Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human OMG Antibodies:
anti-Mouse (Murine) OMG Antibodies:
anti-Rat (Rattus) OMG Antibodies:
Go to our pre-filtered search.
Human Polyclonal OMG Primary Antibody for ELISA, WB - ABIN268750
Zai, King, Wigg, Couto, Wong, Honer, Barr, Kennedy: Genetic study of the myelin oligodendrocyte glycoprotein (MOG) gene in schizophrenia. in Genes, brain, and behavior 2005
Rat (Rattus) Polyclonal OMG Primary Antibody for WB - ABIN1574230
Francosalinas, Cantaert, Nolte, Tak, van Lier, Baeten: Enhanced costimulation by CD70+ B cells aggravates experimental autoimmune encephalomyelitis in autoimmune mice. in Journal of neuroimmunology 2013
Human Polyclonal OMG Primary Antibody for IHC, IHC (p) - ABIN4341421
Chang, Susuki, Dours-Zimmermann, Zimmermann, Rasband: Oligodendrocyte myelin glycoprotein does not influence node of ranvier structure or assembly. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2010
Human Polyclonal OMG Primary Antibody for IF (p), IHC (p) - ABIN673284
Feng, Hao, Yang, Qu, Zheng, Liang, Jin et al.: Transplantation of mesenchymal stem cells promotes the functional recovery of the central nervous system following cerebral ischemia by inhibiting myelin-associated inhibitor expression and neural ... in Experimental and therapeutic medicine 2016
Human Polyclonal OMG Primary Antibody for ELISA, WB - ABIN250992
McGee, Yang, Fischer, Daw, Strittmatter: Experience-driven plasticity of visual cortex limited by myelin and Nogo receptor. in Science (New York, N.Y.) 2005
Retinal nerve fiber layer may be better preserved in MOG-IgG versus AQP4 (show AQP4 Antibodies)-IgG optic neuritis.
Data show that aquaporin-4 (AQP4) and myelin-oligodendrocyte glycoprotein (MOG) autoantibodies double positive neuromyelitis optica spectrum disorder (NMOSD) patients had a multiphase disease course with a high annual relapse rate.
MOG antibody associated optic neuritis tends to involve the anterior optic pathway.
This study demonstrated that increased expression of mRNA of OLIG1 (show OLIG1 Antibodies) in ventral prefrontal white matter in major depressive disorder.
In patients with idiopathic optic neuritis, 27.6% (8/29) were positive for MOG antibodies. Three of the eight MOG-positive patients showed significantly high CSF (show CSF2 Antibodies) levels of myelin basic protein (show MBP Antibodies). Five had optic pain and three had prodromal infection.
Immune modulation by a tolerogenic myelin oligodendrocyte glycoprotein (MOG)10-60 containing fusion protein in the marmoset experimental autoimmune encephalomyelitis model.
These data demonstrate a new role for myelin glycosylation in the control of immune homeostasis in the healthy human brain through the MOG-DC-SIGN (show CD209 Antibodies) homeostatic regulatory axis, which is comprised by inflammatory insults that affect glycosylation.
findings suggest that immune reactions toward MOG and in particular MOG-specific antibodies may play a functional role in multiple sclerosis
Patients with neuromyelitis optica spectrum disorders with MOG antibodies have distinct clinical features, fewer attacks, and better recovery than patients with AQP4 (show AQP4 Antibodies) antibodies or patients seronegative for both antibodies.
Bipolar I disorder and schizophrenia share a number of common genetic risk loci and susceptibility genes including the genes coding for myelin oligodendrocytes glycoprotein (MOG).
Identify nerve growth factor as a binding partner for MOG and demonstrate that this interaction is capable of sequestering NGF (show NGFB Antibodies) from TrkA (show NTRK1 Antibodies)-expressing neurons to modulate axon growth and survival.
Data indicate that in mixed chimeras with bone marrow-encoding myelin oligodendrocyte glycoprotein (MOG), the development of MOG-specific B cells was abrogated, resulting in a lack of MOG-specific B cells in all B cell compartments examined.
Identify immunogenic/encephalitogenic T-cell epitopes within MOG capable of inducing experimental autoimmune encephalomyelitis in C57BL/6 mice.
PLP significantly suppresses both models of experimental autoimmune encephalitis (EAE)even when there is some evidence of epitope spreading in the myelin oligodendrocyte (MOG)38-50-induced EAE model.
CNTF may play a role in the process of remyelination by inducing the MOG expression.
MOG-specific CD4 (show CD4 Antibodies)-positive T cells accumulate within the chemokine (show CCL1 Antibodies)-expressing draining lymph nodes, rather than within the brain or spinal cord during induction of autoimmune encephalomyelitis.
This study provides valuable information about requirements of anti-myelin oligodendrocyte glycoprotein reactivity for being regarded as a prognostic biomarker in a subtype of MS.
Mice immunized with encephalitogenic myelin oligodendrocyte glycoprotein MOG(35-55) peptide develop significant serum levels of anti-MOG antibodies in parallel with disease progression.
B cell-deficient mice exhibit reduced medullary thymic epithelial cell numbers and reduced MOG mRNA expression.
Cell surface targeting of MOG is not disrupted in the absence of the endoplasmic reticulum chaperones.
Cell adhesion molecule contributing to the interactive process required for myelination in the central nervous system.
, MOG alpha-5
, myelin-oligodendrocyte glycoprotein
, oligodendrocyte-myelin glycoprotein
, dystonia 2, torsion (autosomal recessive)
, myelin oligodendrocyte glycoprotein
, Myelin-oligodendrocyte glycoprotein
, myelin-oligodendrocyte glycoprotein-like
, MOG alpha-7Cj
, leucine-rich glycoprotein homolog
, oligodendrocyte myelin glycoprotein
, oligodendrocyte-myelin glycoprotein-like
, myelin-oligodendrocyte glycoprotein isoform beta1