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Browse our anti-DLL3 (DLL3) Antibodies

Full name:
anti-delta Like Protein 3 Antibodies (DLL3)
On are 81 delta Like Protein 3 (DLL3) Antibodies from 14 different suppliers available. Additionally we are shipping DLL3 Proteins (11) and DLL3 Kits (7) and many more products for this protein. A total of 103 DLL3 products are currently listed.
pu, pudgy, SCDO1

Most Popular Reactivities for anti-DLL3 (DLL3) Antibodies

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anti-Human DLL3 Antibodies:

anti-Mouse (Murine) DLL3 Antibodies:

anti-Rat (Rattus) DLL3 Antibodies:

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Top referenced anti-DLL3 Antibodies

  1. Human Polyclonal DLL3 Primary Antibody for EIA, IF - ABIN951923 : Heuss, Ndiaye-Lobry, Six, Israël, Logeat: The intracellular region of Notch ligands Dll1 and Dll3 regulates their trafficking and signaling activity. in Proceedings of the National Academy of Sciences of the United States of America 2008 (PubMed)
    Show all 3 references for ABIN951923

  2. Human Polyclonal DLL3 Primary Antibody for EIA, IF - ABIN951924 : Yerges, Klei, Cauley, Roeder, Kammerer, Moffett, Ensrud, Nestlerode, Marshall, Hoffman, Lewis, Lang, Barrett-Connor, Ferrell, Orwoll, Zmuda: High-density association study of 383 candidate genes for volumetric BMD at the femoral neck and lumbar spine among older men. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2009 (PubMed)
    Show all 3 references for ABIN951924

  3. Human Polyclonal DLL3 Primary Antibody for IF, IHC (p) - ABIN656327 : Maisenbacher, Han, Obrien, Tracy, Erol, Schaffer, Dormans, Zackai, Kusumi: Molecular analysis of congenital scoliosis: a candidate gene approach. in Human genetics 2005 (PubMed)
    Show all 3 references for ABIN656327

More Antibodies against DLL3 Interaction Partners

Human delta Like Protein 3 (DLL3) interaction partners

  1. DLL3 was silenced by methylation in human human hepatocellular carcinoma and it negatively regulates the growth of human hepatocellular carcinoma cells.

  2. We suggest that the three human DLL3 mutations associated with spondylocostal dysplasia are also functionally equivalent to the Dll3(neo) null allele in mice.

  3. mutations in DLL3 cause a consistent pattern of abnormal vertebral segmentation in spondylocostal dysostosis

  4. no novel or previously described mutations are present in our cohort, indicating that DLL3 mutations may not be a major cause of congenital scoliosis.

  5. The intracellular region of Notch (show NOTCH1 Antibodies) ligands Dll1 (show DLL1 Antibodies) and Dll3 regulates their trafficking and signaling activity

Mouse (Murine) delta Like Protein 3 (DLL3) interaction partners

  1. O-fucosylation of DLL3 is required for its function during somitogenesis.

  2. Intriguing changes are observed in the cranio-caudal (show CAD Antibodies) borders of multifidus muscle in mutant Dll3 and Lfng (show LFNG Antibodies) models of idiopathic scoliosis.

  3. Dll3 has a unique function during T-cell development that is distinct from the role played by the other DSL ligands of Notch (show NOTCH1 Antibodies).

  4. Dll3 targets Notch1 (show NOTCH1 Antibodies) for lysosomal degradation preventing Notch1 (show NOTCH1 Antibodies) from undergoing post-translational processing.

  5. Axial skeletal defects caused by mutation in the spondylocostal dysplasia/pudgy gene Dll3 are associated with disruption of the segmentation clock within the presomitic mesoderm.

  6. DLL3 knockout mice have segmentation and neural defects

  7. Notch (show NOTCH1 Antibodies) ligands, including Delta-like1 and 3 and Jagged1 (show JAG1 Antibodies) and Jagged2 (show JAG2 Antibodies), show distinct expression patterns in the developing and adult brain overlapping that of Notch1 (show NOTCH1 Antibodies)

  8. Data describe the genetic interactions between Dll1 (show DLL1 Antibodies), Dll3, Mesp2 (show Mesp2 Antibodies) and Psen1 (show PSEN1 Antibodies), and the roles of Dll1 (show DLL1 Antibodies)- and Dll3-Notch (show NOTCH1 Antibodies) pathways, with or without Psen1 (show PSEN1 Antibodies), in rostrocaudal patterning.

  9. spondylocostal dysostosis (SCD (show SCD Antibodies)) is caused by mutation in Delta-like (show DLK1 Antibodies) 3 (DLL3), Mesoderm posterior 2 (MESP2 (show Mesp2 Antibodies)), and Lunatic fringe (LFNG (show LFNG Antibodies)); three genes that are components of the Notch (show NOTCH1 Antibodies) signaling pathway.

  10. Data demonstrate distinct in vivo functions for DLL1 (show DLL1 Antibodies) and DLL3, and suggest that DLL3 does not antagonize DLL1 (show DLL1 Antibodies) in the presomitic mesoderm.

DLL3 Antigen Profile

Antigen Summary

This gene encodes a member of the delta protein ligand family. This family functions as Notch ligands that are characterized by a DSL domain, EGF repeats, and a transmembrane domain. Mutations in this gene cause autosomal recessive spondylocostal dysostosis 1. Two transcript variants encoding distinct isoforms have been identified for this gene.

Alternative names and synonyms associated with DLL3

  • delta-like 3 (Drosophila) (DLL3) antibody
  • delta-like 3 (Drosophila) (Dll3) antibody
  • pu antibody
  • pudgy antibody
  • SCDO1 antibody

Protein level used designations for DLL3

delta-like protein 3 , delta3 , drosophila Delta homolog 3 , M-Delta-3

10683 Homo sapiens
484508 Canis lupus familiaris
505993 Bos taurus
13389 Mus musculus
114125 Rattus norvegicus
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