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The protein encoded by SMAD9 is a member of the SMAD family, which transduces signals from TGF-beta family members. Additionally we are shipping SMAD9 Proteins (7) and SMAD9 Kits (6) and many more products for this protein.
Showing 10 out of 81 products:
Human Polyclonal SMAD9 Primary Antibody for EIA, ELISA - ABIN251308 : Shi, Massagué: Mechanisms of TGF-beta signaling from cell membrane to the nucleus. in Cell 2003 (PubMed)
Mutations in SMAD9 gene encoding for transforming growth factor (TGF)-[beta] superfamily have been identified in Pulmonary Arterial Hypertension.
Phosphorylated Smad1/5/8/9 specifically bound to the BREs of Smad8/9 gene. The present study reveals that Smad8/9 is a unique R-Smad regulated through the BMP pathway at the mRNA level.
Case Report: hamartomatous olyposis and gastrointestinal ganglioneuromas with SMAD9 germline mutation that reduces PTEN expression.
a detailed computational model for TGF-beta (show TGFB1 Antibodies) signalling that incorporates elements of previous models together with crosstalking between Smad1 (show GARS Antibodies)/5/8 and Smad2 (show SMAD2 Antibodies)/3 channels through a negative feedback loop dependent on Smad7 (show SMAD7 Antibodies).
ATP production by NaF (show IL8 Antibodies) promotes hypertrophy-like changes via activation of phospho-Smad8
TNF (show TNF Antibodies) activated NF-kappaB (show NFKB1 Antibodies) pathway and inhibited the phosphorylation of Smad 1 (show GARS Antibodies)/5/8 and BMP-2 (show BMP2 Antibodies)-induced osteoblastic differentiation in BMMSCs
CREBZF (show CREBZF Antibodies), a novel Smad8-binding protein.
Data suggest that Smads 1, 5 and 8 as potential prognostic markers and therapeutic targets for mTOR (show FRAP1 Antibodies) inhibition therapy of prostate cancer.
Endoglin (show ENG Antibodies) promotes transforming growth factor beta-mediated Smad 1 (show GARS Antibodies)/5/8 signaling and inhibits endothelial cell migration through its association with GIPC (show GIPC1 Antibodies)
Study described the first mutation in SMAD8 in a patient with IPAH, and suggested the involvement of SMAD8 in the pathogenesis of PAH.
Sphingosine 1 phosphate also up-regulated runt-related transcription factor 2 (Runx2 (show RUNX2 Antibodies)) expression through S1PR2 (show S1PR2 Antibodies)/RhoA (show RHOA Antibodies)/ROCK/Smad1 (show SMAD1 Antibodies)/5/8 signaling.
BetA can enhance in vivo osteogenic potentials of BMP2 (show BMP2 Antibodies), possibly via stimulating Smad 1 (show SMAD1 Antibodies)/5/8 and p38 (show CRK Antibodies) pathways, and combination of both agents can be considered as a therapeutic strategy for bone diseases.
findings suggest that Smad9 is a new type of transcriptional regulator in BMP signaling.
The findings demonstrate for the first time that KMUP-1 can promote osteoblast maturation and differentiation in vitro via BMP-2 (show BMP2 Antibodies)/Smad1 (show SMAD1 Antibodies)/5/8 and Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) pathways.
Data indicate that PDGF (show PDGFA Antibodies)-AA promotes mesenchymal stem cel migration via bone morphogenetic protein 2 (BMP2 (show BMP2 Antibodies))-smad (show SMAD1 Antibodies) proteins smad1 (show SMAD1 Antibodies)/5/8 activation requires lysosome-mediated degradation of PDGF (show PDGFA Antibodies) alpha Receptor (PDGFRalpha).
our data suggest a pivotal role for canonical BMP signaling demonstrating distinguished nonoverlapping function of pSmad8 with pSmad1 and pSmad5 in hfSCs regulation and hair morphogenesis but a redundant role in adult hair progenitors differentiation.
Data show that HtrA1 (show HTRA1 Antibodies) is produced during osteoclastogenesis, and negatively regulates osteoblast differentiation, osteoblast differentiation and BMP2 (show BMP2 Antibodies)-induced Smad1 (show SMAD1 Antibodies)/5/8, ERK1/2 and p38 (show CRK Antibodies) phosphorylation in pre-osteoblasts.
FGF2 (show FGF2 Antibodies) inhibited BMP9 (show GDF2 Antibodies)-induced osteogenic differentiation by blocking BMP9 (show GDF2 Antibodies)-induced Smads signaling and subsequently reducing Smads dependent up-regulation of ALK1 (show ACVRL1 Antibodies) and ALK2 (show ACRV1 Antibodies) in mesenchymal stem cells.
a detailed computational model for TGF-beta (show TGFB1 Antibodies) signalling that incorporates elements of previous models together with crosstalking between Smad1 (show SMAD1 Antibodies)/5/8 and Smad2 (show SMAD2 Antibodies)/3 channels through a negative feedback loop dependent on Smad7 (show SMAD7 Antibodies).
The protein encoded by this gene is a member of the SMAD family, which transduces signals from TGF-beta family members. The encoded protein is activated by bone morphogenetic proteins and interacts with SMAD4. Two transcript variants encoding different isoforms have been found for this gene.
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