti, Errico, Bottero, Segnalini, Stoppacciaro, Biffoni, Felli, Mattia, Petrini, Colombo, Peschle, Carè: The promyelocytic leukemia zinc finger-microRNA-221/-222 pathway controls melanoma progression through multiple oncogenic mechanisms. in Cancer research 2008
Emerging evidence shows that PLZF regulates the balance of self-renewal and differentiation in stem cells.
integration of PLZF ChIP-Seq and RNA Pol II RNA-Seq datasets revealed that the early growth response 1 (EGR1 (show EGR1 Antibodies)) transcription factor is a PLZF target for which its level of expression must be reduced to enable progesterone dependent hESC decidualization
NLRP7 (show NLRP7 Antibodies) is involved in Hydatidiform Molar Pregnancy (HYDM1) and interacts with the transcriptional repressor ZBTB16
These results support a prognostic value for loss of cytoplasmic PLZF expression in the stratification of non-small cell lung cancer and a possible role of cytoplasmic shift and down-regulation of PLZF in the pathogenesis of NSCLC.
down-regulation of PLZF is an important molecular process for prostate tumor progression
Down-regulation of PLZF in human hepatocellular carcinoma.PLZF expression was positively correlated with the alkaline phosphatase level in hepatocellular carcinoma patients.
In dental follicle cells, a Runx2 (show RUNX2 Antibodies)-independent differentiation mechanism exists that is regulated by ZBTB16.
PLZF loss enhances castration-resistant prostate cancer tumor growth in a xenograft model.
Data suggest that interleukin-32alpha (IL-32alpha) associates with leukemia zinc finger (PLZF) and protein kinase c epsilon (show PRKCE Antibodies) (PKCvarepsilon), and then inhibits PLZF sumoylation, resulting in suppression of the transcriptional activity of PLZF.
PLZF regulates CCR6 (show CCR6 Antibodies) and is critical for the acquisition and maintenance of the Th17 phenotype in human cells.
Results clearly indicate that PLZF promotes cell cycle exit and cell differentiation into hypertrophic chondrocytes.
plzf has a role in regulating HSC (show FUT1 Antibodies) function that is linked to cell cycle regulation
PLZF expression is highly specific to innate T cells and cannot be induced in conventional T cells via TCR-mediated activation or inflammatory challenge
PLZF expression at the innate lymphoid cell precursor stage has a long-range effect on the functional properties of mature ILC2s and highlights the importance of these cells for innate allergic responses in otherwise immunocompetent mice.
Data suggest that age-related increase in Plzf expression represents a novel molecular signature of spermatogonia aging by functionally arresting their differentiation.
Data indicate the function of promyelocytic leukaemia zinc finger PLZF in the development of innate-like interleukin-17 (show IL17A Antibodies), TcR gamma-delta (IL-17 (show IL17A Antibodies)+Vgamma6+ gammadelta) T cells.
The data suggest that Pak2 (show PAK2 Antibodies) controls thymic Natural Killer T-cell development by providing a signal that links Egr2 (show EGR2 Antibodies) to induce PLZF, in part by regulating signaling lymphocyte activation molecule (show SLAMF1 Antibodies) 6 expression.
The acetyltransferase HAT1 (show HAT1 Antibodies) moderates the NF-kappaB (show NFKB1 Antibodies) inflammatory response by regulating the transcription factor PLZF.
G-protein-coupled receptors, such as CXCR4, regulate autophagy by ZBTB16-mediated ubiquitination and proteasomal degradation of Atg14L .
Data conclude that PLZF is a critical regulator of hepatic gluconeogenesis.
This gene is a member of the Krueppel C2H2-type zinc-finger protein family and encodes a zinc finger transcription factor that contains nine Kruppel-type zinc finger domains at the carboxyl terminus. This protein is located in the nucleus, is involved in cell cycle progression, and interacts with a histone deacetylase. Specific instances of aberrant gene rearrangement at this locus have been associated with acute promyelocytic leukemia (APL). Alternate transcriptional splice variants have been characterized.
zinc finger and BTB domain-containing protein 16
, zinc finger protein 145 (Kruppel-like, expressed in promyelocytic leukemia)
, zinc finger protein PLZF
, promyelocytic leukemia zinc finger protein
, Green's luxoid
, zinc finger protein 145
, Polydactyly-luxate syndrome
, promyelocytic leukemia zinc finger
, zinc finger and BTB domain containing 16 protein
, promyelocytic leukemia zinc finger protein-A
, zinc finger and BTB domain containing 16
, zinc finger and BTB domain-containing protein 16-A
, zinc finger protein PLZF-A