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Human Glutamate Receptor, Ionotropic, AMPA 2 (GRIA2) interaction partners

1. Measured the expression of GRIA2 and GABRA1 (show GABRA1 Antibodies) in patients with methamphetamine-use disorder. Also examined whether miR (show MLXIP Antibodies)-181a down-regulates GRIA2 and GABRA1 (show GABRA1 Antibodies) in a cell-based assay. We further examined the effects of chronic methamphetamine exposure on the expression of miR (show MLXIP Antibodies)-181a, GRIA2 and GABRA1 (show GABRA1 Antibodies). The results demonstrated that serum GRIA2 is higher in patients with methamphetamine-use disorder than in healthy controls.

2. This study demonstrated that a significant decrease in the protein level of GluN2A (show GRIN2A Antibodies) in major depression disorder.

3. both the intracellular C-terminal domain (CTD) and the loop region between the M1 and M2 helices move during activation and the CTD is detached from the membrane

4. The results of this study suggest that neurons in hypothalamic hamartoma may bear Ca(2 (show CA2 Antibodies)+) -permeable AMPA (show GRIA3 Antibodies) receptors(GluA2 ) due to dislocation of ADAR2 (show ADARB1 Antibodies)

5. A transient positive feedback mechanism between AMPAR and stargazin (show CACNG2 Antibodies) has implications for information processing in the brain, because it should allow activity-dependent facilitation of excitatory synaptic transmission through a postsynaptic mechanism.

6. The GluR2 subunit of the AMPA (show GRIA3 Antibodies) receptor is involved in cell migration and calcium signaling.

7. RAB39B (show RAB39B Antibodies) selectively regulates GluA2 trafficking to determine synaptic AMPAR composition

8. GRIA2*CCC polymorphism is genetic risk marker for paranoid schizophrenia in Russians.Low risk genetic markers of paranoid schizophrenia were revealed: in Tatars-GRIA2*T/T (rs43025506) of GRIA2 gene and GRIA2*CCT in Russians.

9. GRIA2 is a useful marker for distinguishing solitary fibrous tumour from most mimics

10. a link between neurodegenerative processes and deficient RNA editing of the GluA2 Q/R site.

Mouse (Murine) Glutamate Receptor, Ionotropic, AMPA 2 (GRIA2) interaction partners

1. GluA2 C-terminus is required for synaptic scaling in CA1 (show CA1 Antibodies) hippocampal pyramidal neurons.

2. translocation of surface GluA1 (show GRIA1 Antibodies), but not GluA2, AMPAR subunits to the synapse requires the amino-terminal domain

3. Critically, by altering the two interacting loops of TARP (show TINAGL1 Antibodies) gamma2 and TARP (show TINAGL1 Antibodies) gamma8, the authors could entirely remove all allosteric modulation of GluA2, without affecting formation of AMPA (show GRIA3 Antibodies) receptor-TARP (show TINAGL1 Antibodies) complexes.

4. the GluA2 subunit via its interaction with GAPDH (show GAPDH Antibodies), play a critical role in cortical neurodevelopment.

5. The results of this study indicate that disrupting GluA2 phosphorylation leads to increased responsivity to acute stress following cocaine exposure and increased vulnerability to chronic stress.

6. Long-term depression proceeds upon stimulation in cerebellar Purkinje cells in mice carrying mutated GluA2 C terminus.

7. The data of this study indicated that GluA2-lacking AMPARs are present at D1R (show DRD1 Antibodies)-expressing MSN (show MSN Antibodies) synapses after withdrawal from both contingent and non-contingent cocaine exposure.

8. Topological regulation of synaptic AMPA (show GRIA3 Antibodies) receptor expression by the RNA-binding protein CPEB3 (show CPEB3 Antibodies) has been demonstrated.

9. Results indicate that disrupting GluA2 phosphorylation and increasing GluA2-mediated transmission in the nucleus accumbens leads to increased vulnerability to cocaine relapse.

10. Chronic stress-elicited depressive behavior may be due to hypertrophy of basolateral amygdala (BLA (show LACTB Antibodies)) neuronal dendrites and increased of Glur1 (show GRIA1 Antibodies)-Glur2 ratio in BLA (show LACTB Antibodies) neurons.