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ICAM1 encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. Additionally we are shipping ICAM-1 Antibodies (955) and ICAM-1 Kits (131) and many more products for this protein.
Showing 10 out of 63 products:
Human ICAM-1 Protein expressed in CHO Cells - ABIN2666823
Greve, Davis, Meyer, Forte, Yost, Marlor, Kamarck, McClelland: The major human rhinovirus receptor is ICAM-1. in Cell 1989
Show all 8 references for ABIN2666823
Mouse (Murine) ICAM-1 Protein expressed in CHO Cells - ABIN2666825
Rothlein, Dustin, Marlin, Springer: A human intercellular adhesion molecule (ICAM-1) distinct from LFA-1. in Journal of immunology (Baltimore, Md. : 1950) 1986
Show all 8 references for ABIN2666825
Human ICAM-1 Protein expressed in Escherichia coli (E. coli) - ABIN1047208
Staunton, Marlin, Stratowa, Dustin, Springer: Primary structure of ICAM-1 demonstrates interaction between members of the immunoglobulin and integrin supergene families. in Cell 1988
Show all 3 references for ABIN1047208
Human ICAM-1 Protein expressed in Human Cells - ABIN2002637
Bhalla, Chugh, Mehrotra, Rathore, Tousif, Prakash Dwivedi, Prakash, Kumar Samuchiwal, Kumar, Kumar Singh, Ghanwat, Kumar, Das, Mohmmed, Malhotra, Ranganathan: Host ICAMs play a role in cell invasion by Mycobacterium tuberculosis and Plasmodium falciparum. in Nature communications 2015
MiR (show MLXIP Proteins)-146a regulates inflammatory macrophage infiltration in Polymyositis/Dermatomyositis by targeting TRAF6 (show TRAF6 Proteins) and affecting the IL-17 (show IL17A Proteins)/ICAM-1 pathway.
The intercellular adhesion molecule-1 K469E polymorphism is associated with susceptibility to vasculitis under the heterozygote model, and the intercellular adhesion molecule-1 G241R polymorphism is associated with vasculitis in Europeans, Behcet's disease, and rheumatoid arthritis.
Increased risk of colorectal cancer associated with a polymorphism in the ICAM-1 gene in a German population.
The results suggest that the genetic polymorphism in ICAM-1 rs5498 single nucleotide polymorphisms with genotype AG and GG is associated with hepatocellular carcinoma risk among smokers.
study provides review of the Genome Wide Association (GWA) studies and summarizes its understanding of functional Single Nucleotide Polymorphism (SNP's) of ICAM-1 clinical association
In Taiwanese women with uterine cervical cancer, there was an association with haplotypes and SNPs of ICAM-1.
ICAM-1 Gly241Arg Polymorphism is Not Associated With Polycystic Ovary Syndrome.
MK2 (show KCNA2 Proteins) post-transcriptionally regulates TNF-alpha (show TNF Proteins)-induced ICAM-1 expression by altering the cytoplasmic localization of HuR (show ELAVL1 Proteins) in human lung microvascular endothelial cells.
High ICAM1 expression is associated with non-insulin (show INS Proteins) resistant women with polycystic ovary syndrome.
study reveals that ICAM-1 E469K polymorphism is not involved in the susceptibility of acromegaly but may be associated with fasting plasma glucose, systolic blood pressure, diastolic blood pressureand HDL (show HSD11B1 Proteins)-C, the cardiovascular risk factors of acromegaly
ICAM1 regulates pathological cardiac remodeling by mediating proinflammatory leukocyte infiltration in the left ventricle and cardiac fibrosis and dysfunction.
Its antiatherogenic impact might be associated with a suppressed adhesion to the endothelium due to down-regulation of endothelial ICAM-1 expression.
Luteolin may inhibit tumor angiogenesis and tumor cell proliferation by down-regulation of LFA- 3 and PCNA (show PCNA Proteins) and up-regulation of ICAM-1 in tumor tissue of tumor-bearing mice, thereby achieving its anti-tumor effect.
our results further highlight the pleiotropic role of ICAM-1 in T-cell-dependent immune responses, with a major role in Treg cell development and suppressive function.
the present results shed light on regulation of expression and function of ICAM-1 on neutrophils and identify it as an additional regulator of neutrophil effector responses in host defense.
we identified intracellular adhesion molecule (ICAM)-1, through fibrinogen binding, as a previously unreported mediator of platelet-monocyte interactions
miR (show MLXIP Proteins)-222 was transported into recipient endothelial cells by endothelial microparticles and functionally regulated expression of its target protein ICAM-1 in vitro and in vivo.
Timely blockade of ICAM-1.LFA-1 (show ITGAL Proteins) interaction prevents disease onset in a mouse model of emphysema.
MRTF-A/B depletion results in an increase in the cell surface expression of ICAM-1 and interactions between HAoECs and leukocytes
neutrophils are reliant on endothelial activation for adhesion, initially via E-selectin (show SELE Proteins) and subsequently, by integrin-mediated binding to ICAM-1 and VCAM-1 (show VCAM1 Proteins), combined with CXCL8 (show IL8 Proteins)-dependent chemotaxis.
ADMA has potent adverse effects on cell proliferation, intracellular ROS (show ROS1 Proteins) generation, cell permeability, levels of ICAM-1, and the tight-junction protein occludin (show OCLN Proteins)
Neutrophil lung infiltrations in porcine reproductive and respiratory syndrome virus infection infected animals is both ICAM-1 dependent and independent.
Chitosan oligosaccharides downregulate the expression of E-selectin (show SELE Proteins) and ICAM-1 by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 Proteins) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Data show that all five molecules, BNP, ICAM-1, TNF-alpha (show TNF Proteins), VCAM-1 (show VCAM1 Proteins) and IL-6 (show IL6 Proteins), quickly and reliably signaled adverse interactions.
Altered shear stress stimulates upregulation of endothelial VCAM-1 (show VCAM1 Proteins) and ICAM-1 in a BMP-4 (show BMP4 Proteins)- and TGF-beta1 (show TGFB1 Proteins)-dependent pathway.
ICAM1 and IL10 (show IL10 Proteins) were upregulated in ventilator-induced lung injury. Nuclear transcription factor AP-1 (show JUN Proteins) may be responsible for this upregulation.
Hepatocellular glycogen (show GYS1 Proteins) decreases the expression of ICAM-1 mRNA of hepatic stellate cells.
This gene encodes a cell surface glycoprotein which is typically expressed on endothelial cells and cells of the immune system. It binds to integrins of type CD11a / CD18, or CD11b / CD18 and is also exploited by Rhinovirus as a receptor.
intercellular adhesion molecule 1
, intercellular adhesion molecule 1-like
, cell surface glycoprotein P3.58
, intercellular adhesion molecule 1 (CD54), human rhinovirus receptor
, major group rhinovirus receptor
, leukocyte adhesion molecule