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Anti-Mullerian hormone is a member of the transforming growth factor-beta gene family which mediates male sexual differentiation. Additionally we are shipping AMH Antibodies (106) and AMH Kits (83) and many more products for this protein.
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nuclear receptor subfamily 5, group A, member 1b is a new candidate for sex determination and differentiation in a way similar to steroidogenic factor 1 (show NR5A1 Proteins), possibly involving AMH
zebrafish Anti-Mullerian hormone (Amh) is regulated by sox9a, sox9b, and cyp19a1a during gonad development
amh is a candidate gene down-regulating cyp19a1a, leading to "juvenile ovary-to-testis" transformation.
It was shown that the male-to-female sex reversal phenotype in hotei medaka mutants is not a direct consequence of anti-Mullerian hormone signaling in supporting cells, but is instead mediated by germ cells.
SNPs at the AMH gene locus that strongly predict serum AMH levels in males.
unilateral ovarian endometriosis had a moderately negative and nonsignificant effect on AMH-based ovarian reserve evaluated prior to surgery, irrespective of age
Anti-Mullerian hormone declined with women's increasing age in both plasma and peritoneal fluid in women with and without endometriosis.
Healthy eumenorrheic late adolescent females with sporadic anovulation display higher AMH blood concentration.
The proportion of circulating proAMH as a percentage of total AMH was twofold higher in boys than men, with girls also exhibiting lesser cleavage of their AMH than women.
Neither AMH nor AMHR2 (show AMHR2 Proteins) polymorphisms were related to age, BMI, hormone levels or ovarian parameters in the follicular phase in women of late reproductive stage.
Data from Poland suggest that serum AMH levels are up-regulated in women with polycystic ovary syndrome (PCOS) and type 1 diabetes (T1DM) as compared to women with T1DM alone; no differences are observed between either PCOS plus T1DM or PCOS alone.
Data from Poland suggest serum AMH levels are up-regulated in women with polycystic ovary syndrome (PCOS) and type 1 diabetes (T1DM) as compared to women with T1DM alone; no differences are observed between either PCOS plus T1DM or PCOS alone. [EDITORIAL]
These data support incorporation of AMH as diagnostic criterion for PCOS regardless of age, and imply that testosterone should not be relied upon in the diagnosis of polycystic ovary syndromein older women.
Data suggest that the ratio of anti-Mullerian hormone precursor (proAMH) to anti-Mullerian hormone (AMHN,C) in the circulation is not altered after it is released from the gonads.
AMH increases GnRH-dependent LH pulsatility and secretion, supporting a central action of AMH on GnRH neurons.
AMH and FOXL2 (show FOXL2 Proteins) collaboratively work to reserve ovarian follicles. AMH is an endogenous target gene of FOXL2 (show FOXL2 Proteins).
Up-regulation of SOX9 (show SOX9 Proteins) in sertoli cells from testiculopathic patients accounts for increasing anti-mullerian hormone expression via impaired androgen receptor (show AR Proteins) signaling.
Male mice require AMH to undergo normal social development.
Data show that Purkinje cells express receptors for Mullerian inhibiting substance (MIS), and that MIS(-/-) male mice have female-like numbers of Purkinje cells and a female-like size to other parts of their cerebellum.
MIS may be involved in anterograde rather than autocrine or retrograde regulation of neurons.
FSH (show BRD2 Proteins) and cAMP stimulate AMH transcription by granulosa cells. FSH (show BRD2 Proteins) and LH have an additive effect, which may be important in polycystic ovary syndrome.
This suggests that MIS is one of the determinants of "boy"-specific behavior.
Role of anti-Mullerian hormone (AMH) as a regulator and marker of ovarian function.
Administration of MIS to male mice induced IEX-1S mRNA in the prostate in vivo, suggesting that MIS may function as an endogenous hormonal regulator of NF-kappaB (show NFKB1 Proteins) signaling and growth in the prostate gland.
Measurement of AMH concentration in the plasma of cows can help to predict their capacity for embryo production in response to gonadotrophin treatment.
In first study to investigate the blood profile and immunohuistochemistry of anti-Mullerian hormone in bovine granulosa-theca cell tumors, the findings indicated that anti-Mullerian hormone is a novel biomarker for granulosa-theca cell tumors in cattle.
Regulation of anti-Mullerian hormone production in the cow.
Intrafollicular AMH was not a marker of cystic development in the cow, but low AMH concentrations in cysts were associated with luteinization.
AMH expression is modulated by androgens in bovine granulosa cells from small follicles.
Anti-Mullerian hormone is a member of the transforming growth factor-beta gene family which mediates male sexual differentiation. Anti-Mullerian hormone causes the regression of Mullerian ducts which would otherwise differentiate into the uterus and fallopian tubes. Some mutations in the anti-Mullerian hormone result in persistent Mullerian duct syndrome.
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, anti-mullerian hormone
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