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Regulator of G protein signaling (RGS) family members are regulatory molecules that act as GTPase activating proteins (GAPs) for G alpha subunits of heterotrimeric G proteins. Additionally we are shipping RGS2 Proteins (16) and RGS2 Kits (1) and many more products for this protein.
Showing 10 out of 82 products:
Human Monoclonal RGS2 Primary Antibody for IHC (p), ELISA - ABIN562644
Hoemme, Peerzada, Behre, Wang, McClelland, Nieselt, Zschunke, Disselhoff, Agrawal, Isken, Tidow, Berdel, Serve, Müller-Tidow: Chromatin modifications induced by PML-RARalpha repress critical targets in leukemogenesis as analyzed by ChIP-Chip. in Blood 2008
Show all 4 references for ABIN562644
Human Polyclonal RGS2 Primary Antibody for WB - ABIN1881749
Bailey, Xie, Do, Montpetit, Diaz, Mohan, Keavney, Yusuf, Gerstein, Engert, Anand: Variation at the NFATC2 locus increases the risk of thiazolidinedione-induced edema in the Diabetes REduction Assessment with ramipril and rosiglitazone Medication (DREAM) study. in Diabetes Care 2010
Show all 2 references for ABIN1881749
Gonadotropin-dependent up-regulation of RGS2 in equine and bovine preovulatory follicles and regulatory controls involved in RGS2 gene expression in granulosa cells.
RGS2 deficiency decreases uterine artery blood flow by increasing myogenic tone at least partly through prolonged G protein activation
Rgs12 (show RGS12 Antibodies) has a crucial role in the function of bone-resorbing OCs, particularly in preclinical models of pathological bone loss associated with inflammation-induced bone loss and postmenopausal osteoporosis.
RGS2 deficiency impairs renal function and autoregulation by increasing renal vascular resistance and reducing renal blood flow.
FBXO44 (show FBXO44 Antibodies)-mediated degradation of RGS2 protein uniquely depends on a Cul4B (show CUL4B Antibodies)/DDB1 (show DDB1 Antibodies) complex.
Regulator of G-protein signaling 2 (RGS2) suppresses premature calcium release in mouse eggs.
genetic variations and increased inflammatory cytokines can lead to RGS2 repression, which exacerbates AHR (show AHR Antibodies) and airway remodeling in asthma.
RGS2 and RGS4 are new interacting partners that play key roles in G protein coupling to negatively regulate kappa-OmicronR signaling.
RGS2-deficient VSMC exhibit higher rates of proliferation and coordinate plasticity of Ca(2 (show CA2 Antibodies)+)-handling mechanisms in response to PDGF (show PDGFA Antibodies) stimulation.
Regulator of G protein signaling 2 deficiency accelerates the progression of kidney fibrosis
These findins indicate that urotensin II (show UTS2 Antibodies)-induced calcium metabolism results in glomerular mesangial cell contraction and suggest that urotensin receptor (show UTS2R Antibodies) activation recruts RGS2 to the plasma membrane.
The RGS2 (-391, C>G) genetic polymorphism may serve as a biomarker to predict a patient's response to antihypertensive drug therapy
MIR4717 regulates human RGS2 and contributes to the genetic risk towards anxiety-related traits.
RGS2 polymorphisms were found to be associated with anxiety disorders and dimensional as well as intermediate phenotypes of anxiety.
RGS2 localizes to the mitotic spindle in a Nek7 (show NEK7 Antibodies)-dependent manner, and along with Nek7 (show NEK7 Antibodies) contributes to spindle morphology and mitotic spindle pole integrity.
Data show that regulator of G protein signaling 2 (RGS2) was stabilized by deubiquitinase monocyte chemotactic protein-induced protein 1 (MCPIP1 (show ZC3H12A Antibodies)).
Epigenetic repression of RGS2 by UHRF1 (show UHRF1 Antibodies) contributes to bladder cancer progression. UHRF1 (show UHRF1 Antibodies) inhibits RGS2 expression by increasing the methylation of CpG nucleotides of the RGS2 promoter.
ACE (show ACE Antibodies) and RGS2 genotypes are not associated with the development of hypertension in patients with type 1 diabetes mellitus.
This work highlights the role of RGS2 as a key regulator of LRRK2 (show LRRK2 Antibodies) activity, function and neuronal toxicity.
Regulator of G protein signaling (RGS) family members are regulatory molecules that act as GTPase activating proteins (GAPs) for G alpha subunits of heterotrimeric G proteins. RGS proteins are able to deactivate G protein subunits of the Gi alpha, Go alpha and Gq alpha subtypes. They drive G proteins into their inactive GDP-bound forms. Regulator of G protein signaling 2 belongs to this family. The protein acts as a mediator of myeloid differentiation and may play a role in leukemogenesis.
regulator of G-protein signaling 2
, regulator of G-protein signalling 2
, regulator of G-protein signaling protein 2
, regulator of G-protein signalling 2, 24kDa
, G0 to G1 switch regulatory 8, 24kD
, G0/G1 switch regulatory protein 8
, cell growth-inhibiting gene 31 protein
, cell growth-inhibiting protein 31