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Human VEGF ELISA Kit for Sandwich ELISA - ABIN625107
Kong, Bai, Nan, Sun, Chen, Qi: Pleiotrophin is a potential colorectal cancer prognostic factor that promotes VEGF expression and induces angiogenesis in colorectal cancer. in International journal of colorectal disease 2011
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Mouse (Murine) VEGF ELISA Kit for Sandwich ELISA - ABIN625186
Yamamoto, Fujita, Tanaka, Kojima, Kanatani, Ishihara, Tachibana: Low oxygen tension enhances proliferation and maintains stemness of adipose tissue-derived stromal cells. in BioResearch open access 2013
Show all 28 Pubmed References
Human VEGF ELISA Kit for Sandwich ELISA - ABIN414845
Nourshahi, Hedayati, Ranjbar: The correlation between resting serum leptin and serum angiogenic indices at rest and after submaximal exercise. in Regulatory peptides 2011
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Rat (Rattus) VEGF ELISA Kit for Sandwich ELISA - ABIN625218
Hao, Huang, Shi, Cheng, Li, Zhou, Guo, Li, Lu, Zhu, Duan: Pulsed electromagnetic field improves cardiac function in response to myocardial infarction. in American journal of translational research 2014
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Human VEGF ELISA Kit for Sandwich ELISA - ABIN365219
Tuo, Guo, Zhang, Wang, Zhou, Xia, Zhang, Wen, Jin: The biological effects and mechanisms of calcitonin gene-related peptide on human endothelial cell. in Journal of receptor and signal transduction research 2013
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Mouse (Murine) VEGF ELISA Kit for Sandwich ELISA - ABIN424307
Obrador, Benlloch, Pellicer, Asensi, Estrela: Intertissue flow of glutathione (GSH) as a tumor growth-promoting mechanism: interleukin 6 induces GSH release from hepatocytes in metastatic B16 melanoma-bearing mice. in The Journal of biological chemistry 2011
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Rat (Rattus) VEGF ELISA Kit for Sandwich ELISA - ABIN625219
Cohen, Gitay-Goren, Neufeld, Levi: High levels of biologically active vascular endothelial growth factor (VEGF) are produced by the baculovirus expression system. in Growth factors (Chur, Switzerland) 1992
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Mouse (Murine) VEGF ELISA Kit for Sandwich ELISA - ABIN365363
Cheng, Hu, Lv, Ling, Jiang: Erythropoietin improves the efficiency of endothelial progenitor cell therapy after myocardial infarction in mice: effects on transplanted cell survival and autologous endothelial progenitor cell mobilization. in The Journal of surgical research 2012
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Chicken VEGF ELISA Kit for Sandwich ELISA - ABIN416595
Jeffreys, Wilson, Thein: Hypervariable 'minisatellite' regions in human DNA. in Nature 1985
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Rat (Rattus) VEGF ELISA Kit for Sandwich ELISA - ABIN416260
Guo, Gao, Lin, Wu, Huang, Dong, Chen, Lu, Fu, Wang, Ma, Chen, Wu, He, Yang, Liao, Zheng, Tan: BMSCs reduce rat granulosa cell apoptosis induced by cisplatin and perimenopause. in BMC cell biology 2014
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Rspo1 (show RSPO1 ELISA Kits) is required for hematopoietic stem cell specification through control of parallel signaling pathways controlling HSC (show FUT1 ELISA Kits) specification: Wnt16 (show WNT16 ELISA Kits)/DeltaC/DeltaD and Vegfa/Tgfbeta1 (show TGFB1 ELISA Kits)
Tmem2 regulates hyaluronic acid turnover to promote normal Vegf signaling during developmental angiogenesis.
low levels of Vegf signaling promote overall vascular endothelial differentiation and cell survival by upregulating etv2 expression, while high levels of Vegf signaling promote arterial and inhibit venous specification.
altering the amount of VEGF signaling in endothelial cells by stimulating them with different VEGF concentrations triggered distinct and mutually exclusive dynamic Ca(2 (show CA2 ELISA Kits)+) signaling responses that correlated with different cellular behaviors.
