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Bat Polyclonal Vip Primary Antibody for IEM, ICC - ABIN617890
Schultea, Dees, Ojeda: Postnatal development of sympathetic and sensory innervation of the rhesus monkey ovary. in Biology of reproduction 1993
Show all 494 references for ABIN617890
Human Polyclonal Vip Primary Antibody for FACS, IHC (p) - ABIN390468
Lu, Owyang: Secretin-induced gastric relaxation is mediated by vasoactive intestinal polypeptide and prostaglandin pathways. in Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society 2009
Human Polyclonal Vip Primary Antibody for IF (p), IHC (p) - ABIN724565
Wieck, El-Nachef, Hou, Spurrier, Holoyda, Schall, Garcia Mojica, Collins, Trecartin, Cheng, Frykman, Grikscheit: Human and Murine Tissue-Engineered Colon Exhibit Diverse Neuronal Subtypes and Can Be Populated by Enteric Nervous System Progenitor Cells When Donor Colon Is Aganglionic. in Tissue engineering. Part A 2015
Human Polyclonal Vip Primary Antibody for IHC, ELISA - ABIN1585657
Lath, Galambos, Rocha, Malek, Gittes, Potoka: Defective pulmonary innervation and autonomic imbalance in congenital diaphragmatic hernia. in American journal of physiology. Lung cellular and molecular physiology 2012
Cat (Feline) Polyclonal Vip Primary Antibody for IHC (fro), IHC (p) - ABIN113519
Karila, Holmgren: Anally projecting neurons exhibiting immunoreactivity to galanin, nitric oxide synthase and vasoactive intestinal peptide, detected by confocal laser scanning microscopy, in the intestine of the Atlantic cod, Gadus morhua in Cell and tissue research 1997
VIP and CRF (show CRH Antibodies) reduce ADAMTS (show ADAMTS13 Antibodies) expression and function in osteoarthritis synovial fibroblasts.
a differential effect of VIP on the expression of TNFalpha (show TNF Antibodies) and IL-6 (show IL6 Antibodies) receptors, since VIP was only able to decreased expression in LPS (show IRF6 Antibodies)-stimulated monocytes but not in 4/74-infected monocytes.
VIP promotes Th17 polarization of memory Th cells in early arthritis.
The circadian rhythm amplitude of motor activity in both Alzheimer disease subjects and age-matched controls is correlated with the number of vasoactive intestinal peptide-expressing suprachiasmatic nucleus neurons.
Skin VIP expression, a marker of sudomotor innervation, was significantly lower in patients with familial transthyretin (show TTR Antibodies) amyloid neuropathy compared to controls.
Alterations in VIP expression may play a role in irritable bowel syndrome.
Interictal CGRP (show S100A12 Antibodies) and, to a lesser degree, VIP levels measured in peripheral blood are of great help in predicting response of chronic migraine patients to Onabotulinumtoxin type A
The antimicrobial peptide, VIP is a neuropeptide with activity against a range of microorganisms from skin, oral, respiratory and gastrointestinal tract sites.
VIP is significantly increased in the patients with chronic obstructive pulmonary disease and pulmonary hypertension.
Serum levels of vasoactive intestinal peptide as a prognostic marker in early arthritis.
High level of co-expression of PACAP (show ADCYAP1 Antibodies) with VIP, SP and CGRP (show CALCA Antibodies) in the distal ganglia of the vagus sensory perikarya directly implicates studied peptides in their functional interaction during nociceptive vagal transduction.
The present study reports for the first time on the co-localisation of CART and VIP in myenteric neurons of the porcine transverse colon.
Data show that PKA and PKC (show PKC Antibodies) pathways are involved in the differential regulation of production of the neuropeptides (Met)enkephalin, galanin (show GAL Antibodies), somatostatin (show SST Antibodies), NPY (show NPY Antibodies), and VIP.
Serum VIP was elevated significantly in the cholesterol gallstone groups compared to controls.
The intestinal concentration of VIP increased gradually during development. The levels of VIP and VIPR1 (show VIPR1 Antibodies) in liver gradually decreased during development.
majority of GnRH neurons in male and female mice express functional VIP receptors; effects of VIP on GnRH neurons do not alter across the estrous cycle
Backup mechanisms maintain PACAP (show ADCYAP1 Antibodies)/VIP-induced arterial vasodilation in PACAP (show ADCYAP1 Antibodies)-deficient mice.
in cortical VIP neurons, experience-dependent gene transcription regulates visual acuity by activating the expression of IGF1 (show IGF1 Antibodies), thus promoting the inhibition of disinhibitory neurons and affecting inhibition onto cortical pyramidal neurons
Chronic loss of VIP in mice leads to a disruption of certain but not all immunological compartments.
neuropeptides CGRP (show CALCA Antibodies) and VIP have an important role in suppressing bone resorptive activities through RANKL/OPG (show TNFSF11 Antibodies) pathway, similar to mechanical loading.
The results show in vivo a primary role for VIP chronic exposure in CFTR (show CFTR Antibodies) membrane stability and function and confirm in vitro data.
VIP signaling modulates the output from the olfactory bulb to maintain circadian rhythms in the mammalian olfactory system.
VIP neuropeptide-mediated cAMP-PKA signaling has an important role in hepatic homeostasis and cytoprotection in vivo
Loss of the VIP gene orchestrates a panoply of pathogenic genes which are detrimental to both left and right cardiac homeostasis.
Vasoactive intestinal peptide may be an essential factor for Hertwig's epithelial root sheath development.
The protein encoded by this gene belongs to the glucagon family. It stimulates myocardial contractility, causes vasodilation, increases glycogenolysis, lowers arterial blood pressure and relaxes the smooth muscle of trachea, stomach and gall bladder. Alternative splicing occurs at this locus and two transcript variants encoding distinct isoforms have been identified.
, vasoactive intestinal peptide
, histidine-isoleucine/vasoactive intestinal polypeptide
, vasoactive intestinal polypeptide type I
, vasoactive intestinal polypeptide
, vasoactive instestinal peptide
, PHI, peptide histidine isoleucine