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CD69 encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. Additionally we are shipping CD69 Antibodies (409) and CD69 Proteins (15) and many more products for this protein.
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The generation of cancer-specific CD8 (show CD8A ELISA Kits)(+) CD69(+)-expressing lymphocytes that inhibit colon cancer growth has been described.
Maintenance of immune tolerance by Foxp3 (show FOXP3 ELISA Kits)+ regulatory T cells requires CD69 expression.
AIM(-/-) mice were highly susceptible to steatosis-associated HCC (show FAM126A ELISA Kits) development, whereas no AIM(+/+) mouse developed the disease despite comparable liver inflammation and fibrosis in response to a long-term high-fat diet.
CD69 interference with S1P1 (show S1PR1 ELISA Kits) function regulates peripheral T cell retention.
The NKG2A (show KLRC1 ELISA Kits) expression was increased in hepatic CD69-NK cells.
Report role of ZAP-70 (show ZAP70 ELISA Kits) kinase mutation/deletion on thymocyte-positive selection via CD69 signaling.
The early activation marker CD69 regulates the expression of chemokines and CD4 (show CD4 ELISA Kits) T cell accumulation in intestine.
Data indicate that CD69 is upregulated on endogenous dendritic cells (DC) upon activation.
Intron I acts as an important regulatory element of CD69 expression.
Data show that CD69 is critical for the generation and maintenance of professional memory T-helper (Th) lymphocytes, which can efficiently help humoral immunity in the late phase.
results demonstrate the functional and mechanistic interplays between CD69 and S100A8 (show S100A8 ELISA Kits)/S100A9 (show S100A9 ELISA Kits) in supporting Treg-cell differentiation
Elevated expression of CD69 and CD161 (show KLRB1 ELISA Kits) on NK cells can be considered as immunological risk markers in RSA and IVF (show SCN5A ELISA Kits) failure.
Human tumor-infiltrating CD4 (show CD4 ELISA Kits)+CD69+ T cells suppress T cell proliferation via membarene -bound TGF-beta1 (show TGFB1 ELISA Kits).
these findings identify CD69 and galectin-1 (show LGALS1 ELISA Kits) to be a novel regulatory receptor-ligand pair that modulates Th17 effector cell differentiation and function.
REVIEW: CD69 exerts a complex immunoregulatory role in humans, and that it could be considered as a target molecule for the therapy of immune-mediated diseases
Following coculture with GTKO/CD46 (show CD46 ELISA Kits) pig mesenchymal stromal cells, it is possible that upregulation of CD69 on human T cells initiates signaling events that would regulate CD4 (show CD4 ELISA Kits)+ and CD8 (show CD8A ELISA Kits)+ T cell activation and differentiation.
In patients with allergic rhinitis CD69 antigen is overexpressed on human peripheral blood natural killer cells reflecting their activation status.
This is the first report of the regulation of CD69 expression by LMP-1 (show PDLIM7 ELISA Kits), and this novel finding may, thus, represent an important link between the EBV oncoprotein LMP-1 (show PDLIM7 ELISA Kits) and its critical role in the development of EBV-associated diseases.
CD69 is induced by integrin alpha4beta1 outside-in signalling and T-cell receptor signalling.
CD69 overexpression is associated with the human T-cell leukemia virus type 1 infection and adult T-cell leukemia.
This gene encodes a member of the calcium dependent lectin superfamily of type II transmembrane receptors. Expression of the encoded protein is induced upon activation of T lymphocytes, and may play a role in proliferation. Furthermore, the protein may act to transmit signals in natural killer cells and platelets.
, early activation antigen CD69
, Very Early Activation Antigen
, C-type lectin domain family 2, member C
, CD69 antigen (p60, early T-cell activation antigen)
, activation inducer molecule (AIM/CD69)
, early T-cell activation antigen p60
, early lymphocyte activation antigen
, leukocyte surface antigen Leu-23