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anti-Human PTH Antibodies:
anti-Rat (Rattus) PTH Antibodies:
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Human Monoclonal PTH Primary Antibody for ELISA, WB - ABIN1724741
Koshizuka, Ogata, Shiraki, Hosoi, Seichi, Takeshita, Nakamura, Kawaguchi: Distinct association of gene polymorphisms of estrogen receptor and vitamin D receptor with lumbar spondylosis in post-menopausal women. in European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society 2006
Show all 2 references for ABIN1724741
Human Monoclonal PTH Primary Antibody for EIA, IHC (fro) - ABIN191965
Tampe, Broszio, Manneck, Missbichler, Blind, Müller, Schmidt-Gayk, Armbruster: Characterization of antibodies against human N-terminal parathyroid hormone by epitope mapping. in Journal of immunoassay 1992
Show all 2 references for ABIN191965
Human Monoclonal PTH Primary Antibody for ELISA, ICC - ABIN260228
Koh, Hogue, Sosa: A Novel Ex Vivo Method for Visualizing Live-Cell Calcium Response Behavior in Intact Human Tumors. in PLoS ONE 2016
Human Monoclonal PTH Primary Antibody for EIA, IHC (p) - ABIN781709
Habener, Rosenblatt, Potts: Parathyroid hormone: biochemical aspects of biosynthesis, secretion, action, and metabolism. in Physiological reviews 1984
Human Monoclonal PTH Primary Antibody for EIA, IHC (fro) - ABIN191904
Mägerlein, Hock, Adermann, Müller-Beckmann, Neidlein, Forssmann, Stein: A new immunoenzymometric assay for bioactive N-terminal human parathyroid hormone fragments and its application in pharmacokinetic studies in dogs. in Arzneimittel-Forschung 1998
Elevated PTH induces the transition of endothelial cells to chondrogenic cells via endothelial-mesenchymal transition, possibly mediated by the nuclear translocation of beta-catenin (show CTNNB1 Antibodies).
Cys (show DNAJC5 Antibodies) mutation at the 25th residue of hPTH(1-34) may result in a high bone mass phenotype.
Common genetic variants located near genes involved in vitamin D metabolism and calcium and renal phosphate transport associated with differences in circulating parathyroid hormone concentrations
PTH pretreatment prevented TGF-beta1 (show TGFB1 Antibodies) and high glucose-induced Smad2 (show SMAD2 Antibodies)/3 phosphorylation and consequent upregulation of fibronectin (show FN1 Antibodies) and type IV collagen (show COL4 Antibodies) within 4 h.
FGFR1c and PTHR (show PTHLH Antibodies) signaling pathways converge on NHERF1 (show SLC9A3R1 Antibodies) to inhibit PTH- and FGF23 (show FGF23 Antibodies)-sensitive phosphate transport and down-regulate NPT2A (show SLC34A1 Antibodies).
In patients receiving dual antiplatelet therapy for coronary artery disease, higher PTH levels are associated with an increased ADP-mediated platelet reactivity and suboptimal response to clopidogrel.
Parathyroid hormone gene rs6256 variants are not associated with susceptibility to colorectal cancer
PTH levels were significantly higher in patients with aldosterone producing adenomas.
Although there was a trend for a negative association in women, no statistically significant association was found between endogenous PTH and knee osteoarthritis.
The purpose of this study was to investigate the prevalence of adenomatous colon polyps (ACP) as they occur in subjects with diabetes mellitus (DM) and chronic kidney disease.
These data highlight the ability of PTH to phosphorylate beta-catenin (show CTNNB1 Antibodies) directly via PKA.
These data suggest that prostaglandin E2 acting via EP4R (show PTGER4 Antibodies) on bone marrow macrophages committed to the osteoblast cell lineage, stimulated secretion of a factor or factors that acted to suppress PTH-stimulated osteoblast differentiation.
These results suggest that in vivo PTH treatment increased in vitro osteoclastogenesis and resorption without altering the number of osteoclast precursors.
Osteoblast 2-deoxyglucose uptake and glycogen (show GYS2 Antibodies) synthesis were increased after exposure to low concentrations (0.1 nmol/l and above) of PTH.
Parathyroid hormone stimulated growth and decreased Col-X deposition via phosphotidylinositol-3,4,5 triphosphate kinase and mitogen activating protein kinase pathways in avian sterna.
likely involvement of the Sp family in regulating PTH gene expression through interactions with an Sp1 (show SP1 Antibodies) DNA element in the hormone's promoter.
alternative cis (show CISH Antibodies)-acting protein-binding elements may determine the regulation of PTH mRNA stability in response to changes in serum calcium and phosphate
a possible role for the Wnt (show WNT2 Antibodies) signaling pathway in PTH actions in bone
Data suggest that calcium-mediated destabilization of parathyroid hormone mRNA requires gene transcription and involves increases in cytosolic Ca.
Mmp13 (show MMP13 Antibodies) is selectively regulated of by 1,25-Dihydroxyvitamin D3, PTH, and Osterix (show SP7 Antibodies) through distal enhancers.
PTHrP (show PTHLH Antibodies) and PTH mediate wasting through a common mechanism involving PTHR (show PTH1R Antibodies).
Usp2 (show USP2 Antibodies) is required for the PTH1-34-induced proliferation of osteoblasts
These results highlight the role of distal enhancers in the regulation of RANKL (show TNFSF11 Antibodies) expression by PTH and perhaps 1,25(OH)2D3 and suggest that the RL-D2 and RL-D5 enhancers contribute in either an additive or synergistic manner to regulate bone remodeling.
These results indicate that PTH-mediated inhibition of renal phosphate transport involves phosphorylation of S77 of the NHERF-1 (show SLC9A3R1 Antibodies) PDZ (show INADL Antibodies) I domain and the dissociation of NHERF-1 (show SLC9A3R1 Antibodies)/Npt2a (show SLC34A1 Antibodies) complexes.
It was concluded that endogenously secreted PTH and GHR (show GHR Antibodies) signaling in bone are necessary to establish radial bone growth and optimize mineral acquisition during growth.
findings suggest that XLalphas (show GNAS Antibodies) enhances Gq/11 signaling to mediate the renal actions of PTH during early postnatal development.
bone adaptation during exercise is not only a function of dynamic loading, but also PTH release, and that PTH signaling contributes differently at the structural and tissue levels.
IGF1 (show IGF1 Antibodies) signaling plays a greater role in the skeletal actions of cPTH in the female mouse than in the male mouse, which may underlie the sex differences in the response to cPTH.
Downregulation of PTH receptor expression mediated by intracellular oxidant stress as the mechanisms in hyperlipidemia-induced PTH resistance.
The protein encoded by this gene is a hormone secreted by parathyroid cells. This hormone elevates blood Ca2+ level by dissolving the salts in bone and preventing their renal excretion. Defects in this gene are a cause of familial isolated hypoparathyroidism (FIH).
, parathyroid hormone 1
, preproparathyroid hormone
, hypothalamic parathyroid hormone
, thyroid hormone
, probable peptidyl-tRNA hydrolase