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Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. Additionally we are shipping KLRG1 Proteins (12) and many more products for this protein.
Showing 10 out of 129 products:
Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2658355
Huntington, Tabarias, Fairfax, Brady, Hayakawa, Degli-Esposti, Smyth, Tarlinton, Nutt: NK cell maturation and peripheral homeostasis is associated with KLRG1 up-regulation. in Journal of immunology (Baltimore, Md. : 1950) 2007
Show all 8 references for ABIN2658355
Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2662779
Gründemann, Bauer, Schweier, von Oppen, Lässing, Saudan, Becker, Karp, Hanke, Bachmann, Pircher: Cutting edge: identification of E-cadherin as a ligand for the murine killer cell lectin-like receptor G1. in Journal of immunology (Baltimore, Md. : 1950) 2006
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Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2660217
Blaser, Kaufmann, Pircher: Virus-activated CD8 T cells and lymphokine-activated NK cells express the mast cell function-associated antigen, an inhibitory C-type lectin. in Journal of immunology (Baltimore, Md. : 1950) 1999
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Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2659381
Robbins, Nguyen, Takahashi, Mikayama, Biron, Brossay: Cutting edge: inhibitory functions of the killer cell lectin-like receptor G1 molecule during the activation of mouse NK cells. in Journal of immunology (Baltimore, Md. : 1950) 2002
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Human Monoclonal KLRG1 Primary Antibody for FACS - ABIN2660863
McMahon, Zajac, Jamieson, Corral, Hammer, Ahmed, Raulet: Viral and bacterial infections induce expression of multiple NK cell receptors in responding CD8(+) T cells. in Journal of immunology (Baltimore, Md. : 1950) 2002
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Human Monoclonal KLRG1 Primary Antibody for ICFC - ABIN2661956
Robbins, Terrizzi, Sydora, Mikayama, Brossay: Differential regulation of killer cell lectin-like receptor G1 expression on T cells. in Journal of immunology (Baltimore, Md. : 1950) 2003
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Human Polyclonal KLRG1 Primary Antibody for ELISA, WB - ABIN564422
Yoon, Lim, Park, Lee: Cell-to-cell contact with hepatitis C virus-infected cells reduces functional capacity of natural killer cells. in Journal of virology 2011
Ly6C, 4-1BB (show TNFRSF9 Antibodies), and KLRG1 have roles in the activation of lamina propria lymphocytes in the small intestine in a mouse model of Crohn's disease
This finding provides a rationale for the reciprocal expression of KLRG1 and CD103 (show ITGAE Antibodies) in different CD8 (show CD8A Antibodies)(+) T-cell subsets.
Cytotoxic KLRG1-expressed CD8 (show CD8A Antibodies) effector cells and defective T regulatory cells cause murine autoimmune cholangitis.
KLRG1(+) iNKT cells coexpressing CD49d and granzyme A (show GZMA Antibodies) persisted for several months and displayed a potent secondary response to cognate antigen.
high levels of TfRs such as those found on activated lymphocytes were found to be associated with decreased KLRG1 inhibitory function, indicating that TfRs may sequester KLRG1 from interacting with cadherins.
data further demonstrate that the inhibitory activity of KLRG1 is decreased in T cells expressing high levels of TfR (show TFRC Antibodies), indicating that association of KLRG1 with TfR (show TFRC Antibodies) hinders KLRG1-mediated silencing.
Data indicate that CD103 (show ITGAE Antibodies)(+)CD8 (show CD8A Antibodies)(+) T(RM) cells developed in the skin from epithelium-infiltrating precursor cells that lacked expression of the effector-cell marker KLRG1.
Data indicate that increments of CD8 (show CD8A Antibodies) + effector memory T cells in human and mouse chronic lymphocytic leukemia (CLL)(Emu-TCL1 (show TCL1A Antibodies) model) were due to an expansion of the inhibitory killer cell lectin-like receptor (show Klra6 Antibodies) G1 (KLRG1) expressing cellular subset.
Despite their differences, KLRG1+ and KLRG1- Treg cells proved similarly potent in suppressing experimental autoimmune encephalomyelitis.
KLRG1(-/-) mice had a significant survival extension after Mycobacterium tuberculosis infection compared to wild-type controls, and maintained a significantly lower level of pulmonary bacterial load throughout chronic infection.
The results suggest that a disease-associated SNP located within the 3'UTR (show UTS2R Antibodies) of KLRG1 directly interferes with miR (show MLXIP Antibodies)-584-5p binding, allowing for KLRG1 mRNA differential accumulation, which in turn may contribute to pathogenesis of pemphigus.
Demonstrate the presence of increased KLRG1-expressing T-cells in tuberculosis-treated individuals, and present KLRG1 as a marker of decreased human T-cell proliferation following BCG (show SLC11A1 Antibodies)-vaccination.
More T cells were KLRG1+ in the synovial fluid of patients with spondylarthritis/rheumatoid arthritis than crystal arthritis or controls. Those T cells were more functionally active, and migrated towards the synovial fluid of SpA (show FASL Antibodies)/RA patients.
KLRG1 was overexpressed on CD4 (show CD4 Antibodies)(+) T cells.
These results indicate that KLRG1 negatively regulates natural killer cell numbers and functions via the Akt (show AKT1 Antibodies) pathway, thus providing a novel marker and therapeutic target for hepatitis C virus infection.
cytomegalovirus positive had lower proportions of NK-cells expressing inhibitory receptors (KLRG1 and CD158a (show KIR2DL1 Antibodies))
trastuzumab-mediated antibody-dependent cellular cytotoxicity was markedly enhanced by KLRG1-negative peripheral blood mononuclear cells
Natural killer (NK) cells are lymphocytes that can mediate lysis of certain tumor cells and virus-infected cells without previous activation. They can also regulate specific humoral and cell-mediated immunity. The protein encoded by this gene belongs to the killer cell lectin-like receptor (KLR) family, which is a group of transmembrane proteins preferentially expressed in NK cells. Studies in mice suggested that the expression of this gene may be regulated by MHC class I molecules. Alternatively spliced transcript variants have been reported, but their full-length natures have not yet been determined.
killer cell lectin-like receptor subfamily G, member 1
, killer cell lectin-like receptor subfamily G member 1
, mast cell function-associated antigen 2F1
, C-type lectin domain family 15 member A
, C-type lectin domain family 15, member A
, ITIM-containing receptor MAFA-L
, MAFA-like receptor
, mast cell function-associated antigen (ITIM-containing)
, mast cell function-associated antigen