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anti-Human Epiregulin Antibodies:
anti-Mouse (Murine) Epiregulin Antibodies:
anti-Rat (Rattus) Epiregulin Antibodies:
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three-dimensional structure of the EPR antibody (the 9E5(Fab (show FANCB Antibodies)) fragment) in the presence and absence of EPR
Together, these studies lead to identification of a novel pathway involving EREG and MMP-1 (show MMP1 Antibodies) that contributes to the formation of early stage breast cancer
These results suggested that EREG is one of the molecules involved in glioma malignancy
Data indicate that the effects of epiregulin (EREG) and V-ATPase (show ATP6V1H Antibodies) (TCIRG1 (show TCIRG1 Antibodies)) single nucleotide polymorphism (SNP) on pulmonary tuberculosis susceptibility, to the extent that they exist, are dependent on gene-gene interactions in West African populations.
Patients homozygous for the minor allele A of EREG rs12641042 had a significantly higher 3-year survival rate than patients with allele C (HR 0.48; P=0.034), but significance was lost in multivariable analysis
Epiregulin is a transcriptional target of TSC2 (tuberin (show TSC2 Antibodies)).
Epiregulin promotes the proliferation of liver progenitor cells and DNA synthesis by hepatocytes and is upregulated in the serum of patients with liver injury.
Plasma HGF and EREG levels are associated with resistance to treatment with anti-EGFR antibodies in KRAS wild-type patients with metastatic colorectal cancer.
Data suggest that EREG (epiregulin), AREG (amphiregulin (show AREG Antibodies)), and BTC (betacellulin (show BTC Antibodies)) induced prostaglandin E2 production by induction of COX-2 (prostaglandin-endoperoxide synthase 2 (show PTGS2 Antibodies)) through MAP kinase (show MAPK1 Antibodies) signaling in granulosa cells.
In pre-treated K-ras (show HRAS Antibodies) wild-type status colorectal cancer, patients with high EREG gene expression appear to benefit more from cetuximab therapy compared with low expression.
Pre-maturation with cAMP modulators in conjunction with EGF (show EGF Antibodies)-like peptides during in vitro maturation enhances mouse oocyte developmental competence.
EREG and tumor-associated fibroblasts play a crucial role in controlling tumor growth in colitis-associated neoplasms.
Epiregulin can effectively mature isolated cumulus-oocyte complexes, but fails as a substitute for the hCG (show CGA Antibodies)/epidermal growth factor (show EGF Antibodies) stimulus on cultured follicles.
This study implicates that EREG mediates signals downstream of Areg (show AREG Antibodies) mRNA expression and that EGFR (show EGFR Antibodies)-ERBB2 (show ERBB2 Antibodies) signals contributes to regulation of ovulation process.
A TLR-->MyD88 (show MYD88 Antibodies)-->AREG (show AREG Antibodies)/EREG-->EGFR (show EGFR Antibodies) signaling pathway is represented in nonhematopoietic cells of the intestinal tract, responds to microbial stimuli once barriers are breached, and mediates protection against dextran sulfate sodium-induced colitis.
epidermal growth factor (EGF (show EGF Antibodies)) family members amphiregulin (show AREG Antibodies), epiregulin, and beta-cellulin are paracrine mediators that propagate the LH signal throughout the ovulatory follicle
Epiregulin plays a critical role in immune/inflammatory-related responses of keratinocytes and macrophages.
Data suggest that epiregulin (Ereg) deficiency does not alter intestinal cancer susceptibility, but that Ereg-null mice are highly susceptible to cancer-predisposing intestinal damage caused by oral dextran sulfate sodium.
Epiregulin is a member of the epidermal growth factor family. Epiregulin can function as a ligand of EGFR (epidermal growth factor receptor), as well as a ligand of most members of the ERBB (v-erb-b2 oncogene homolog) family of tyrosine-kinase receptors.