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Human IL17F ELISA Kit for Sandwich ELISA - ABIN625014
Ding, Hu, Xu, Huang, Zhang, Li, Hu, Ni, Hou: Serum IL-17F combined with VEGF as potential diagnostic biomarkers for oral squamous cell carcinoma. in Tumour biology 2015
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Rat (Rattus) IL17F ELISA Kit for Sandwich ELISA - ABIN1706083
Wang, Zhang, Yang, Nurieva, Chang, Ojeda, Kang, Schluns, Gui, Jetten, Dong: Transcription of Il17 and Il17f is controlled by conserved noncoding sequence 2. in Immunity 2012
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Rat (Rattus) IL17F ELISA Kit for Sandwich ELISA - ABIN432432
You, Zhang, Niu, Zhu, Wang, Yue, Sun: Expression of IL-17A and IL-17F in lipopolysaccharide-induced acute lung injury and the counteraction of anisodamine or methylprednisolone. in Cytokine 2014
our data suggest that IL-20 (show IL20 ELISA Kits) subfamily cytokines, particularly IL-20 (show IL20 ELISA Kits), IL-22 (show IL22 ELISA Kits), and IL-24 (show IL24 ELISA Kits), might provide therapeutic benefit for patients with Diabetic foot ulcers (DFU) .
this study shows that IL-24 (show IL24 ELISA Kits) promotes Pseudomonas aeruginosa keratitis through the suppression of early protective mucosal immunity
Loss of IL-24 (show IL24 ELISA Kits) expression is associated with breast cancer.
our results indicate that Th17 cells contribute to the airway vascular remodeling in asthma by mediating EPC (show TCF21 ELISA Kits) chemotaxis, as well as PMVEC tube formation, via IL-17A (show IL17A ELISA Kits) rather than IL-17F.
Data indicate that IL-17A (show IL17A ELISA Kits) repressed IL-17F secretion in vitro and in vivo.
A psoriasis-like skin inflammation in mice with epidermis-specific inhibition of NF-kappaB (show NFKB1 ELISA Kits) was triggered by TNFR1 (show TNFRSF1A ELISA Kits)-dependent upregulation of interleukin-24 (show IL24 ELISA Kits) & activation of STAT3 (show STAT3 ELISA Kits) signaling in keratinocytes.
The concentrations in plasma of IL-17A (show IL17A ELISA Kits), IL-17F, and the IL-17AF heterodimer all were increased greatly in mice after endotoxemia.
IL-24 (show IL24 ELISA Kits) suppresses the growth of normal vascular smooth muscle cells by inhibiting hydrogen peroxide-induced reactive oxygen species (ROS (show ROS1 ELISA Kits)) production through the regulation of mitochondrial ROS (show ROS1 ELISA Kits) production and expression of antioxidant enzymes
IL-17A (show IL17A ELISA Kits) and IL-17F secreted by CD4 (show CD4 ELISA Kits)+Th17 cells specific to lung self-antigens are critical mediators of autoimmunity leading to the pathogenesis of obliterative airway disease.
our study provides the evidence that functional IL-23R (show IL23R ELISA Kits) rs1884444 G/T and IL-17F rs763780 A/G polymorphisms may be a new genetic susceptibility factor to SLE, especially in the Polish population.
IL23R (show IL23R ELISA Kits) rs10889677 and IL17A (show IL17A ELISA Kits) rs2275913 were not associated with the susceptibility to Necrotizing enterocolitis (NEC). In conclusion, data suggest that a variant of IL17F (rs763780) may contribute to the development of NEC.
Our results confirmed IL17A (show IL17A ELISA Kits) and IL17F as potential candidate genes involved in RA. They play pivoting roles in the susceptibility and in clinical features of RA disease. Responses to RA treatments are differently conditioned by polymorphisms in IL17A (show IL17A ELISA Kits) and IL17F genes.
IL17A (show IL17A ELISA Kits) and IL17F gene polymorphism are not the important factors associated with susceptibility and some clinical parameters of rheumatoid arthritis in a Polish population.
we aimed to investigate the associations between the 7383A/G and 7488A/G polymorphisms of the interleukin (IL)-17F gene with disease activity and clinical outcomes in Turkish patients with ankylosing spondylitis
Using an in-vitro migration assay, B cells were shown to migrate towards both IL-17A (show IL17A ELISA Kits) and IL-17F. These observations indicate a direct chemotactic effect of IL-17 (show IL17A ELISA Kits) cytokines on primary peripheral blood B cells with higher effect being on asthmatic B cells.
IL-17 (show IL17A ELISA Kits)-related cytokines expression was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbation, suggesting a pathogenic role of IL-17F in frequent exacerbators.
IL-17A (-197G/A) and IL-17F (7488T/C) SNPs were not associated with susceptibility to rheumatoid arthritis or secondary Sjogrens syndrome (sSS, p > 0.05 for both SNPs). In addition, they did not influence RA activity or clinical markers of SS.
It can be stated that the IL17A (show IL17A ELISA Kits) and IL17F polymorphisms are not markers of susceptibility to psoriasis. However, the IL17F polymorphism may affect the response to NB-UVB therapy.
Elevated aAbs against IL-17F correlate with disease activity in patients with early rheumatoid arthritis.
The protein encoded by this gene is a cytokine that shares sequence similarity with IL17. This cytokine is expressed by activated T cells, and has been shown to stimulate the production of several other cytokines, including IL6, IL8, and CSF2/GM_CSF. This cytokine is also found to inhibit the angiogenesis of endothelial cells and induce endothelial cells to produce IL2, TGFB1/TGFB, and monocyte chemoattractant protein-1.
, IL-4-induced secreted protein
, melanoma differentiation-associated gene 7 protein
, th2-specific cytokine FISP
, cytokine-like protein Mob-5
, interleukin 17F
, cytokine ML-1
, mutant IL-17F