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Mouse (Murine) WNT10A ELISA Kit for Sandwich ELISA - ABIN837288
Zhang, Hao, Meng, Liu, Zhao, Hu, Ding, Wang, He, Pan, Fang, Chen, Hu, Jia: Digital gene expression tag profiling analysis of the gene expression patterns regulating the early stage of mouse spermatogenesis. in PLoS ONE 2013
Wnt10a regulates proliferation and apoptosis of embryonic palatal mesenchymal cells at least partially through the canonical Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling pathway.
Data indicate that that Wnt10a regulates Dspp (show DSPP ELISA Kits) expression in mesenchymal cells.
Histone methyltransferase G9a (show EHMT2 ELISA Kits) represses adipogenesis by inhibiting PPARgamma (show PPARG ELISA Kits) expression and facilitating Wnt10a expression.
Mechanisms downstream of beta-catenin (show CTNNB1 ELISA Kits) are required for Wnt6 (show WNT6 ELISA Kits), Wnt10a and Wnt10b (show WNT10B ELISA Kits) to influence differentiation of mesenchymal precursors.
WNT10A may be a novel angio/stromagenic growth factor
WNT10A promotes the proliferation of DPCs and negatively regulates their odontoblastic differentiation.
WNT10A exonic variant increases the risk of keratoconus by decreasing corneal thickness
p.Arg113Cys, p.Phe228Ile, newly identified p.Arg171Leu may represent aetiological mutations underlying MLIA w/associated dental anomalies, implicating coding variants in WNT10A gene
this study demonstrated that common variations in WNT10A have pleiotropic effects on the morphology of ectodermal appendages.
WNT10A may induce kidney fibrosis and associate with kidney dysfunction in acute interstitial nephritis.
High WNT10A expression promotes an invasive and self-renewing phenotype in esophageal squamous cell carcinoma
Patients with bi-allelic WNT10A mutations have severe tooth agenesis.
The novel c.-14_7delinsC mutation might be the etiological variant of the WNT10A gene responsible for the permanent tooth agenesis in the Egyptian family.
Barrel-shaped mandibular incisors and severe hypodontia appear to be associated with homozygous or compound heterozygous mutations of WNT10A.
Our study has demonstrated for the first time that agenesis of the maxillary permanent canines is a distinct entity, associated with mutations in WNT10A.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It is strongly expressed in the cell lines of promyelocytic leukemia and Burkitt's lymphoma. In addition, it and another family member, the WNT6 gene, are strongly coexpressed in colorectal cancer cell lines. The gene overexpression may play key roles in carcinogenesis through activation of the WNT-beta-catenin-TCF signaling pathway. This gene and the WNT6 gene are clustered in the chromosome 2q35 region.
wingless-type MMTV integration site family, member 10A
, Wnt signaling ligand
, Wnt10a protein
, hypothetical protein
, WNT10A protein
, protein Wnt-10a-like
, protein Wnt-10a
, wingless related MMTV integration site 10a