Browse our KCNJ5 (KCNJ5) ELISA Kits

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Potassium Inwardly-Rectifying Channel, Subfamily J, Member 5 ELISA Kits (KCNJ5)
On are 5 Potassium Inwardly-Rectifying Channel, Subfamily J, Member 5 (KCNJ5) ELISA Kits from 4 different suppliers available. Additionally we are shipping KCNJ5 Antibodies (52) and KCNJ5 Proteins (9) and many more products for this protein. A total of 73 KCNJ5 products are currently listed.
cir, GIRK4, KATP1, Kir3.4, LQT13, xcir
list all ELISA KIts Gene Name GeneID UniProt
Human KCNJ5 KCNJ5 9541 Q86X95
Mouse KCNJ5 KCNJ5 16521 P48545
KCNJ5 29713 P48548

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Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 5 (KCNJ5) interaction partners

  1. KCNJ5 mutations predominate in large zona fasciculata (ZF)-like Aldosterone-producing Adenomas.

  2. Mutations in KCNJ5 cause the excessive autonomous aldosterone secretion of Aldosterone-producing Adenomas.

  3. KCNJ5 genetic mutation plays a role in the development of primary aldosteronism in aldosterone producing adenomas.

  4. Study provides new evidence, indicating that some glutamate receptor ionotropic kainate 4 (show GRIK4 ELISA Kits) variants modulate the response to electroconvulsive therapy in patients with depression resistant to treatment, suggesting a role for kainate receptor modulation.

  5. documented for the first time the expression of inflammation-related genes in aldosterone-producing adenomas (APAs) and the correlation of their expression levels with the KCNJ5 mutation status and mRNA expression levels of steroidogenic enzymes, indicating the pathophysiological relevance of inflammation-related genes in APAs

  6. GIRK1 (show KCNJ3 ELISA Kits)/GIRK4 hetero-tetramers are not activated by Na+, but rather are in a permanent state of high responsiveness to G proteins beta-gamma, suggesting that the GIRK1 (show KCNJ3 ELISA Kits) subunit functions like a GIRK4 subunit with Na+ permanently bound.

  7. japanese Aldosterone-Producing Adenoma patients may have distinct features including a higher prevalence of KCNJ5 mutations, no gender difference in the frequency of these mutations, and characteristics similar to the zona glomerulosa.

  8. Novel somatic KCNJ5 variants likely cause adenomas by loss of potassium selectivity, similar to previously described mutations.

  9. KCNJ5 mutations in aldosterone-producing adenomas are more frequent in women; however, this gender dimorphism is a reported phenomenon of Western but not East Asian populations (review).

  10. The present study demonstrated the high prevalence of somatic KCNJ5 mutations in Korean patients with aldosterone-secreting adenoma. Carriers of somatic KCNJ5 mutations were more likely to be female.

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 5 (KCNJ5) interaction partners

  1. These results provide a novel molecular mechanism for autocrine negative feedback regulation of insulin (show INS ELISA Kits) secretion.

  2. study establishes the role of f-channels in cardiac automaticity and indicates that arrhythmia related to HCN loss-of-function may be managed by pharmacological or genetic inhibition of GIRK4 channels

  3. Data indicate taht m2R-RGS6 (show RGS6 ELISA Kits)-IKACh pathway sets heart rate variability independently from the autonomic input.

  4. Therefore, the lack of proper functioning of the cardio-protective K(ATP) system in the mdx (show DMD ELISA Kits) cardiomyocytes may be part of the mechanism contributing to development of cardiac disease in dystrophic patients.

  5. Data suggest HL-1 (show ASGR1 ELISA Kits) cells express GIRK1 (show KCNJ3 ELISA Kits)/4 and M2 muscarinic receptors and are a good model to study acetylcholine-activated potassium currents.

  6. Data show that the composition of the Kir3.1 (show KCNJ3 ELISA Kits) and Kir (show GEM ELISA Kits) 3.4 subunits of the G protein-gated potassium channel (show KCNAB2 ELISA Kits) changes during embryonic development.

  7. These data implicate GIRK4-containing channels in signaling crucial to energy homeostasis and body weight.

Pig (Porcine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 5 (KCNJ5) interaction partners

  1. Blockade of K(ATP) channels further diminished (approximately 45%) the repayment of flow debt (show PLXNB2 ELISA Kits) in lean but not metabolic syndrome swine.

KCNJ5 Antigen Profile

Antigen Summary

Potassium channels are present in most mammalian cells, where they participate in a wide range of physiologic responses. The protein encoded by this gene is an integral membrane protein and inward-rectifier type potassium channel. The encoded protein, which has a greater tendency to allow potassium to flow into a cell rather than out of a cell, is controlled by G-proteins. It may associate with two other G-protein-activated potassium channels to form a heteromultimeric pore-forming complex.

Alternative names and synonyms associated with KCNJ5

  • corepressor interacting with RBPJ, 1 (CIR1) Elisa Kit
  • corepressor interacting with RBPJ, 1 (cir1) Elisa Kit
  • potassium inwardly-rectifying channel, subfamily J, member 5 (KCNJ5) Elisa Kit
  • potassium inwardly-rectifying channel, subfamily J, member 5 (Kcnj5) Elisa Kit
  • cir Elisa Kit
  • GIRK4 Elisa Kit
  • KATP1 Elisa Kit
  • Kir3.4 Elisa Kit
  • LQT13 Elisa Kit
  • xcir Elisa Kit

Protein level used designations for KCNJ5

CBF1 interacting corepressor , CBF1-interacting corepressor , corepressor interacting with RBPJ 1 , recepin , G protein-activated inward rectifier potassium channel 4 , IRK-4 , cardiac ATP-sensitive potassium channel , heart KATP channel , inward rectifier K+ channel KIR3.4 , CIR , GIRK-4 , KATP-1 , cardiac inward rectifier , inward rectifier K(+) channel Kir3.4 , potassium channel, inwardly rectifying subfamily J member 5 , GIRK4 , inward rectifying K channel , potassium inwardly-rectifying channel J5

9541 Homo sapiens
446936 Xenopus laevis
426529 Gallus gallus
3762 Homo sapiens
16521 Mus musculus
29713 Rattus norvegicus
395925 Gallus gallus
489284 Canis lupus familiaris
397448 Sus scrofa
100352473 Oryctolagus cuniculus
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