Browse our anti-Angiotensin I Converting Enzyme 1 (ACE) Antibodies

Human Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. Review/Meta-analysis: ACE I/D polymorphism may be a genetic molecular marker to predict systemic lupus erythematosus but not lupus nephritis.

2. Association of insertion/deletion (I/D) polymorphism at angiotensin-converting enzyme gene (ACE) with depression in Chinese adolescents experiencing the 2008 Wenchuan earthquake.

3. The ACE (I/D) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.

4. polymorphisms in the eNOS (show NOS3 Antibodies) "A/A" (homozygous mutant) and ACE "I/D" genotypes might contribute to the increased risk of NSCLC in the South Indian population.

5. The study attempts to understand the distribution and extent of association of ACE I/D gene polymorphism with cardiometabolic risk factors among Bhils from rural and urban settings.

6. Data suggest that the insertion/deletion (I/D) ACE (angiotensin converting enzyme) gene polymorphism may be associated with MI occurrence among younger patients.

7. The analysis of genotype coexistence revealed a higher incidence of the combination of the ACE II and the PAI-1 (show SERPINE1 Antibodies) 4G/4G genotypes in the control group (10.0 vs.5.9% in control group; p = 0.17). CONCLUSIONS: The obtained results suggest no apparent association between the ACE I/D, PAI-1 (show SERPINE1 Antibodies) 4G/5G polymorphisms and increased RM susceptibility in the analyzed Polish population.

8. An involvement of the ACE polymorphism in element imbalances in hypertension

9. Concomitant presence of non-functional variant in ACTN3 (show ACTN3 Antibodies) gene decreased this beneficiary effect of ACE mutation on SBPR3.

10. There were significant differences in sarcopenic obesity according to ACE I/D genotype, Women who were ACE DD presented lower risk of sarcopenic obesity than those in the ACE II and ACE ID groups

Mouse (Murine) Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. Resveratrol upregulated ACE2 (show ACE2 Antibodies) and inhibited abdominal aortic aneurysm growth in a mouse model.

2. In mice with renal injury induced by L-NAME pretreatment, renal tubular epithelial ACE is essential for renal angiotensin II accumulation and salt-sensitive hypertension

3. Studied ACE role in peptide processing and for MHC class II antigen presentation; found ACE level effects efficiency of antigen presentation, and overexpression or inhibition of ACE alters the CD4 (show CD4 Antibodies)(+) T-cell and antibody response.

4. ACE enhances the oxidative response and bactericidal activity of neutrophils.

5. this study shows that angiotensin-converting enzyme inhibitor captopril rescues mice from endotoxin-induced lethal hepatitis

6. Smooth muscle cell-derived ACE contributes to atherosclerosis, independent of circulating ACE activity and blood pressure.

7. Intestinal ACE shedding is increased by DSS (show PMP22 Antibodies)-induced intestinal inflammation and parallels local corticosterone production. ACE product angiotensin II stimulates corticosterone formation in healthy intestine.

8. Renin (show REN Antibodies) angiostensin system contributes to 20-HETE-mediated microvascular remodeling in hypertension and that 20-HETE-driven microvascular remodeling independent of blood pressure elevation does not fully rely on ACE activity in the vascular endothelium.

9. angiotensin-converting enzyme has an essential role in hypertension induced by nitric oxide synthesis inhibition

10. Suggest that the ACE2 (show ACE2 Antibodies)-ACE imbalance plays an important role in the pathogenesis of severe acute pancreatitis and that pancreatic ACE2 (show ACE2 Antibodies) is an important factor in determining the severity of SAP (show APCS Antibodies).

Pig (Porcine) Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. Maintaining the balance of the ACE2 (show ACE2 Antibodies)/ACE axis is important for inhibiting pulmonary apoptosis during acute pulmonary embolism.

2. Tissue Ang I (show AGT Antibodies)-II conversion depends exclusively on the ACE C-domain, whereas both domains contribute to conversion by soluble ACE and to bradykinin degradation at tissue sites.

3. The contributions of the C-domain and N-domain differ between DDs and IIs genotype

Rabbit Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. Corneal cells express ACE, AT(1) and AT(2)receptors. ACE inhibitor enalapril decreased corneal angiogenesis in VEGF (show VEGFA Antibodies)-induced corneal neovascularization. ACE inhibitors may be novel therapy to treat corneal angiogenesis.

2. Angiotensin-converting enzyme (ACE) residues encompassing 343 to 655 of the germinal form are required for its cleavage-secretion.

Cow (Bovine) Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. a portion of the extracellular region of tACE (show ADAM17 Antibodies) (containing its catalytic site) is released from bovine sperm during capacitation, and tACE (show ADAM17 Antibodies) activity may be required for sperm capacitation.

2. The results indicate that the two active sites within bovine somatic ACE exhibit strong negative cooperativity.

3. Phorbol 12-myristate 13-acetate (PMA) induced Egr-1 and ATF-2 binding to the ACE promoter, whereas Ets-1 binding was suppressed by PMA.

Horse (Equine) Angiotensin I Converting Enzyme (Peptidyl-Dipeptidase A) 1 (ACE) interaction partners

1. Precompetition angiotensin converting enzyme (ACE) activity in endurance horses competing in an 80 km event was not associated with either finishing position or heart rates, indicating that the enzyme is not a good predictor of performance.

2. This research investigated angiotensin-converting enzyme activity in the plasma of racehorses and demonstrated that its activity is increased in horses with higher degrees of hemorrhage and is a promising biomarker for pulmonary hemorrhage.

3. These results show an increase in ACE activity up to fatigue and a return to baseline values at 30 min post exercise.

4. The aim of this study was to investigate the correlation between angiotensin-converting enzyme (ACE) activity in the equine endometrium and the degree of endometrial periglandular fibrosis.