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Human Monoclonal GRIA2 Primary Antibody for FACS, ELISA - ABIN969524
Shen, Jin: [GluR2 expression in the developing rat inferior colliculus and the relationship with development of synapse]. in Lin chuang er bi yan hou tou jing wai ke za zhi = Journal of clinical otorhinolaryngology, head, and neck surgery 2010
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Human Polyclonal GRIA2 Primary Antibody for IHC, IHC (p) - ABIN4314579
Leja, Essaghir, Essand, Wester, Oberg, Tötterman, Lloyd, Vasmatzis, Demoulin, Giandomenico: Novel markers for enterochromaffin cells and gastrointestinal neuroendocrine carcinomas. in Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc 2009
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Rat (Rattus) Polyclonal GRIA2 Primary Antibody for WB - ABIN361455
Murphy, Tcharnaia, Beshara, Jones: Cortical development of AMPA receptor trafficking proteins. in Frontiers in molecular neuroscience 2012
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Rat (Rattus) Monoclonal GRIA2 Primary Antibody for IP, WB - ABIN1742378
Jedlicka, Vnencak, Krueger, Jungenitz, Brose, Schwarzacher: Neuroligin-1 regulates excitatory synaptic transmission, LTP and EPSP-spike coupling in the dentate gyrus in vivo. in Brain structure & function 2015
Rat (Rattus) Polyclonal GRIA2 Primary Antibody for IP, WB - ABIN1742379
Luchkina, Huupponen, Clarke, Coleman, Keinänen, Taira, Lauri: Developmental switch in the kinase dependency of long-term potentiation depends on expression of GluA4 subunit-containing AMPA receptors. in Proceedings of the National Academy of Sciences of the United States of America 2014
Chicken Polyclonal GRIA2 Primary Antibody for WB - ABIN2473833
Egebjerg, Kukekov, Heinemann: Intron sequence directs RNA editing of the glutamate receptor subunit GluR2 coding sequence. in Proceedings of the National Academy of Sciences of the United States of America 1994
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Human Polyclonal GRIA2 Primary Antibody for CM, ICC - ABIN2746695
Puthussery, Gayet-Primo, Taylor, Haverkamp: Immunohistochemical identification and synaptic inputs to the diffuse bipolar cell type DB1 in macaque retina. in The Journal of comparative neurology 2012
Measured the expression of GRIA2 and GABRA1 (show GABRA1 Antibodies) in patients with methamphetamine-use disorder. Also examined whether miR (show MLXIP Antibodies)-181a down-regulates GRIA2 and GABRA1 (show GABRA1 Antibodies) in a cell-based assay. We further examined the effects of chronic methamphetamine exposure on the expression of miR (show MLXIP Antibodies)-181a, GRIA2 and GABRA1 (show GABRA1 Antibodies). The results demonstrated that serum GRIA2 is higher in patients with methamphetamine-use disorder than in healthy controls.
This study demonstrated that a significant decrease in the protein level of GluN2A (show GRIN2A Antibodies) in major depression disorder.
both the intracellular C-terminal domain (CTD) and the loop region between the M1 and M2 helices move during activation and the CTD is detached from the membrane
The results of this study suggest that neurons in hypothalamic hamartoma may bear Ca(2 (show CA2 Antibodies)+) -permeable AMPA (show GRIA3 Antibodies) receptors(GluA2 ) due to dislocation of ADAR2 (show ADARB1 Antibodies)
A transient positive feedback mechanism between AMPAR and stargazin (show CACNG2 Antibodies) has implications for information processing in the brain, because it should allow activity-dependent facilitation of excitatory synaptic transmission through a postsynaptic mechanism.
The GluR2 subunit of the AMPA (show GRIA3 Antibodies) receptor is involved in cell migration and calcium signaling.
RAB39B (show RAB39B Antibodies) selectively regulates GluA2 trafficking to determine synaptic AMPAR composition
GRIA2*CCC polymorphism is genetic risk marker for paranoid schizophrenia in Russians.Low risk genetic markers of paranoid schizophrenia were revealed: in Tatars-GRIA2*T/T (rs43025506) of GRIA2 gene and GRIA2*CCT in Russians.
GRIA2 is a useful marker for distinguishing solitary fibrous tumour from most mimics
a link between neurodegenerative processes and deficient RNA editing of the GluA2 Q/R site.
GluA2 C-terminus is required for synaptic scaling in CA1 (show CA1 Antibodies) hippocampal pyramidal neurons.
translocation of surface GluA1 (show GRIA1 Antibodies), but not GluA2, AMPAR subunits to the synapse requires the amino-terminal domain
Critically, by altering the two interacting loops of TARP (show TINAGL1 Antibodies) gamma2 and TARP (show TINAGL1 Antibodies) gamma8, the authors could entirely remove all allosteric modulation of GluA2, without affecting formation of AMPA (show GRIA3 Antibodies) receptor-TARP (show TINAGL1 Antibodies) complexes.
the GluA2 subunit via its interaction with GAPDH (show GAPDH Antibodies), play a critical role in cortical neurodevelopment.
The results of this study indicate that disrupting GluA2 phosphorylation leads to increased responsivity to acute stress following cocaine exposure and increased vulnerability to chronic stress.
Long-term depression proceeds upon stimulation in cerebellar Purkinje cells in mice carrying mutated GluA2 C terminus.
The data of this study indicated that GluA2-lacking AMPARs are present at D1R (show DRD1 Antibodies)-expressing MSN (show MSN Antibodies) synapses after withdrawal from both contingent and non-contingent cocaine exposure.
Topological regulation of synaptic AMPA (show GRIA3 Antibodies) receptor expression by the RNA-binding protein CPEB3 (show CPEB3 Antibodies) has been demonstrated.
Results indicate that disrupting GluA2 phosphorylation and increasing GluA2-mediated transmission in the nucleus accumbens leads to increased vulnerability to cocaine relapse.
Chronic stress-elicited depressive behavior may be due to hypertrophy of basolateral amygdala (BLA (show LACTB Antibodies)) neuronal dendrites and increased of Glur1 (show GRIA1 Antibodies)-Glur2 ratio in BLA (show LACTB Antibodies) neurons.
Glutamate receptors are the predominant excitatory neurotransmitter receptors in the mammalian brain and are activated in a variety of normal neurophysiologic processes. This gene product belongs to a family of glutamate receptors that are sensitive to alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA), and function as ligand-activated cation channels. These channels are assembled from 4 related subunits, GRIA1-4. The subunit encoded by this gene (GRIA2) is subject to RNA editing (CAG->CGG\; Q->R) within the second transmembrane domain, which is thought to render the channel impermeable to Ca(2+). Human and animal studies suggest that pre-mRNA editing is essential for brain function, and defective GRIA2 RNA editing at the Q/R site may be relevant to amyotrophic lateral sclerosis (ALS) etiology. Alternative splicing, resulting in transcript variants encoding different isoforms, (including the flip and flop isoforms that vary in their signal transduction properties), has been noted for this gene.
AMPA-selective glutamate receptor 2
, Glutamate receptor 2
, glutamate receptor ionotropic, AMPA 2
, glutamate receptor 2
, glutamate receptor B
, AMPA glutamate receptor 2
, AMPA receptor GluR2/B
, AMPA selective glutamate receptor
, glutamate receptor AMPA 2