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The protein encoded by IL25 is a cytokine that shares sequence similarity with interleukin 17. Additionally we are shipping Interleukin 25 Proteins (63) and Interleukin 25 Kits (60) and many more products for this protein.
Showing 10 out of 130 products:
Human Polyclonal IL25 Primary Antibody for EIA, IHC (p) - ABIN952925
Xu, Zhang, Wang, Xu, Liu, Han, Wang, Zuo, Li: Opposing roles of IL-17A and IL-25 in the regulation of TSLP production in human nasal epithelial cells. in Allergy 2010
Show all 5 references for ABIN952925
Human Monoclonal IL25 Primary Antibody for FACS, WB - ABIN1043795
Conti, Shen, Nayyar, Stocum, Sun, Lindemann, Ho, Hai, Yu, Jung, Filler, Masso-Welch, Edgerton, Gaffen: Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis. in The Journal of experimental medicine 2009
Human Polyclonal IL25 Primary Antibody for IF (p), IHC (p) - ABIN748013
Al-Samadi, Moossavi, Salem, Sotoudeh, Tuovinen, Konttinen, Salo, Bishehsari: Distinctive expression pattern of interleukin-17 cytokine family members in colorectal cancer. in Tumour biology 2015
Mouse (Murine) Polyclonal IL25 Primary Antibody for ELISA - ABIN2000439
Kleinschek, Owyang, Joyce-Shaikh, Langrish, Chen, Gorman, Blumenschein, McClanahan, Brombacher, Hurst, Kastelein, Cua: IL-25 regulates Th17 function in autoimmune inflammation. in The Journal of experimental medicine 2007
IL-22 (show IL22 Antibodies) restrains tapeworm-mediated protection against colitis via regulation of IL-25 expression
IL-25 promotes the function of CD4 (show CD4 Antibodies)+CD25 (show IL2RA Antibodies)+ T regulatory cells and prolongs skin-graft survival in murine models
IL-25 and IL-33 (show IL33 Antibodies) are critical for induction of Th2-type cytokine-mediated allergic airway eosinophilia, but not Th17-type cytokine-mediated airway neutrophilia, at the local sites of lungs in the challenge phase of mice sensitized EC with OVA
Suggest that chronic exposure of murine airways to IL-25 alone is able to reproduce a local angiogenic milieu in asthmatic mice.
we have demonstrated that the expression of ET-1 (show EDN1 Antibodies) and IL-25 was coordinately upregulated in the lesional keratinocytes of patients with AD and a murine AD model.
Colonic isolated lymphoid follicles develop in the absence of a number of factors required for the development of their small intestinal counterparts and can be specifically suppressed by factors other than IL-25.
These data demonstrate that dysregulated IL-25 expression contributes to lipid accumulation, whereas exogenous IL-25 protects against hepatic steatosis through IL-13 (show IL13 Antibodies) activation of STAT6 (show STAT6 Antibodies).
Concerted activity of IgG1 antibodies and IL-4 (show IL4 Antibodies)/IL-25-dependent effector cells trap helminth larvae in the tissues following vaccination with defined secreted antigens, providing sterile immunity to challenge infection.
tuft cells constitutively express IL-25 to sustain group 2 innate lymphoid cell homeostasis in the resting lamina propria in mice
beta-catenin (show CTNNB1 Antibodies) expression in invariant Natural Killer T cell precursors promotes differentiation toward iNKT2 and iNKT17 effector subsets and supports enhanced capacity to produce type 2 and 17 cytokines which in turn augment lung inflammation in mice.
Therefore, IL-17E/IL-17RB signaling represents a potential therapeutic target for treatment of hepatocellular carcinoma
IL-25 upregulated PD-L1 (show CD274 Antibodies) surface expression through the signaling pathways of JNK (show MAPK8 Antibodies) and STAT3 (show STAT3 Antibodies), with STAT3 (show STAT3 Antibodies) found to constitutively occupy the proximal region of the PD-L1 (show CD274 Antibodies) promoter.
IL-25 overexpression was observed in eosinophilic chronic rhinosinusitis and may serve as a mediator of eosinophilic CRS (show CARS Antibodies).
expression of immunoreactivity for IL-17A (show IL17A Antibodies), IL-17RA (show IL17RA Antibodies), IL-17E, and IL-17F (show IL17F Antibodies) was significantly elevated in prostatic tissue from benign prostatic hyperplasia and prostate cancer compared with that in controls
This article focuses on evidence for the role of IL-25, IL-33 (show IL33 Antibodies) and TSLP (show TSLP Antibodies) in human allergic disease; although this triad of cytokines is described as epithelial derived, it is clear that their expression within lung tissue is much more widespread.[Review]
The levels of mRNA for IL-25 were not significantly elevated in parotid gland tissues from Kimura Disease patients as compared to controls.
asthmatic epithelial cells have an increased intrinsic capacity for expression of a pro-type 2 cytokine in response to a viral infection, and IL-25 is a key mediator of RV-induced exacerbations of pulmonary inflammation
Because IL-25 has a major role in triggering type 2 responses, bronchial epithelial IL-25 expression is likely a key determinant of type 2 response activation in asthma
Human chorionic gonadotropin up-regulates Il25, promoting the proliferation of decidual stromal cells by activation of JNK (show MAPK8 Antibodies) and AKT (show AKT1 Antibodies) signal pathways.
The protein encoded by this gene is a cytokine that shares sequence similarity with interleukin 17. This cytokine can induce NF-kappaB activation, and stimulate the production of interleukin 8. Both this cytokine and interleukin 17B are ligands for the cytokine receptor IL17BR. Studies of a similar gene in mice suggest that this cytokine may be a pro-inflammatory cytokine favoring the Th2-type immune response. Alternative splicing results in multiple transcript variants.
, interleukin 25-like
, UPF0556 protein C19orf10 homolog
, interleukin 17E