Bis(2,3-dibromo-4,5-dihydroxybenzyl) ether downregulate the expression of VEGF and VEGFR, and simultaneously regulates VEGF signaling pathway to prevent angiogenesis.
noncanonical function of tars (show TARS ELISA Kits) regulates vascular development presumably by modulating the expression of vegfa
Methylglyoxal acts on smaller blood vessels in zebrafish via the VEGF receptor (show FLT1 ELISA Kits) signaling cascade, thereby describing a new mechanism that can explain vascular complications under hyperglycemia and elevated MG concentrations.
The translation initiation factor eIF3i (show EIF3I ELISA Kits) up-regulates VEGF-A, accelerates cell proliferation, and promotes angiogenesis in embryonic development.
lack of SULF1 (show SULF1 ELISA Kits) expression downregulates VEGFA-mediated arterial marker expression, confirming that Sulf1 (show SULF1 ELISA Kits) mediates arterial specification by regulating VegfA165 activity.
yolk-derived 17beta-estradiol sets the ventral boundary of hemogenic vascular niche specification by antagonizing the dorsal-ventral regulatory limits of VEGF.
VEGFA produced in the somites is required to initiate adult haemangioblast programming in the adjacent dorsal lateral plate mesoderm.
sequential, isoform-specific VEGFA signaling successively induces the endothelial, arterial, and hematopoietic stem cell programs in the dorsal aorta.
VEGF induces stromal cell migration or recruitment that are required for blood vessel formation.
increased VEGF(170) levels disturb Hand-1 (show HAND1 ELISA Kits) expression in the region required for normal heart morphogenesis
data for the first time demonstrate a calpain/PTP1B/VEGFR2 negative feedback loop in the regulation of VEGF-induced angiogenesis. Modulation of local PTP1B and/or calpain activities may prove beneficial in the treatment of impaired wound healing in diabetes.
Studied the effect of cyclic uniaxial stretch (20%, 1 Hz), with and without the stimulation of vascular endothelial growth factor (VEGF), on sprouting angiogenesis by employing a stretchable three-dimensional cell culture model.
Study shows that VEGFA mRNA in mammalian endothelial cells undergoes programmed translational readthrough (PTR) generating VEGF-Ax, an isoform containing a unique 22-amino-acid C terminus extension.
Data indicate that superoxide dismutase (SOD) inhibited high glucose (HG)-induced expression of uPAR and VEGF in bovine retinal microvascular endothelial cell (REC).
Exposure of endothelial cells to VEGF, high glucose, or hydrogen peroxide up-regulated the XBP1 (show XBP1 ELISA Kits)/IRE1 alpha (show ERN1 ELISA Kits) and ATF6 (show ATF6 ELISA Kits) arms of the unfolded protein response compared with untreated cells.
Microvascular endothelial cells are more susceptible than aortic cells to advanced glycation end products-enhanced permeability and that AGE-enhanced permeability is dependent on VEGF expression induced by reactive oxygen species.
VEGF supports germ cell survival and sperm production in bulls.
analysis of polymorphisms of bovine VEGF gene and their associations with growth traits in Chinese cattle
VEGF mRNA expression at estrus was higher than at the early I, early II and late luteal stages (P<0.05), whereas VEGF protein content was greatest at the early I luteal stage and decreased thereafter
Alterations in the expression of VEGF-A and bFGF (show FGF2 ELISA Kits) systems suggest that angiogenic factors are involved in abnormal placental development in cloned gestations, contributing to impaired fetal development and poor survival rates.
Corticotrophin-releasing hormone accelerated tumor angiogenesis by upregulating VEGF expression and secretion in colon cancer cells.
In multivariate analysis, only serum VEGF-A correlated to diabetes duration, whereas VEGF-C (show VEGFC ELISA Kits) only correlated to HbA1c and fasting blood glucose.
The study aimed to assess the usefulness of the determination of cytokines: IL-8, VEGF and its soluble receptors: VEGF-R1, VEGF-R2 in patients with endometrial cancer. The increased levels of VEGF may be useful in the preoperative assessment of the status of para-aortic lymph nodes.
It is likely that these neuronal factors, under the influence of estrogen, could induce physiological inflammation during cervical remodeling by promoting the expression of vascular endothelial growth factor, among other factors
Our results reveal that aflibercept significantly lowers the amount of unbound VEGF as well as the proliferation rate of HUVEC. Moreover, in contrast to specifications given by the manufacturer, aflibercept retains its full inhibitory effect up to at least 120h after transference from the original vial into the injection syringe.
