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SOD2 is a member of the iron/manganese superoxide dismutase family. Additionally we are shipping Superoxide Dismutase 2, Mitochondrial Proteins (48) and Superoxide Dismutase 2, Mitochondrial Kits (41) and many more products for this protein.
Showing 10 out of 277 products:
Chicken Polyclonal SOD2 Primary Antibody for ICC, FACS - ABIN151490
Dumont, Wille, Stack, Calingasan, Beal, Lin: Reduction of oxidative stress, amyloid deposition, and memory deficit by manganese superoxide dismutase overexpression in a transgenic mouse model of Alzheimer's disease. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2009
Show all 15 references for ABIN151490
Chicken Polyclonal SOD2 Primary Antibody for IP, IHC - ABIN361648
Adachi, Ohta, Yamada, Futenma, Kato, Hirano: Quantitative analysis of extracellular-superoxide dismutase in serum and urine by ELISA with monoclonal antibody. in Clinica chimica acta; international journal of clinical chemistry 1993
Show all 10 references for ABIN361648
Chicken Polyclonal SOD2 Primary Antibody for ICC, IF - ABIN361656
Gao, Flores, Leff, Bose, McCord: Synthesis and anti-inflammatory activity of a chimeric recombinant superoxide dismutase: SOD2/3. in American journal of physiology. Lung cellular and molecular physiology 2003
Show all 10 references for ABIN361656
Chicken Polyclonal SOD2 Primary Antibody for EIA, IHC (fro) - ABIN264859
Liu, Kato, Nakata, Kogure, Kato: An immunohistochemical study of copper/zinc superoxide dismutase and manganese superoxide dismutase in rat hippocampus after transient cerebral ischemia. in Brain research 1993
Show all 5 references for ABIN264859
Chicken Polyclonal SOD2 Primary Antibody for IHC (fro), IHC (p) - ABIN151468
Dumont, Stack, Elipenahli, Jainuddin, Gerges, Starkova, Yang, Starkov, Beal: Behavioral deficit, oxidative stress, and mitochondrial dysfunction precede tau pathology in P301S transgenic mice. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2011
Show all 5 references for ABIN151468
Dog (Canine) Monoclonal SOD2 Primary Antibody for IF, WB - ABIN968602
Ambrosone, Freudenheim, Thompson, Bowman, Vena, Marshall, Graham, Laughlin, Nemoto, Shields: Manganese superoxide dismutase (MnSOD) genetic polymorphisms, dietary antioxidants, and risk of breast cancer. in Cancer research 1999
Show all 5 references for ABIN968602
Human Polyclonal SOD2 Primary Antibody for IF (p), IHC (p) - ABIN737361
Khan, Chen, Wan, Tania, Xu, Chen, Zhang: Regulatory effects of resveratrol on antioxidant enzymes: a mechanism of growth inhibition and apoptosis induction in cancer cells. in Molecules and cells 2013
Show all 3 references for ABIN737361
Chicken Polyclonal SOD2 Primary Antibody for EIA, IHC (fro) - ABIN264861
Kurobe, Inagaki, Kato: Sensitive enzyme immunoassay for human Mn superoxide dismutase. in Clinica chimica acta; international journal of clinical chemistry 1991
Show all 3 references for ABIN264861
Mouse (Murine) Polyclonal SOD2 Primary Antibody for ICC, IHC (fro) - ABIN1078543
Zhao, Li, Han, Yue, Shi, Wang, Guo, Lu: DDAH1 deficiency promotes intracellular oxidative stress and cell apoptosis via a miR-21-dependent pathway in mouse embryonic fibroblasts. in Free radical biology & medicine 2016
Human Polyclonal SOD2 Primary Antibody for WB - ABIN2787727
Martin, Barker, Doll, Pine, Mechanic, Bowman, Harris, Hein: Manganese superoxide dismutase gene coding region polymorphisms lack clinical incidence in general population. in DNA and cell biology 2008
Prion (show PRNP Antibodies) propagation exacerbates an apoptotic pathway whereby mitochondrial dysfunction follows mislocalisation of SOD2 to cytosolic caspases permitting its degradation.
