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may regulate lung development during late gestation\; expression is induced by glucocorticoids [RGD, Feb 2006].. Additionally we are shipping CRISPLD2 Proteins (5) and CRISPLD2 Kits (4) and many more products for this protein.
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Human Polyclonal CRISPLD2 Primary Antibody for IHC, IHC (p) - ABIN4300522
Yoo, Shin, Kim, Choi, Ferguson, Fazleabas, Young, Lessey, Ha, Jeong: CRISPLD2 is a target of progesterone receptor and its expression is decreased in women with endometriosis. in PLoS ONE 2014
Show all 2 references for ABIN4300522
Human Polyclonal CRISPLD2 Primary Antibody for WB - ABIN2784006
Chiquet, Lidral, Stal, Mulliken, Moreno, Arcos-Burgos, Arco-Burgos, Valencia-Ramirez, Blanton, Hecht: CRISPLD2: a novel NSCLP candidate gene. in Human molecular genetics 2007
Our data suggest that CRISPLD2 may have a unique anti-HMGB1 (show HMGB1 Antibodies) effect via miRNA155 and play an important role in immune balance.
CRISPLD2 is a target of progesterone receptor (show PGR Antibodies) and its expression is decreased in women with endometriosis.
Results demonstrate genetic polymorphism of CRISPLD2 gene is associated with an increased risk of non-syndromic cleft lip with or without cleft palate.
findings define a role for LGL1 in fibroblast expansion and migration, epithelial cell migration, and mesenchymal-epithelial signaling, key processes in fetal lung development.
results support the hypothesis that variants in the CRISPLD2 gene may be involved in the etiology of NS CL(P).
Both LCCL-domains of human CRISPLD2 have high affinity for lipid A.
There is a decreased expression of CRISPLD2 in septic shock and it is association with procalcitonin in sepsis.
CRISPLD2 gene contributes to the etiology of NSCLP in the Northwestern Chinese population. SNP rs1546124 is significantly related to NSCLP, associated with both CL/P and CPO (show CPOX Antibodies) groups, and SNP rs4783099 is significantly associated with CPO (show CPOX Antibodies).
These novel findings suggest that CRISPLD1 plays a role in Nonsyndromic cleft lip and palate (NSCLP) through the interaction with CRISPLD2 and folate pathway genes.
the present investigation did not support the hypothesis of the involvement of CRISPLD2 in nonsyndromic cleft lip and palate
CRISPLD2 is expressed in the mandible, palate and nasopharynx regions during craniofacial development at E13.5-E17.5, respectively. Altogether, these data suggest that genetic variation in CRISPLD2 has a role in the etiology of NSCLP.
may regulate lung development during late gestation\; expression is induced by glucocorticoids
cysteine-rich secretory protein LCCL domain containing 2
, cysteine-rich secretory protein LCCL domain-containing 2
, LCCL domain containing cysteine-rich secretory protein 2
, LCCL domain-containing cysteine-rich secretory protein 2
, cysteine-rich secretory protein 11
, late gestation lung protein 1