Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
ARR18 interaction negatively interferes with the transcriptional activity of bZIP63 on the PDH1 promoter. [PDH1]
ProDH potentiates the oxidative burst and cell death associated with the hypersensitive response.
ProDH-silenced plants displayed reduced ROS (show ROS1 ELISA Kits) and cell death levels as well as enhanced susceptibility in response to avirulent pathogens.
bZIP11 target processes are identified using transiently increased nuclear bZIP11 levels and genome-wide expression analysis; the effect of bZIP11 on ASN1 (show ASNS ELISA Kits) and GBF6 are reported.
PRODH/POX and succinate regulated mitochondrial respiration.
PRODH/POX knockdown decreased DNA and collagen biosynthesis, whereas increased prolidase (show PEPD ELISA Kits) activity and intracellular proline level in MCF-7shPRODH/POX cells.
this study shows that PRODH plays a causative role in DNA damage-induced senescence through the enzymatic generation of reactive oxygen species
the frequency of a recurrent small 22q11.2 deletion encompassing PRODH and the neighboring DGCR6 (show DGCR6 ELISA Kits) gene in three case-control studies, was studied.
The findings support a major role for the PRODH 757TT, 1766GG, and 1852AA genotypes alone and in combination for schizophrenia susceptibility.
Thirty-five percent of the subjects were hyperprolinemic, allele carriers of PRODH rs450046 had a lower full-scale intelligence compared to T allele carriers
GR and KLF15 (show KLF15 ELISA Kits) physically interact via low affinity GR binding sites within glucocorticoid response elements (GREs) for PRODH and AASS (show AASS ELISA Kits) that contribute to combinatorial regulation with KLF15 (show KLF15 ELISA Kits).
results suggest that PRODH and COMT may interact to contribute to the ASD phenotype in individuals with VCFS
Functional COMT (show COMT ELISA Kits), but not PRODH, variant affects IQ and executive functions in 22q11.2DS subjects during neurodevelopment with a maximal effect at adulthood
Data indicate that a functional proline dehydrogenase (PRODH) variant associated with schizophrenia that may have a neurochemical impact, altering brain function, but is not responsible for the cortical reductions found in the disorder.
The current study demonstrates that sensory gating impairments that are typical of schizophrenia are found in 22q11.2DS subjects. Our results further suggest that COMT (show COMT ELISA Kits) and PRODH genetic variations contribute to sensory gating.
This gene encodes a mitochondrial protein that catalyzes the first step in proline degradation. Mutations in this gene are associated with hyperprolinemia type 1 and susceptibility to schizophrenia 4 (SCZD4). This gene is located on chromosome 22q11.21, a region which has also been associated with the contiguous gene deletion syndromes, DiGeorge and CATCH22. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
proline dehydrogenase (oxidase) 1
, Proline dehydrogenase (Proline oxidase)
, proline dehydrogenase
, proline dehydrogenase, mitochondrial-like
, proline dehydrogenase 1, mitochondrial
, proline dehydrogenase, mitochondrial
, proline oxidase
, p53-induced gene 6 protein
, proline oxidase 2
, proline oxidase, mitochondrial
, tumor protein p53 inducible protein 6
, proline dehydrogenase (proline oxidase)