Since VEGF contributes to disturbed vasculature in chronic obstructive pulmonary disease (COPD (show ARCN1 ELISA Kits)), altered miR (show MLXIP ELISA Kits)-503 production might play a role in modulating fibroblast-mediated vascular homeostasis in COPD (show ARCN1 ELISA Kits).
results showed that fascaplysin inhibited ovarian cancer cell proliferation, invasion and migration, as well as inducing S arrest and cell apoptosis. Treatment with fascaplysin also suppressed CDK4 (show CDK4 ELISA Kits), cyclin D1 (show CCND1 ELISA Kits), Bcl-2 (show BCL2 ELISA Kits), and VEGFA expression at protein levels
Patients with mixed dyslipidaemia have higher plasma VEGF levels. Rosuvastatin monotherapy at high dose reduces VEGF values, whereas a significant increase is observed in patients receiving the combination of low rosuvastatin dose with omega-3 PUFAs.
The variation in physical adaptation to exercise training in diabetes mellitus type 2 was associated with changes in expression of vascular endothelial growth factor A in muscle.
Serum VEGF is a useful marker for prediction of ovarian cancer microvessel density and tumor VEGF expression.
VEGF protein levels were also higher in the ipsilateral hemisphere of WT mice compared to Par-1 (show MARK2 ELISA Kits) KO mice after glioma cell implantation.
the importance of VEGF derived from tumor-infiltrating myeloid cells for initiating vascularization in gliomas
Low VEGF expression is associated with liver fibrosis.
We conclude that HIF-1 (show HIF1A ELISA Kits) is not a major regulator of Vegfa expression during wound healing; rather, it serves to maintain basal levels of expression of Vegfa and its target genes in intact skin, which are required for optimal granulation tissue formation in response to wounding.
Mechanical strain stimulates vasculogenesis of embryonic stem cells by the intracellular messengers ROS (show ROS1 ELISA Kits), NO and calcium as well as by upregulation of angiogenesis guidance molecules and the angiogenic growth factors VEGF, FGF-2 (show FGF2 ELISA Kits) and PDGF (show PDGFA ELISA Kits)-BB.
This study identifies YAP/TAZ as central mediators of VEGF signaling
Ectopic midline vascularisation in endothelial Nrp1 (show NRP1 ELISA Kits) and Vegfa(188/188) mutants caused additional axonal exclusion zones within the chiasm.
RUNX1T1 (show RUNX1T1 ELISA Kits) serves as a common angiogenic driver for vaculogenesis and functionality of endothelial lineage cells
VEGF inhibition decreases local CFH (show CFH ELISA Kits) and other complement regulators in the eye and kidney through reduced VEGFR2 (show KDR ELISA Kits)/PKC-alpha (show PKCa ELISA Kits)/CREB (show CREB1 ELISA Kits) signaling.
Vascular endothelial growth factor (VEFG)-A, originally described as an angiogenic factor, belongs to a super-family of glycoproteins, and signals through tyrosine kinase (show TYRO3 ELISA Kits) receptors VEGF receptor (show FLT1 ELISA Kits) 1 (VEGFR1 (show FLT1 ELISA Kits)) and VEGF receptor 2 (VEGFR2) and coreceptors, neuropilin (NP) 1 (show NRP1 ELISA Kits) and 2.
It was concluded that VEGF and factor VIII are important growth factors associated with fetal development in pigs and are identified in all uterine segments.
results are consistent with a participation of (VEFG) in the regulation of the dynamics of oviductal fluid secretion and the oviduct contractibility
Here we demonstrate that VEGF-165 mediates MSC (show MSC ELISA Kits) differentiation into ECs via VEGFR-2 (show KDR ELISA Kits)-dependent induction of Sox18 (show SOX18 ELISA Kits), which ultimately coordinates the transcriptional upregulation of specific markers of the EC phenotype.
VEGF was significantly downregulated 7 days after cryotherapy of the sclera and stayed at that level until day 14. It returned to baseline by day 21.
