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Meng, Ganesan Adaikan, Srilatha: Hydrogen sulfide promotes nitric oxide production in corpus cavernosum by enhancing expression of endothelial nitric oxide synthase. in International journal of impotence research 2013
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Here we demonstrate that the Drosophila relaxin (show 0 ELISA Kits) receptor homolog Lgr3 (show TSHR ELISA Kits), a leucine-rich repeat-containing G-protein-coupled receptor (show GPBAR1 ELISA Kits), is required for Dilp8-dependent growth coordination and developmental delay during the regeneration checkpoint; we demonstrate that Lgr3 (show TSHR ELISA Kits) activity in both the CNS and PG is necessary for NOS activation in the PG following damage
Nitric Oxide Synthase Regulates Growth Coordination During Drosophila melanogaster Imaginal Disc Regeneration
Hb9 (show MNX1 ELISA Kits) governs neuronal specification and differentiation and activates expression of Nos and fd59a in the Drosophila CNS
Nitric oxide synthase is not essential for Drosophila development.
biochemical characterization of Drosophila nitric oxide synthase
Oxygenase domain of Drosophila melanogaster Nos: unique kinetic parameters enable a more efficient NO release.
Reductase domain of Drosophila melanogaster Nos: redox transformations, regulation, and similarity to mamalian homologues.
These data suggest that BmNOS has an important role in the early embryonic development of the B. mori.
no significant difference in frequency of NOS2-1659C/T polymorphism was observed between patients and controls. None of the studied SNPs were associated with erosive disease, seropositivity or extra-articular manifestations. The -277A/G and -1026 G/T promoter polymorphisms in iNOS (show NOS2 ELISA Kits) may confer susceptibility to rheumatoid arthritis (RA). in South Indian Tamils.
This is the first reported evidence for NO-enhanced bystander aggressiveness in the context of PDT (show TWIST1 ELISA Kits). In the clinical setting, such effects could be averted through pharmacologic use of iNOS (show NOS2 ELISA Kits) inhibitors as non-ionizing photodynamic therapy adjuvants
This increase was inhibited in the presence of the nonspecific iNOS (show NOS2 ELISA Kits) inhibitor aminoguanidine (AG).
our study shows that the expression of iNOS is increased in both central airways and the alveolar parenchyma, but not in BAL cells, in uncontrolled asthmatics as compared to controlled asthmatics and healthy controls.
We found that lowering the glucose concentration increased expression of genes coding for inducible nitric oxide syntheas, NOS2 and NOS2A (show NOS2 ELISA Kits) resulting in enhanced production of nitric oxide
Downregulation of inducible NO synthetase (iNOS (show NOS2 ELISA Kits)) resulted in downregulation of heme oxygenase 1 (HO-1 (show HMOX1 ELISA Kits)), and, conversely, upregulation of iNOS (show NOS2 ELISA Kits) enhanced HO-1 (show HMOX1 ELISA Kits) activity.
Data suggest that NOS1 and NOS2 play roles in stress-induced surge in nitric oxide (NO) production; NO serves as mediator for development of secondary neurological disorders associated with stress such as anxiety and anxiety disorders. [REVIEW]
ATM (show ATM ELISA Kits)-reactive oxygen species-iNOS (show NOS2 ELISA Kits) axis regulates nitric oxide mediated cellular senescence.
The risk of developing chronic pancreatitis is not increased by the presence of the iNOS (show NOS2 ELISA Kits)-2087A>G polymorphism.
NOS2 rs2779248, NOS2 rs1137933, and NOS3 rs3918188 genetic polymorphisms are potentially related to the susceptibility to type 2 diabetes mellitus (T2DM), and the rs1800783 polymorphism might be considered as genetic risk factors for diabetic nephropathy.
Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. This gene encodes a nitric oxide synthase which is expressed in liver and is inducible by a combination of lipopolysaccharide and certain cytokines. Three related pseudogenes are located within the Smith-Magenis syndrome region on chromosome 17.
, NO synthase
, nitric oixide synthase
, nitric oxide synthase
, Nitric oxide synthase, brain
, inducible nitric oxide synthase-like protein
, NOS type II
, NOS, type II
, hepatocyte NOS
, inducible NO synthase
, inducible NOS
, nitric oxide synthase 2A (inducible, hepatocytes)
, nitric oxide synthase, inducible
, nitric oxide synthase, macrophage
, peptidyl-cysteine S-nitrosylase NOS2