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anti-Mouse (Murine) MNAT1 Antibodies:
anti-Human MNAT1 Antibodies:
anti-Rat (Rattus) MNAT1 Antibodies:
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Deletion of the TFIIH kinase subunit Mat1 in mouse fibroblasts leads to dramatically reduced RNA polymerase II Ser5 phosphorylation.
investigated the function of the Cdk7 (show CDK7 Antibodies).cyclin (show PCNA Antibodies) H.Mat1 complex in murine embryonic stem (ES) cells and preimplantation embryos to determine whether it regulates the unique cell cycle structure and transcriptional network of pluripotent cells
This study demonstrates a requirement for MAT1 in the operation of PGC-1 coactivators that control cell metabolism.
study found that Mat1 (show MAT1A Antibodies) and Cdk7 (show CDK7 Antibodies) levels are undetectable in adipose tissues in vivo and downregulated during adipogenesis
Expressions of components of the CAK complex (show CCNH Antibodies), CDK7 (show CDK7 Antibodies), MAT1 (show MAT1A Antibodies), and Cyclin H (show CCNH Antibodies) are elevated in breast cancer.
The intrinsically programmed MAT1 (show MAT1A Antibodies) expression and fragmentation regulate granulopoiesis.
The cyclin H (show CCNH Antibodies)/cdk7 (show CDK7 Antibodies)/Mat1 (show MAT1A Antibodies) kinase activity is regulated by CK2 (show CSNK2A1 Antibodies) phosphorylation of cyclin H (show CCNH Antibodies).
Retinoid-induced G1 arrest and differentiation activation are associated with a switch to enzyme hypophosphorylation of retinoic acid receptor alpha (show RARA Antibodies).
interaction with MTA1 (show MTA1 Antibodies) and role in regulating estrogen receptor (show ESR1 Antibodies) transactivation functions
In response to ATRA, PML (show PML Antibodies)/RARalpha (show RARA Antibodies) is dissociated from CAK (show CCNH Antibodies), leading to MAT1 (show MAT1A Antibodies) degradation, G1 arrest, and decreased CAK (show CCNH Antibodies) phosphorylation of PML (show PML Antibodies)/RARalpha (show RARA Antibodies)
Retinoic-acid-induced RAR (show RARA Antibodies)-CAK (show CCNH Antibodies) signaling events appear to proceed intrinsically during granulocytic development of normal primitive hematopoietic cells. ALDH-governed RA availability may mediate this process by initiating RAR (show RARA Antibodies)-CAK (show CCNH Antibodies) signaling.
These results suggested that genetic variants in CAK (show CCNH Antibodies) genes, Cdk7 (show CDK7 Antibodies), cyclin H (show CCNH Antibodies), MAT1 (show MAT1A Antibodies), might modulate the risk of lung cancer in a gene-gene interaction mode, which consist to the biochemical interaction of corresponding proteins.
The protein encoded by this gene, along with cyclin H and CDK7, forms the CDK-activating kinase (CAK) enzymatic complex. This complex activates several cyclin-associated kinases and can also associate with TFIIH to activate transcription by RNA polymerase II. Two transcript variants encoding different isoforms have been found for this gene.
CDK-activating kinase assembly factor MAT1
, cdk-activating kinase assembly factor mat1
, CDK7/cyclin H assembly factor
, CDK7/cyclin-H assembly factor
, RING finger protein MAT1
, menage a trois 1
, RING finger protein 66
, cyclin G1 interacting protein
, cyclin-G1-interacting protein
, menage a trois 1 (CAK assembly factor)
, menage a trois homolog 1, cyclin H assembly factor