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may be involved in cartilage development [RGD, Feb 2006].. Additionally we are shipping MIA Proteins (21) and MIA Kits (10) and many more products for this protein.
Showing 10 out of 39 products:
The effects of MIA/CD-RAP on transcriptional regulation in chondrocytes, through the regulation of p54(nrb (show NONO Antibodies)) via YBX1 (show YBX1 Antibodies) contributes to the understanding of chondrogenesis.
data provide evidence for a critical role of SOX10 (show SOX10 Antibodies) in melanoma cell invasion through the regulation of MIA and highlight its role as a therapeutic target in melanoma
Focus on the quantitative analysis of the MIA protein as a prognostic tool because it has proven to be a useful serum marker for documenting disease progression of malignant melanoma. Review.
Functional promoter analysis identified the transcription factor YBX1 (show YBX1 Antibodies) as the mediator of MIA activation of p54(nrb (show NONO Antibodies)) transcription.
MIA protein is present in non-segmental vitiligo (show MITF Antibodies) skin and may cause the detachment of melanocytes; its target is integrin alpha5beta1, which determines the breaking and/or weakening of connections among melanocytes and basal membrane
Results show that S-100B, MIA and LDH levels were significantly higher in patients with advanced melanoma than in disease-free patients or healthy controls.
assessed the utility of melanoma inhibitory activity (MIA) serum marker in the follow up and primary diagnosis of stage III melanoma patients
Further diagnostics should be initiated in uveal melanoma patients with serum MIA above 8.3ng/ml.
Data suggest that plasma markers: CEACAM, ICAM-1 (show ICAM1 Antibodies), osteopontin (show SPP1 Antibodies), MIA, TIMP-1 (show TIMP1 Antibodies) and S100B (show S100B Antibodies) particularly when assessed in combination, can be used to monitor patients for disease recurrenc.
The cell-specific production rate of MIA was quantitatively proportional to the aggrecan (show ACAN Antibodies) gene expression level in the early and middle phase of cartilage chondrocyte differentiation.
Attenuated LM expressing MIA.
as observed in other knockout models of molecules important for cartilage development and differentiation, viability and functional integrity is reached by remarkable molecular redundancy in MIA/CD-RAP knockout mice.
The cartilage protein melanoma inhibitory activity contributes to inflammatory arthritis.
Expression analysis of cartilage tissue derived from MIA-/- mice revealed strong downregulation of nuclear RNA-binding protein 54-kDa.
MIA/CD-RAP stabilizes cartilage differentiation and inhibits differentiation into bone potentially by regulating signaling processes during differentiation.
transcriptional mechanism by which CD-RAP expression is suppressed by IL-1 beta (show IL1B Antibodies)
Cdrap/Mia is located between two housekeeping genes
DNA promoter segment from -2,251 bp to -2,068 bp of the CD-RAP gene contains elements critical for gene expression. It demonstrates activation or repression of gene expression in vitro and in vivo based on cell type and content of transcription factors.
A potential tumor suppressor role of Mia/Cd-rap in pituitary cells.
may be involved in cartilage development
melanoma-derived growth regulatory protein
, melanoma inhibitory activity-like
, Melanoma-derived growth regulatory protein
, melanoma inhibitory activity 1
, melanoma inhibitory activity protein
, cartilage derived retinoic acid sensitive protein
, cartilage-derived retinoic acid-sensitive protein
, melanoma inhibitory activity/condrocyte-derived retinoic acid sensitive protein homolog (MIA/CD-RAP)
, melanoma inhibitory activity-like protein