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The ninjurin protein is upregulated after nerve injury both in dorsal root ganglion neurons and in Schwann cells (Araki and Milbrandt, 1996. Additionally we are shipping Ninjurin 1 Kits (11) and Ninjurin 1 Proteins (5) and many more products for this protein.
Showing 10 out of 40 products:
Dog (Canine) Monoclonal NINJ1 Primary Antibody for IF, IP - ABIN968171
Araki, Milbrandt: Ninjurin, a novel adhesion molecule, is induced by nerve injury and promotes axonal growth. in Neuron 1996
Show all 3 references for ABIN968171
Human Polyclonal NINJ1 Primary Antibody for IF (p), IHC (p) - ABIN1386415
Matsuki, Kabara, Saito, Shimamura, Minoshima, Nishimura, Aonuma, Takehara, Hasebe, Kawabe: Ninjurin1 Is a Novel Factor to Regulate Angiogenesis Through the Function of Pericytes. in Circulation journal : official journal of the Japanese Circulation Society 2015
High ninjurin 1 expression is associated with Prostate Cancer.
Ninj1 expression was detected in both HSMCs and HUVECs, and was higher in the former. Ninj1 expression was enhanced by hypoxia in HUVECs.
Ninj1 modulates TLR4 (show TLR4 Antibodies) signaling, which itself plays a major role in systemic inflammatory response syndrome and sepsis.
The NINJ1 gene polymorphism plays an important role in nerve degeneration in leprosy patients.
Data show an important cell-specific role for Ninjurin-1 in the transmigration of inflammatory APCs (show APCS Antibodies) across the BBB (show ALMS1 Antibodies).
Our data suggests that Ninjurin 1 asp110ala SNP could be a valuable genetic marker of nerve damage in leprosy.
Ninj1 negatively regulates the formation of neovessels, that is, the EC-tube, by reducing the trophic effects of capillary pericytes.
Inhibition of Ninjurin 1 restores erectile function through dual angiogenic and neurotrophic effects in the diabetic mouse.
The dual neurotrophic and angiogenic effects of Ninj1 blockade may provide a good opportunity for treating erectile dysfunction resulting from radical prostatectomy.
Ninj1 plays a role in p53 (show TP53 Antibodies)-mediated tumor suppression in addition to nerve regeneration.
the N-terminal ectodomain of mouse Ninjurin1, which may act as a chemoattractant, is cleaved by MMP9 (show MMP9 Antibodies).
Ninj1 protein has an important role in vascular regression mediated by macrophages during early eye development and suggest the biological relevance of Ninj1 on vascular homeostasis.
The ninjurin protein is upregulated after nerve injury both in dorsal root ganglion neurons and in Schwann cells (Araki and Milbrandt, 1996
, nerve injury-induced protein 1
, nerve injury-induced protein-1