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Cell surface receptor for SEMA4A and for class 3 semaphorins, such as SEMA3A, SEMA3C and SEMA3E. Additionally we are shipping Plexin D1 Proteins (4) and Plexin D1 Kits (1) and many more products for this protein.
Showing 10 out of 67 products:
Human Polyclonal PLXND1 Primary Antibody for FACS, IHC - ABIN4900364
Lu, Evans, Hirano, Liu: Dual role for Islet-1 in promoting striatonigral and repressing striatopallidal genetic programs to specify striatonigral cell identity. in Proceedings of the National Academy of Sciences of the United States of America 2014
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Human Polyclonal PLXND1 Primary Antibody for IHC, ELISA - ABIN185358
Gu, Yoshida, Livet, Reimert, Mann, Merte, Henderson, Jessell, Kolodkin, Ginty: Semaphorin 3E and plexin-D1 control vascular pattern independently of neuropilins. in Science (New York, N.Y.) 2005
Show all 2 references for ABIN185358
Human Polyclonal PLXND1 Primary Antibody for ELISA, IHC - ABIN268775
Parikh, Eisner, Venugopalan, Yang, Lam, Bhattacharya: Proteomic analyses of corneal tissue subjected to alkali exposure. in Investigative ophthalmology & visual science 2011
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Human Polyclonal PLXND1 Primary Antibody for EIA - ABIN358510
van der Zwaag, Hellemons, Leenders, Burbach, Brunner, Padberg, Van Bokhoven: PLEXIN-D1, a novel plexin family member, is expressed in vascular endothelium and the central nervous system during mouse embryogenesis. in Developmental dynamics : an official publication of the American Association of Anatomists 2002
Plexin D1 plays a role in collagen contraction in human lung fibroblasts.
Findings suggest that Plexin-D1/class III semaphorin (Sema3E (show SEMA3E Antibodies)) axis is triggered in systemic sclerosis (SSc (show CYP11A1 Antibodies)) endothelium.
finding that PLXND1 and REV3L (show REV3L Antibodies) mutations are responsible for a proportion of MBS (show PPP1R12A Antibodies) patients suggests that de novo mutations in other genes might account for other MBS (show PPP1R12A Antibodies) patients
The data indicate that Plexin-D1 operates in a cell context-specific fashion, mediating different synaptogenic outcomes depending upon neuron type.
Plxnd1 is a novel regulator of VAT growth, body fat distribution, and insulin (show INS Antibodies) sensitivity in both zebrafish and humans
The identification and characterization of SH3BP1 (show SH3BP1 Antibodies) as a novel downstream effector of Sema3E (show SEMA3E Antibodies)-PlexinD1 provides an explanation for how extracellular signals are translated into cytoskeletal changes and unique cell behavior.
It is therefore suggested that SEMA3C (show SEMA3C Antibodies) signaling, propagated through the heterodimer receptor plexin-D1/neuropilin (show NRP1 Antibodies), is important for truncus arteriosus septation
A critical role of Sema3E (show SEMA3E Antibodies)/Plexin D1 interaction in tumor resistance to apoptosis.
Strong expression of plexD1 was detected in endothelial cells of cervical cancer samples, yet no expression was seen in endothelial cells of normal cervical tissues, which suggests a potential role of PlexD1 in cervical cancer-associated angiogenesis
analysis of Sema3E (show SEMA3E Antibodies)/plexin-D1 mediated epithelial-to-mesenchymal transition in ovarian endometrioid cancer
Our results demonstrate that sema3e (show SEMA3E Antibodies)/plexin D1 modulates IS formation and Ag-scanning activities of thymocytes within thymic tissues.
Sema3E (show SEMA3E Antibodies)/PlexinD1 signaling controls the motogenic potential of Cajal-Retzius (CR) cells in vitro and in vivo. Absence of Sema3E (show SEMA3E Antibodies)/PlexinD1 signalling increased the migratory properties of CR cells.
Sema3E (show SEMA3E Antibodies)-PlexinD1 signaling is involved in the development of CNV. Stimulation of the pathway has therapeutic potential for CNV.
Proprioceptive sensory afferents that express PlexinD1 avoid forming monosynaptic connections with neurons in Sema3E (show SEMA3E Antibodies)(+) motor pools yet are able to form direct connections with neurons in Sema3E (show SEMA3E Antibodies)(off) motor pools.
beta1 integrin adhesion is controlled by the plexinD1-sema3E (show SEMA3E Antibodies) axis in mice
Data an association between Plexin-B2 (show PLXNB2 Antibodies) and Plexin-D1 with the negative regulation of IL-12 (show IL12A Antibodies)/IL-23p40 in DCs.
This study demonstrated that Sema3E (show SEMA3E Antibodies) and Plexin-D1 specify the degree of glutamatergic connectivity between a specific source and target in the complex circuitry of the basal ganglia.
a novel role of Sema3E-Plexin-D1 function in modulating angiogenesis via a VEGF-induced feedback mechanism
Sema3E (show SEMA3E Antibodies)-PlexinD1 signaling selectively suppresses disoriented angiogenesis in ischemic retinopathy in mice
Plexin-D1 is expressed by B lymphocytes. PlxnD1 deficient B cells fail to participate efficiently in the GC reaction. And, recall immune responses are attenuated in the absence of plexin-D1, leading to defective Ab production.
Somite expression of known vascular guidance cues, efnb2, sema3a2, and plexinD1 are disrupted, suggesting that the inter-somitic vessel vascular phenotype is due to disruption of these cues.
We show that proper blood vessel pathfinding requires the endothelial receptor PlexinD1 and semaphorin signals, and we identify mutations in plexinD1 in the zebrafish vascular patterning mutant out of bounds
Loss of plxnB2 (show PLXNB2 Antibodies) results in delayed ISV sprouting identical to that seen in sema3e (show SEMA3A Antibodies) morphants, while loss of plexinD1 in out of bounds (obd) mutants results in precocious ISV sprouting.
Cell surface receptor for SEMA4A and for class 3 semaphorins, such as SEMA3A, SEMA3C and SEMA3E. Plays an important role in cell-cell signaling, and in regulating the migration of a wide spectrum of cell types. Regulates the migration of thymocytes in the medulla. Regulates endothelial cell migration. Plays an important role in ensuring the specificity of synapse formation. Mediates anti-angiogenic signaling in response to SEMA3E. Required for normal development of the heart and vasculature.
, out of bounds