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Cow (Bovine) Polyclonal MLZE Primary Antibody for IHC, WB - ABIN2777545
Norquay, Yang, Sheppard, Gregoire, Dodd, Reith, Cattini: RFX1 and NF-1 associate with P sequences of the human growth hormone locus in pituitary chromatin. in Molecular endocrinology (Baltimore, Md.) 2003
Human Polyclonal MLZE Primary Antibody for ICC, IF - ABIN2668773
Betancourt, Katzman, Chen: Nuclear factor one B regulates neural stem cell differentiation and axonal projection of corticofugal neurons. in The Journal of comparative neurology 2014
Human Polyclonal MLZE Primary Antibody for IHC, IHC (p) - ABIN4339338
Taglietti, Maroli, Cermenati, Monteverde, Ferrante, Rossi, Cossu, Beltrame, Messina: Nfix Induces a Switch in Sox6 Transcriptional Activity to Regulate MyHC-I Expression in Fetal Muscle. in Cell reports 2016
Nfix deficiency delays intermediate progenitor cell production and prolongs the neurogenic window, resulting in an increased number of neurons in the postnatal forebrain.
Nfix is required for maintaining muscle physiology postnatally and for the proper timing of muscle regeneration.
The data indicate that NFIX influences the lineage specification of postnatal subventricular zone neural stem cells, specifically suppressing oligodendrogenesis.
The ependymal layer of the lateral ventricles was frequently absent in Nfix(-/-) mice
NFIX acts as a regulator of lineage specification in the haematopoietic system.
NFIX plays a central role in hippocampal morphogenesis, regulating the formation of neuronal and glial populations within this
Bbx (show BBX Antibodies) is a downstream target of NFIX during development of the forebrain.
The findings demonstrate that the level of Nfix expression during development and within the adult is essential for the function of the hippocampus during learning and memory.
Nfix is a novel regulator of murine hematopoietic stem and progenitor cell survival.
NFIX is robustly induced when neural stem cells enter quiescence
Plasma miR (show MLXIP Antibodies)-1914* and -1915 interact with NFIX RNA.
Report novel mutations of NFIX gene causing Marshall-Smith syndrome or Sotos (show NSD1 Antibodies)-like syndrome.
miR (show MLXIP Antibodies)-1290 functions as a tumor oncogene (show RAB1A Antibodies) in the progression of esophageal squamous cell carcinoma by targeting NFIX
NFIX analysis should be considered in patients presenting with overgrowth, macrocephaly and developmental delay including those in whom Sotos (show NSD1 Antibodies) syndrome has been considered clinically but are negative for pathogenic NSD1 (show NSD1 Antibodies) variants.
TGF-beta (show TGFB1 Antibodies)-mediated suppression of ANT2 (show SLC25A5 Antibodies) through NF1 (show NF1 Antibodies)/Smad4 (show SMAD4 Antibodies) complexes contributes to oxidative stress and DNA damage during induction of cellular senescence.
Deletions in the 3' part of the NFIX gene including a recurrent Alu-mediated deletion of exon 6 and 7 account for previously unexplained cases of Marshall-Smith syndrome.
missense mutations in NFIX were able to cause Sotos (show NSD1 Antibodies)-like features.
DNA methylation (show HELLS Antibodies) shows genome-wide association of NFIX, RAPGEF2 (show RAPGEF2 Antibodies) and MSRB3 (show MSRB3 Antibodies) with gestational age at birth.
NFI (show NFIC Antibodies)-X3 and STAT3 (show STAT3 Antibodies) control the migration of differentiating astrocytes as well as migration and invasion of glioma cells via regulating YKL-40 (show CHI3L1 Antibodies) expression.
NFI-X3 activates GFAP expression, in part, by inducing alterations in the nucleosome architecture that lead to the increased recruitment of RNA polymerase II
NFIX had highest expression in brain; medial expression in lung and muscle; and lower expression in spleen, kidney, liver and heart of both embryo and adult cattle. However, expression levels in adult tissues were significantly decreased.
Two sequence variants of the Nfix gene are significantly associated with growth traits at different ages.
During vertebrate evolution, the transcription factor Nfix lost some specific functions, probably as a consequence of the different environment in which teleosts and mammals develop.
CCAAT box binding transcription factor (show NFIA Antibodies) (NF-Y) B (show NFYB Antibodies) is a critical modulator of neural crest cell's gene expression governing embryonic cartilage growth
The protein encoded by this gene is a transcription factor that binds the palindromic sequence 5'-TTGGCNNNNNGCCAA-3 in viral and cellular promoters. The encoded protein can also stimulate adenovirus replication in vitro. Three transcript variants encoding different isoforms have been found for this gene.
CCAAT-box-binding transcription factor
, TGGCA-binding protein
, nuclear factor 1 X-type
, nuclear factor 1/X
, NF1-X1 protein
, nuclear factor I-X protein
, Nuclear factor 1/X