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AGGF1 encodes an angiogenic factor that promotes proliferation of endothelial cells. Additionally we are shipping AGGF1 Kits (25) and AGGF1 Proteins (7) and many more products for this protein.
Showing 10 out of 90 products:
Human Polyclonal AGGF1 Primary Antibody for WB - ABIN658163
Fan, Ouyang, Timur, He, You, Hu, Ke, Driscoll, Chen, Wang: Novel roles of GATA1 in regulation of angiogenic factor AGGF1 and endothelial cell function. in The Journal of biological chemistry 2009
Mouse (Murine) Polyclonal AGGF1 Primary Antibody for EIA, IHC (fro) - ABIN1105263
Tian, Kadaba, You, Liu, Timur, Yang, Chen, Szafranski, Rao, Wu, Housman, DiCorleto, Driscoll, Borrow, Wang: Identification of an angiogenic factor that when mutated causes susceptibility to Klippel-Trenaunay syndrome. in Nature 2004
Bmp-induced activation of beta-catenin (show CTNNB1 Antibodies) through Aggf1 regulates caudal (show CAD Antibodies) vein development by promoting the Nr2f2 (show NR2F2 Antibodies)-dependent differentiation of venous endothelial cells and their survival.
aggf1 is involved in differentiation of both hematopoietic and endothelial lineages and acts upstream of scl (show TAL1 Antibodies), fli1 (show FLI1 Antibodies), and etsrp (show ETV2 Antibodies) in specification of hemangioblasts
Our study establishes AGGF1 as an angiogenic factor (show VEGFA Antibodies) with an important role in the specification of vein identity and suggests that AGGF1-mediated AKT (show AKT1 Antibodies) signaling is responsible for establishing venous cell fate
AGGF1 gene polymorphism does not affect the risk of varicose veins of the legs in ethnic Russians.
Overexpression of AGGF1 is correlated with angiogenesis in hepatocellular carcinoma.
AGGF1 reduces myocardial apoptosis and inflammation and enhances angiogenesis, leading to decreased infarct size after I/R injury.
results of this study indicate that hypoxia down-regulates expression of the AGGF1 protein, but not AGGF1 mRNA, by inducing expression of miR (show MLXIP Antibodies)-27a
AGGF1 potently attenuated TNF-alpha (show TNF Antibodies) triggered NF-kappaB (show NFKB1 Antibodies) pathway, as indicated by the decreased promoter activity, nuclear distribution and phosphorylation of NF-kappaB (show NFKB1 Antibodies) p65 (show GORASP1 Antibodies) subunit as well as the increased protein level of IkappaBalpha (show NFKBIA Antibodies).
analysis of sphingolipid modulation of angio (show VEGFA Antibodies)genic factor expression in n (show ARHGEF16 Antibodies)euroblastoma
results define VG5Q as an angiogenic factor (show VEGFA Antibodies), establish VG5Q as a susceptibility gene for Klippel-Trenaunay syndrome, and show that increased angiogenesis is a molecular pathogenic mechanism of KTS
VG5Q, E133K is a mutation that causes Klippel-Trenaunay and other overgrowth syndromes.
This review describes the somatic mutation for angiogenic factor VG5Q, which may be the cause of the multisystem disorder Klippel-Trenaunay syndrome.
identified two tagSNPs, rs13155212 and rs7704267 that capture information for all common variants in AGGF1 as a candidate susceptibility gene for Klippel-Trenaunay syndrome
Sphingolipid modulation of angiogenic factor (show VEGFA Antibodies) expression in neuroblastoma (show ARHGEF16 Antibodies)
This gene encodes an angiogenic factor that promotes proliferation of endothelial cells. Mutations in this gene are associated with a susceptibility to Klippel-Trenaunay syndrome. Pseudogenes of this gene are found on chromosomes 3, 4, 10 and 16.
angiogenic factor with G patch and FHA domains 1
, angiogenic factor VG5Q
, G patch domain-containing protein 7
, vasculogenesis gene on 5q protein