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anti-Human Angiotensin II Antibodies:
anti-Rat (Rattus) Angiotensin II Antibodies:
anti-Mouse (Murine) Angiotensin II Antibodies:
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Human Polyclonal Angiotensin II Primary Antibody for IF (p), IHC (p) - ABIN670521
Anand, Yiangou, Sinisi, Fox, MacQuillan, Quick, Korchev, Bountra, McCarthy, Anand: Mechanisms underlying clinical efficacy of Angiotensin II type 2 receptor (AT2R) antagonist EMA401 in neuropathic pain: clinical tissue and in vitro studies. in Molecular pain 2015
Show all 2 Pubmed References
Unfavorable genotype of polymorphic variants of candidate gene participating in endothelial dysfunction AGT (show AGXT Antibodies) (Thrl74Met and Met23SThr) was associated with changes in levels of their active substances in individuals exposed to mercury.
These findings reveal the critical role of hypoxia in producing local angiotensin II by a lactate-chymase (show CMA1 Antibodies)-dependent mechanism and highlight the importance of local angiotensin II in regulating radioresistance of hypoxic tumor cells.
The reduced urinary AGT (show AGXT Antibodies)/creatinine in Autralian Indigenous pregnant women may reflect subclinical renal dysfunction which limits the ability of the kidney to maintain sodium balance and could indicate an increased risk of pregnancy complications and/or future renal disease.
ACE2 (show ACE2 Antibodies) and other enzymes can form ANG-(1 (show ANGPT1 Antibodies)-7) directly or indirectly from either the decapeptide ANG I (show AGT Antibodies) or from ANG II (show AGT Antibodies). [review]
The ACE (show ACE Antibodies) and AGT (show AGXT Antibodies) gene polymorphisms are not associated with the progress of diabetes developing into retinopathy in Chinese patients with type 2 diabetes.
AGT (show AGXT Antibodies) M235T and T174M variants contribute to an increased risk of developing preeclampsia (PE), and for M235T to PE severity.
Data, including data using network analysis, suggest that angiotensinogen (AGT (show AGT Antibodies)), mitogen-activated protein kinase-14 (MAPK14 (show MAPK14 Antibodies)), and prothrombin (show F2 Antibodies) (F2) in placental villous tissues are core factors in early embryonic development; these studies involved proteomics and bioinformatics analysis of altered protein expression in placental villous tissue from early recurrent miscarriage patients in comparison to control tissues.
The AGT (show AGXT Antibodies) (M235T) gene polymorphism do not seem to have a significant effect on the development of clinical properties or cardiovascular comordities of acromegalic patients.
Angiotensin II has a role in increasing glomerular permeability by beta-arrestin mediated nephrin (show NPHS1 Antibodies) endocytosis
After donor nephrectomy, increasing uAGT (show DPAGT1 Antibodies) levels can be the result of activation of the intrarenal renin (show REN Antibodies)-angiotensin system affecting the compensatory changes in the remaining kidney.
ANG II is up-regulated in serum and heart tissues of mice with EAM and that ANG II significantly drives monocyte/macrophage infiltration through the C-C chemokine receptor 2/5 (CCR2/5) axis.
results established that A20 (show TNFAIP3 Antibodies) is involved in the renoprotective effect by calcitriol via negatively modulating the NF-kappaB (show NFKB1 Antibodies) pathway and necroptotic pathway in AngII-induced renal injury.
NLRP3 (show NLRP3 Antibodies) gene deletion attenuates Ang II (show AGT Antibodies)-induced NLRP3 (show NLRP3 Antibodies) inflammasome activation, phenotypic transformation from a contractile phenotype to a synthetic phenotype and proliferation in primary mice Vascular Smooth Muscle Cells.
adipocyte-derived Agt (show AGXT Antibodies) has essentially no contribution to the plasma concentration and no impact on blood pressure compared to liver-derived Agt (show AGXT Antibodies).
Lung ischemia-reperfusion injury causes a dysregulation of circulating Ang 2 (show ANGPT2 Antibodies) levels and plasma PREP (show PREP Antibodies) activity, although no direct link between both phenomena could be shown.
Inhibition of TLR4 (show TLR4 Antibodies) ameliorates AngII-impaired cavernosal relaxation, decreases TNF-alpha (show TNF Antibodies) levels, and restores Nitric Oxide bioavailability, demonstrating that TLR4 (show TLR4 Antibodies) partly mediates AngII-induced cavernosal dysfunction.
Our study is the first to show the important role of IL-6 (show IL6 Antibodies) in regulating cardiac pathogenesis via inflammation and apoptosis during AngII-induced hypertension. We also provide a novel link between IL-6 (show IL6 Antibodies)/STAT3 (show STAT3 Antibodies) and EndoG (show ENDOG Antibodies)/MEF2A (show MEF2A Antibodies) pathway that affects cardiac hypertrophy during AngII stimulation.
this study demonstrated that Ang II (show AGT Antibodies) could increase TRPC6 (show TRPC6 Antibodies) induced Ca(2 (show CA2 Antibodies)+) influx and enhance autophagy through increasing reactive oxygen species levels in podocytes, and autophagy could protect Ang II (show AGT Antibodies)-treated podocytes.
These results implied that AngII could effectively induce EpiCs to differentiate into vascular smooth muscle-like cells through the AT1 receptor (show AGTRAP Antibodies).
Results suggest the involvement of angiotensin II (Ang II), through its angiotensin type-1 receptor (AT1R (show AGTRAP Antibodies)) in the inflammation induced by Aah (show ASPH Antibodies) venom, in the heart and the aorta.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll