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anti-Human Angiotensin II Antibodies:
anti-Rat (Rattus) Angiotensin II Antibodies:
anti-Mouse (Murine) Angiotensin II Antibodies:
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Human Polyclonal Angiotensin II Primary Antibody for IHC, ELISA - ABIN1583979
Matsuura-Hachiya, Arai, Ozeki, Kikuta, Nishiyama: Angiotensin-converting enzyme inhibitor (enalapril maleate) accelerates recovery of mouse skin from UVB-induced wrinkles. in Biochemical and biophysical research communications 2014
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Human Polyclonal Angiotensin II Primary Antibody for IF (p), IHC (p) - ABIN670521
Anand, Yiangou, Sinisi, Fox, MacQuillan, Quick, Korchev, Bountra, McCarthy, Anand: Mechanisms underlying clinical efficacy of Angiotensin II type 2 receptor (AT2R) antagonist EMA401 in neuropathic pain: clinical tissue and in vitro studies. in Molecular pain 2015
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After donor nephrectomy, increasing uAGT (show DPAGT1 Antibodies) levels can be the result of activation of the intrarenal renin (show REN Antibodies)-angiotensin system affecting the compensatory changes in the remaining kidney.
M235T polymorphism of the AGT (show AGXT Antibodies) gene seems unrelated to the development or the clinical course of endometriosis.
AGT (show AGXT Antibodies) missense polymorphisms are not associated with diabetic nephropathy in our subset of Slovenian type 2 diabetes mellitus patients
Association of AGT (show AGXT Antibodies) single nucleotide polymorphism rs3789678 and gestational hypertension in Chinese population.
The T allele of AGT (show AGXT Antibodies) may play a role in the pathogenesis of preeclampsia in South African Black women.
These data indicate that Ang II (show AGT Antibodies)-AT2R (show AGTR2 Antibodies) regulates human bone marrow MSC (show MSC Antibodies) migration by signaling through the FAK (show PTK2 Antibodies) and RhoA (show RHOA Antibodies)/Cdc42 (show CDC42 Antibodies) pathways.
Data suggest that up-regulaton of Ang-(1 (show ANGPT1 Antibodies)-7) levels in follicular fluid correlates with increases in number of mature oocytes retrieved upon ovarian stimulation in preparation for in vitro fertilization.
Urinary angiotensinogen (show AGT Antibodies) and renin (show REN Antibodies) excretion are elevated in CKD patients. Both parameters are negatively associated with eGFR (show EGFR Antibodies) and these associations are independent of urinary albumin (show ALB Antibodies) excretion
Reduced IL-18 (show IL18 Antibodies) serum concentration in children after HUS (show CFH Antibodies) with no difference in its urine concentration may indicate a loss of the protective effects of this cytokine on renal function due to previously occurred HUS (show CFH Antibodies).
Angiotensin II initiates hepatocyte epithelial-mesenchymal transition by activating the NOX-derived H2O2-mediated NLRP3 (show NLRP3 Antibodies) inflammasome/IL-1ss/Smad (show SMAD1 Antibodies) circuit.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (show TNFAIP3 Antibodies) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (show NFKB1 Antibodies) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (show NLRP3 Antibodies) gene deletion attenuates Ang II (show AGT Antibodies)-induced NLRP3 (show NLRP3 Antibodies) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (show AGXT Antibodies) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (show AGXT Antibodies).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (show ANGPT2 Antibodies) levels and plasma PREP (show PREP Antibodies) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (show TLR4 Antibodies) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (show TNF Antibodies) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (show TLR4 Antibodies) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (show IL6 Antibodies) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (show IL6 Antibodies)/STAT3 (show STAT3 Antibodies) and EndoG (show ENDOG Antibodies)/MEF2A (show MEF2A Antibodies) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II (show AGT Antibodies) could increase TRPC6 (show TRPC6 Antibodies) induced Ca(2 (show CA2 Antibodies)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (show AGT Antibodies)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (show AGTRAP Antibodies).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (show AGTRAP Antibodies)) in the inflammation induced by Aah (show ASPH Antibodies) venom, in the heart and the aorta.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll