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GABA is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. Additionally we are shipping GABRA3 Proteins (6) and GABRA3 Kits (4) and many more products for this protein.
Showing 10 out of 65 products:
Cow (Bovine) Polyclonal GABRA3 Primary Antibody for IHC (fro), WB - ABIN372630
McKernan, Rosahl, Reynolds, Sur, Wafford, Atack, Farrar, Myers, Cook, Ferris, Garrett, Bristow, Marshall, Macaulay, Brown, Howell, Moore, Carling, Street, Castro, Ragan, Dawson, Whiting: Sedative but not anxiolytic properties of benzodiazepines are mediated by the GABA(A) receptor alpha1 subtype. in Nature neuroscience 2000
Show all 6 references for ABIN372630
Rat (Rattus) Polyclonal GABRA3 Primary Antibody for ICC, IHC - ABIN1742459
Tzanoulinou, García-Mompó, Castillo-Gómez, Veenit, Nacher, Sandi: Long-Term Behavioral Programming Induced by Peripuberty Stress in Rats Is Accompanied by GABAergic-Related Alterations in the Amygdala. in PLoS ONE 2014
Cow (Bovine) Polyclonal GABRA3 Primary Antibody for IHC, WB - ABIN2776110
Chou: Modelling extracellular domains of GABA-A receptors: subtypes 1, 2, 3, and 5. in Biochemical and biophysical research communications 2004
Human Polyclonal GABRA3 Primary Antibody for WB - ABIN871662
Kaupmann, Huggel, Heid, Flor, Bischoff, Mickel, McMaster, Angst, Bittiger, Froestl, Bettler: Expression cloning of GABA(B) receptors uncovers similarity to metabotropic glutamate receptors. in Nature 1997
Cow (Bovine) Polyclonal GABRA3 Primary Antibody for IHC, WB - ABIN2792260
Hadingham, Wingrove, Le Bourdelles, Palmer, Ragan, Whiting: Cloning of cDNA sequences encoding human alpha 2 and alpha 3 gamma-aminobutyric acidA receptor subunits and characterization of the benzodiazepine pharmacology of recombinant alpha 1-, alpha 2-, alpha 3-, and alpha 5-containing human gamma-aminobutyric ac in Molecular pharmacology 1993
Gabra3 activates the AKT (show AKT1 Antibodies) pathway to promote breast cancer cell migration, invasion and metastasis. The A-to-I RNA-edited form of Gabra3 is found only in non-invasive breast cancers. Edited Gabra3 suppresses breast cancer cell invasion and metastasis.
The N-terminal region of GABRA3 and the GlyR beta subunit (show POLG Antibodies) occupies the same binding site of gephyrin (show GPHN Antibodies).
high expression of GABBR2 (show GABBR2 Antibodies) with a low expression of GABR(A3) may predict a better outcome in non-small cell lung cancer
A novel role for GA-repeat core promoter to regulate gene expression in the genes such as GABRA3 involved in development and evolution.
We identified truncating mutations in distinct X-linked gamma-aminobutyric acid A (GABAA (show GABRg1 Antibodies)) receptor subunit-encoding genes, GABRQ (show GABRQ Antibodies) and GABRA3.this is the first report of ASD (show ARSD Antibodies) patients with truncating mutations in GABA receptors genes.
The results indicate that there may be specific GABA receptor gene expression variation in migraine, particularly involving the GABRA3 and GABBR2 (show GABBR2 Antibodies) genes.
The ratios of beta(3)/beta(2) and alpha(5)/alpha(1) subunit protein expression in Angelman syndrome (show ube3a Antibodies) cortex were significantly decreased when compared with controls.
GABRA3 gene was overexpressed in lung cancer and rarely expressed in other cancers
These findings suggest that common variation in the GABRA2 (show GABRA2 Antibodies), GABRA3, GABRA6 (show GABRA6 Antibodies), and GABRG2 (show GABRG2 Antibodies) genes does not play a major role in liability to anxiety spectrum disorders.
The results of our study indicate that GABRA 3 gene might also be involved in the genetic pathophysiology of unipolar major depressive disorder
Absence of the Gabra3 receptor subunit slows recovery from ethanol-induced incoordination.
Quantitative trait locus analysis identifies Gabra3 as a regulator of behavioral despair in mice
Despite the exclusive expression of alpha3-containing GABAA (show GABRg1 Antibodies) receptors in the reticular thalamic nucleus, these receptors do not seem to be critical for the mediation of the effects of diazepam on the sleep EEG
editing of the Gabra-3 mRNA is important for normal brain development.The extent of editing is low at birth but increases with age, reaching close to 100% in the adult brain.
The phenotypes following global deletion of the GABAA (show GABRg1 Antibodies) receptor alpha3 subunit provided relevant insights into the potential behavioural relevance of this specific receptor subtypes in the modulation of both affective and cognitive functions.
Deletion of the GABAAR (show GABRG2 Antibodies) alpha3 subunit reduces evoked thalamic oscillatory activity.
Resutls show that the variation in editing gave receptors like HTR2C (show HTR2C Antibodies) and GABA(A) (gamma-aminobutyric acid type A) a different set of protein isoforms during development from those in the adult animal.
GABA is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. Chloride conductance of these channels can be modulated by agents such as benzodiazepines that bind to the GABA-A receptor. At least 16 distinct subunits of GABA-A receptors have been identified.
gamma-aminobutyric acid (GABA) A receptor, alpha 3
, gamma-aminobutyric acid A receptor, alpha 3
, gamma-aminobutyric acid A receptor, alpha 3-like
, gamma-aminobutyric acid receptor subunit alpha-3-like
, GABA(A) receptor subunit alpha-3
, GABA(A) receptor, alpha 3
, gamma-aminobutyric acid receptor subunit alpha-3
, gamma-animobutyric acid (GABA) A receptor, alpha 3
, GABA-A receptor alpha-3 subunit
, Gamma-animobutyric acid (GABA) A receptor alpha 3
, Gamma-animobutyric acid (GABA) A receptor, alpha 3
, gamma-aminobutyric acid (GABA-A) receptor, subunit alpha 3
, gamma-aminobutyric acid receptor, subunit alpha 3