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Methamphetamine antibody (Biotin)

The Mouse Monoclonal anti-Methamphetamine antibody is suitable to detect Methamphetamine. It has been validated for ELISA.
Catalog No. ABIN723188
$538.77
Plus shipping costs $50.00
100 μL
Shipping to: United States
Delivery in 16 to 20 Business Days

Quick Overview for Methamphetamine antibody (Biotin) (ABIN723188)

Target

Methamphetamine (M-Amp)

Reactivity

Please inquire

Host

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Mouse

Clonality

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Monoclonal

Conjugate

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This Methamphetamine antibody is conjugated to Biotin

Application

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ELISA

Clone

4D2
  • Cross-Reactivity (Details)

    Methamphetamine

    Purification

    Purified by Protein G.

    Immunogen

    KLH conjugated Methamphetamine

    Isotype

    IgG
  • Restrictions

    For Research Use only
  • Format

    Liquid

    Concentration

    1 μg/μL

    Buffer

    Aqueous buffered solution containing 0.01M TBS ( pH 7.4) with 1 % BSA, 0.03 % Proclin300 and 50 % Glycerol.

    Preservative

    Sodium azide

    Precaution of Use

    This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

    Storage

    -20 °C

    Storage Comment

    Store at -20°C for 12 months.

    Expiry Date

    12 months
  • Target

    Methamphetamine (M-Amp)

    Alternative Name

    Methamphetamine

    Target Type

    Chemical

    Background

    Synonyms: d-Desoxyephedrine hydrochloride, d-N, -Dimethylphenethylamine hydrochloride, Methylamphetamine hydrochloride, METH.

    Background: Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.

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