Depletion of superoxide dismutases promotes muscular and neuronal ROS (show ROS1 Antibodies) accumulation which may have a significant effect on age-dependent impairment of the Drosophila adults.
Paraquat exposure, but not Sod2 knockdown, resulted in increased carbonylated protein relative abundance.
The functional SOD1 (show SOD1 Antibodies) and SOD2 genes knockout and their overexpression in neurons and glial tissue increase the sensitivity of Drosophila melanogaster to oxidative stress conditions.
ocytes lacking maternal SOD2 protein develop into adults just like normal SOD2-containing oocytes suggesting that maternal SOD2-mediated protection against mitochondrial ROS (show ROS1 Antibodies) is not essential for oocyte viability
Muscles appear to be more sensitive to superoxide attack relative to the neurons and such overt phenotypes observed in SOD2-deficient animals can be directly attributed to the muscle.
Overexpression of Cu,ZnSOD (show SOD1 Antibodies) and MnSOD in transgenic Drosophila.
Ablation of mitochondrial SOD2 through expression of a GAL4 (show LGALS4 Antibodies)-regulated, inverted-repeat Sod2 RNA-interference causes increased endogenous oxidative stress, loss of respiratory chain & TCA cycle components,& early-onset mortality in young adults.
Effects of overexpression of copper-zinc and manganese superoxide dismutases, catalase, and thioredoxin reductase genes on longevity.
The chromosomal deficiency Df(2R)017 significantly up-regulated MnSOD mRNA by 1.7-fold. Deficiency in four other genomic intervals, Df(1)ct-J4, Df(2L)BSC4, Df(3L)66C-G28 and Df(3R)Scr (show SCRIB Antibodies), down-regulated MnSOD expression.
SOD1 (show SOD1 Antibodies) and SOD2 provide independent protection to compartment-specific protein iron-sulfur clusters against attack by superoxide generated under oxidative stress
The effects of hypericin and methoxyestradiol on the expression of SOD2 in MCF-7 cells and the sensitivity of these cells to photodynamic therapy are reported.
Mn-SOD overexpression is a common event in colorectal cancer
variants of GPx1 (show GPX1 Antibodies) and MnSOD should not be considered as a risk factor of laryngeal squamous cell carcinoma.
There were no significant between-group differences in the CC genotype distribution of the p.Ala16Val (C>T) polymorphism in the SOD2 gene.
Results showed no statistically significant difference between the vitiligo (show MITF Antibodies) and MnSOD Ala-9Val and GPx1 (show GPX1 Antibodies) Pro198Leu gene polymorphisms in Turkish population.
Authors identify miR (show MLXIP Antibodies)-146a as a potential tumor suppressor in patients with EOC. miR (show MLXIP Antibodies)-146a downregulates the expression of SOD2 and enhances ROS (show ROS1 Antibodies) generation, leading to increased apoptosis, inhibition of proliferation, and enhanced sensitivity to chemotherapy.
endogenous Alu RNA accumulation in hyperglycemic endothelial cells provokes endothelial oxidative stress and dysfunction by suppressing SOD2 and eNOS expression at both transcription and translation levels via NFkappaB signaling pathway.
The MnSOD, GPX1 (show GPX1 Antibodies) and CAT genotypes and allele frequencies of acute kidney injury patients did not differ significantly from those of healthy controls.
These results suggest that the MnSOD Ala-9Val polymorphism may not influence smoking status in a Chinese male schizophrenia population.
In this review, we will conglomerate the current aspects by which MnSOD can contribute to embryonic stem cells' and adult stem cells' functions and interpret the necessity of understanding MnSOD for further stem cell mediated applications
show that ischemia markedly potentiated the expression of arginase-1 (show ARG1 Antibodies), and also induced the SOD2 in the wound tissue.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (show IL8 Antibodies) and PTGS2 (show PTGS2 Antibodies), and decreased the expression of SOD2, GPX3 (show GPX3 Antibodies), DAB2 (show DAB2 Antibodies), and NR3C1 (show NR3C1 Antibodies). TNF (show TNF Antibodies) and IL6 (show IL6 Antibodies) levels were also decreased while those of NAMPT (show NAMPT Antibodies) were unaffected.
