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Dido1 induces the expression of Integrin alphaV, thereby promoting the attachment, migration, invasion and apoptosis resistance of melanoma cells.
Data show that CSE1L (show CSE1L Proteins), DIDO1 and RBM39 (show RBM39 Proteins) mRNA expression levels correlated with chromosome 20q DNA copy number status.
Gambogic acid induces the apoptosis of Raji cells through DIO-1 upregulation, nuclear translocation, Bcl-xL (show BCL2L1 Proteins) downregulation and caspase 3 (show CASP3 Proteins) activation.
Novel transcription factors identified in human CD34 antigen (show CD34 Proteins)-positive hematopoietic stem cells.
Dido3, the largest splice variant of the Dido gene, is a centrosome-associated protein whose disruption leads to supernumerary centrosomes, failure to maintain cellular mitotic arrest, and early degradation of the mitotic checkpoint (show BUB3 Proteins) protein BubR1 (show BUB1B Proteins).
Gambogic acid induces DIDO1-mediated apoptosis in Jurkat T cells.
Embryonic stem cells mainly express DIDO3 and their differentiation after leukemia inhibitory factor (show LIF Proteins) withdrawal requires DIDO1 expression. DIDO3 regulates DIDO1 expression.
Death inducer obliterator (Dido3)-dependent targeting of histone deacetylase 6 (HDAC6 (show HDAC6 Proteins)) is a key determinant of cilium size in growth-arrested cells
Dido1 is able to regulate self-renewal of mouse embryonic stem cells.
Dido3 PHD (show PDC Proteins) interacts with histone H3K4me3 and regulates expression of stemness genes in embryonic stem cells.
loss of Dido3 expression compromises differentiation of embryonic stem cells in vitro and of epiblast cells in vivo, resulting in early embryonic death at around day 8.5 of gestation
Dido3, the largest splice variant of the Dido gene, is a centrosome-associated protein (show BLOC1S2 Proteins) whose disruption leads to supernumerary centrosomes, failure to maintain cellular mitotic arrest, and early degradation of the mitotic checkpoint (show BUB3 Proteins) protein BubR1 (show BUB1B Proteins).
results indicate that histone H3 (show HIST3H3 Proteins) lysine 4 demethylation modulates DIDO3 localization in meiosis and suggest epigenetic regulation of the synaptonemal complex
Apoptosis, a major form of cell death, is an efficient mechanism for eliminating unwanted cells and is of central importance for development and homeostasis in metazoan animals. In mice, the death inducer-obliterator-1 gene is upregulated by apoptotic signals and encodes a cytoplasmic protein that translocates to the nucleus upon apoptotic signal activation. When overexpressed, the mouse protein induced apoptosis in cell lines growing in vitro. This gene is similar to the mouse gene and therefore is thought to be involved in apoptosis. Alternatively spliced transcripts have been found for this gene, encoding multiple isoforms.
death-associated transcription factor 1
, death-inducer obliterator 1
, death inducer-obliterator-2
, death inducer-obliterator-3
, death associated transcription factor 1
, death inducer-obliterator 1
, death-inducer obliterator 1-like