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Acts as a negative mediator of apoptosis. Additionally we are shipping TNFAIP8 Proteins (12) and and many more products for this protein.
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The indel chr5:118704153:D, located within TNFAIP8, showed an association with plantar fascial disorders at genome-wide significance (p<5x10(-8)) with small effects (odds ratios=0.93 and 1.07 per allele, respectively).
TNFAIP8 v2 may contribute to both carcinogenesis and chemotherapeutic resistance by broadly suppressing p53 (show TP53 Antibodies) activity, thus offsetting p53 (show TP53 Antibodies)-dependent tumor suppression.
TNFAIP8 seems to regulate the cell survival and cancer progression processes in a multifaceted manner. (Review)
induction of TNFAIP8 is critical for the evasion of apoptosis by tumor cells expressing the K120R variant of p53 (show TP53 Antibodies).
Of these, three CpG sites on TNFAIP8 and PON1 (show PON1 Antibodies) genes (corresponding to: cg23917399; cg07086380; and cg07404485, respectively) were significantly differentially methylated between black and non-black individuals. The three CpG sites showed lower methylation status among infants of black women.
TNFAIP8 regulates Hippo (MST1 (show MST1 Antibodies)/2) signaling through its interaction with LATS1 (show LATS1 Antibodies).
TNFAIP8 overexpression is correlated with axillary lymph node metastasis and poor prognosis in invasive ductal breast carcinoma.
that MicroRNA-9-TNFAIP8 might represent a promising diagnostic biomarker for gastric cancer patients and could be a potential therapeutic target in the prevention and treatment of gastric cancer
these findings suggest that TNFAIP8 overexpression is a potential biomarker to identify pN0 esophageal squamous cell carcinoma patients at higher risk of lymphatic recurrence who may benefit from adjuvant therapy.
Expression of TNFAIP8 is up-regulated in human gastric cancer and regulates cell proliferation, invasion and migration.
Study revealed that TIPE overexpression obviously augmented the volume and weight of the tumors, indicating that TIPE facilitates tumor formation probably through its antiapoptotic effect on JNK (show MAPK8 Antibodies) and p38 (show CRK Antibodies) activation.
TNFAIP8 expression following CLP may be associated with the pathogenesis of immune dysfunction in splenic T lymphocytes in mice.
CD4 (show CD4 Antibodies)+ T lymphocyte proliferative activity was significantly down-regulated when TNFAIP8 gene was silenced by siRNA in mice at 24 h post burn. Down-regulation of TNFAIP8 can significantly decrease expression levels of IL-2 (show IL2 Antibodies) and soluble IL-2R at 24 h after thermal injury.
TNFAIP8 regulates RAC1 signaling during TNF (show TNF Antibodies)-alphaR activation and L. monocytogenes invasion of cells and that this regulation is crucial for controlling cell death and lethal L. mono-cytogenes infection of mice.
Data indicate that mutant Tnfaip8/Oxi-alpha has been crystallized and X-ray data have been collected to 1.8 A using synchrotron radiation.
Tnfaip8 expression is associated with susceptibility to Staphylococcus aureus sepsis.
Data suggest that Galphai-TNFAIP8-mediated rescue of pre-oncogenic cells enhances progression to oncogenic transformation, providing a selective target to inhibit cellular transformation.
Tnfaip8 is crucial in regulating glucocorticoid-mediated apoptosis of thymocytes
Acts as a negative mediator of apoptosis. Suppresses the TNF-mediated apoptosis by inhibiting caspase-8 activity but not the processing of procaspase-8, subsequently resulting in inhibition of BID cleavage and caspase-3 activation (By similarity).
tumor necrosis factor, alpha-induced protein 8
, TNF alpha-induced protein 8
, tumor necrosis factor alpha-induced protein 8
, NF-kappa-B-inducible DED-containing protein
, TNF-induced protein GG2-1
, head and neck tumor and metastasis-related protein
, TNF-induced protein