Data suggest that during ectogenesis of blastocysts infected with bovine Herpesvirus type 5, expression of SOD2 (Mn) remains high indicating intense mitochondrial activity; this effect may be involved in inhibition of apoptosis in infected embryos.
expression profile of SOD2 in follicles: oocytes (SOD2 restricted to ooplasm (show NLRP5 Antibodies)); cumulus cells (expressed some SOD2); follicular fluid (small follicles show increased amounts of SOD2 in comparison with large follicles)
Shear stress influences spatial variations in vascular Mn-SOD expression with implications for LDL nitration.
The expression of SOD1 (show SOD1 Antibodies) and SOD2 through the course of the estrous cycle is reported.
Heart mitochondrial nucleoids contained SOD2, aortic endothelial cell mitochondrial nucleoids did not
Thus, a ROS (show ROS1 Antibodies)-dependent epigenetic positive regulation of Sod2 gene expression likely represents a defense mechanism prolonging eNOS (show NOS3 Antibodies) function in aging mouse femoral arteries.
The results indicated that the inconsistency between Mn SOD expression and its activity might contribute to the development of recognition dysfunction induced by chronic Al overload.
Evidence for a tumor suppressive role of MnSOD in liver, where the Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) and hypoxia pathway may be crucial elements.
SOD2 overexpression in cardiomyocytes enhances mitochondrial function and metabolic vasodilation, leading to a phenotype of supernormal cardiac function.
Pluripotency transcription factors Nanog (show NANOG Antibodies) and Oct4 (show POU5F1 Antibodies) positively modulate Sod2 gene transcription in induced pluripotent stem cells.
Data show that mechanical overloading elevated mitochondrial superoxide generation and downregulated superoxide dismutase 2 (Sod2) expression in knee chondrocytes.
Data show that tissue specific deletion of manganese superoxide dismutase (Sod2) caused elevated signs of oxidative stress, retinal pigment epithelium (RPE (show RPE Antibodies)) dysfunction and showed some key features of age-related macular degeneration (AMD (show AMD1 Antibodies)). .
Mutated myocilin (show MYOC Antibodies) and heterozygous Sod2 deficiency act synergistically in a mouse model of open-angle glaucoma
Data indicate sirtuin 3 (Sirt3 (show SIRT3 Antibodies))-manganese superoxide dismutase (MnSOD) axis as a negative component in nicotine-induced mitochondrial oxidative stress and mitochondrial DNA (mtDNA) damage.
This gene is a member of the iron/manganese superoxide dismutase family. It encodes a mitochondrial protein that forms a homotetramer and binds one manganese ion per subunit. This protein binds to the superoxide byproducts of oxidative phosphorylation and converts them to hydrogen peroxide and diatomic oxygen. Mutations in this gene have been associated with idiopathic cardiomyopathy (IDC), premature aging, sporadic motor neuron disease, and cancer. Alternate transcriptional splice variants, encoding different isoforms, have been characterized.
, manganese superoxide dismutase
, Mn Sod
, Mn superoxide dismutase
, superoxide dismutase 2, mitochondrial
, superoxide dismutase [Mn], mitochondrial
, Mn-superoxide dismutase
, mitochodnrial SOD
, mitochondrial Mn-superoxide dismutase 2
, mitochondrial MnSOD
, mitochondrial superoxide dismutase 2
, superoxide dismutase
, superoxide dismutase 2
, superoxide dismutase 2 (Mn)
, superoxide dismutase-2
, indophenoloxidase B
, manganese-containing superoxide dismutase
, mangano-superoxide dismutase
, superoxide dismutase (Mn type)
, manganous superoxide dismutase
, manganese SOD
, Superoxide dimutase 2, mitochondrial