VEGF production, blood vessel network and follicle remodeling are impaired by the antiprogesterone RU486.
findings suggest that the augmented neovascular response seen with VEGF administration was through the VEGF-induced upregulation of Notch (show NOTCH1 ELISA Kits) signaling.
interleukin-1beta-induced vascular endothelial growth factor in airway smooth muscle cells
Upregulation of VEGF during hypoxia in chondrocyte is mediated partially through HIF-1alpha (show HIF1A ELISA Kits).
data shows that members of the VEGF-VEGFR (show KDR ELISA Kits) system are temporally and spatially well localized for playing key roles during umbilical cord formation and its intensive growth observed after day 75 of pregnancy
cardiac-specific and hypoxia-induced coexpression of VEGF and Ang1 (show ANGPT1 ELISA Kits) improves the perfusion and function of porcine MI heart through the induction of angiogenesis and cardiomyocyte proliferation
Peripheral Blood-Derived Mesenchymal Stromal Cells Promote Angiogenesis via Paracrine Stimulation of Vascular Endothelial Growth Factor Secretion
TNF may up-regulate VEGF and stimulate angiogenesis in the mare early corpus luteum.
After acoustic trauma, vascular endothelial growth factor was up-regulated both in the cochlea and vestibule. Expression in both structures suggests a reparative role with potentially therapeutic implications.
Studied the role of T help 17 cells (Th17) and STAT3-VEGF pathway in pathogenesis of psoriasis.
VEGF mRNA abundance was present at low levels at 16 h post-ovulation and remained low at 72 h, but the level increased at 144 h in uterus.
correlation was observed between CT perfusion parameters (BF, BV, PS, and MAV) and expression of VEGF and MVD (show MVD ELISA Kits) in tumor tissue
Physiologic ischemic training stimulates VEGF-mediated mobilization of endothelial progenitor cells as well as angiogenesis.
Suggest supplemental oxygen inhibits HIF-1alpha/VEGF signaling to reduce smooth muscle proliferation in rabbit arteriovenous fistula model.
Therefore, it was reasonable to speculate that the increased expression of VEGF and MMP-9 (show MMP9 ELISA Kits) in residual hepatic tumor cells and tumor angiogenesis post-embolization would be responsible for the increased metastatic potentiality and invasiveness.
VEGF expression peaked at 8 weeks after meniscus transplantation
Studied the biocompatibility and neovascularization of the PLGA nanospheres wrapped with vascular endothelial growth factor (VEGF).
VEGF induces TGF-beta1 (show TGFB1 ELISA Kits) expression and myofibroblast transformation after glaucoma surgery.
The results of this study are promising concerning the role of VEGF as a diagnostic marker in moderate osteoarthritis
The overexpression of VEGF increased tumor growth and vascularization, favored cyst formation, and reduced tumor necrosis.
VEGF plays an important role in choroid-retinal endothelial cell proliferation and tube formation.
These findings suggest that FBLN5 (show FBLN5 ELISA Kits) may interfere with choroidal neovascularization by downregulating VEGF, CXCR4 (show CXCR4 ELISA Kits), and TGFB1 (show TGFB1 ELISA Kits) expression and inhibiting choroidl endothelial cell proliferation.
Apelin may play a role in the development of central retinal vein occlusion (CRVO). Apelin has a unique upstream signaling pathway, independent of the VEGF pathway.
Dll4 play an important role in choroidal neovascularization (CNV) angiogenesis, which appears to be regulated by HIF-1alpha and VEGF during the progression of CNV under hypoxic conditions.
A functional network involving VEGF, IGF2, and MMP9 (show MMP9 ELISA Kits) in early placental trophoblast cells and maternal endometrium appears to be important for normal placentation.
This gene is a member of the PDGF/VEGF growth factor family and encodes a protein that is often found as a disulfide linked homodimer. This protein is a glycosylated mitogen that specifically acts on endothelial cells and has various effects, including mediating increased vascular permeability, inducing angiogenesis, vasculogenesis and endothelial cell growth, promoting cell migration, and inhibiting apoptosis. Elevated levels of this protein is linked to POEMS syndrome, also known as Crow-Fukase syndrome. Mutations in this gene have been associated with proliferative and nonproliferative diabetic retinopathy. Alternatively spliced transcript variants, encoding either freely secreted or cell-associated isoforms, have been characterized. There is also evidence for the use of non-AUG (CUG) translation initiation sites upstream of, and in-frame with the first AUG, leading to additional isoforms.
, vascular endothelial growth factor A
, vascular endothelial growth factor A-A
, vascular endothelial growth factor A-a
, vascular endothelial growth factor 164
, vascular permeability factor
, vascular endothelial growth factor 188
, angiogenic factor
, Vascular permeability factor
, endothelial growth factor-164
, vascular endothelial growth factor VEGF