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Bat Polyclonal Vip Primary Antibody for IEM, ICC - ABIN617890
Schultea, Dees, Ojeda: Postnatal development of sympathetic and sensory innervation of the rhesus monkey ovary. in Biology of reproduction 1993
Show all 494 Pubmed References
Human Polyclonal Vip Primary Antibody for IF (p), IHC (p) - ABIN724565
Wieck, El-Nachef, Hou, Spurrier, Holoyda, Schall, Garcia Mojica, Collins, Trecartin, Cheng, Frykman, Grikscheit: Human and Murine Tissue-Engineered Colon Exhibit Diverse Neuronal Subtypes and Can Be Populated by Enteric Nervous System Progenitor Cells When Donor Colon Is Aganglionic. in Tissue engineering. Part A 2015
Human Polyclonal Vip Primary Antibody for IHC, ELISA - ABIN1585657
Lath, Galambos, Rocha, Malek, Gittes, Potoka: Defective pulmonary innervation and autonomic imbalance in congenital diaphragmatic hernia. in American journal of physiology. Lung cellular and molecular physiology 2012
Cow (Bovine) Polyclonal Vip Primary Antibody for IHC (p) - ABIN2477078
Stojsic, Brasanac, Bilanovic, Mitrovic, Stevanovic, Boricic: Large-cell neuroendocrine carcinoma of the ampulla of Vater. in Medical oncology (Northwood, London, England) 2010
this study shows that that VIP exerts anti-inflammatory effects by inhibiting the MAPK (show MAPK1 Antibodies)-p47phox phosphorylation-NOX2 (show CYBB Antibodies) activation axis, and that VIP dampens carrageenan-induced inflammation in rats
Both mast cells and VIP appear to be important factors in the regulation of this bacterial translocation through the colonic mucosa of irritable bowel syndrome patients.
we found that VIP inhibits NFAT (show NFATC1 Antibodies) nuclear translocation in primary human pulmonary artery smooth muscle cells (PASMC). Early activation of NFATc3 (show NFATC3 Antibodies) in IPF patients may contribute to disease progression and the increase in VIP expression could be a protective compensatory mechanism
VIP and CRF (show CRH Antibodies) reduce ADAMTS (show ADAMTS13 Antibodies) expression and function in osteoarthritis synovial fibroblasts.
a differential effect of VIP on the expression of TNFalpha (show TNF Antibodies) and IL-6 (show IL6 Antibodies) receptors, since VIP was only able to decreased expression in LPS (show IRF6 Antibodies)-stimulated monocytes but not in 4/74-infected monocytes.
VIP promotes Th17 polarization of memory Th cells in early arthritis.
The circadian rhythm amplitude of motor activity in both Alzheimer disease subjects and age-matched controls is correlated with the number of vasoactive intestinal peptide-expressing suprachiasmatic nucleus neurons.
Skin VIP expression, a marker of sudomotor innervation, was significantly lower in patients with familial transthyretin (show TTR Antibodies) amyloid neuropathy compared to controls.
Alterations in VIP expression may play a role in irritable bowel syndrome.
Interictal CGRP (show S100A12 Antibodies) and, to a lesser degree, VIP levels measured in peripheral blood are of great help in predicting response of chronic migraine patients to Onabotulinumtoxin type A
High level of co-expression of PACAP (show ADCYAP1 Antibodies) with VIP, SP and CGRP (show CALCA Antibodies) in the distal ganglia of the vagus sensory perikarya directly implicates studied peptides in their functional interaction during nociceptive vagal transduction.
The present study reports for the first time on the co-localisation of CART and VIP in myenteric neurons of the porcine transverse colon.
Data show that PKA and PKC (show PKC Antibodies) pathways are involved in the differential regulation of production of the neuropeptides (Met)enkephalin, galanin (show GAL Antibodies), somatostatin (show SST Antibodies), NPY (show NPY Antibodies), and VIP.
Serum VIP was elevated significantly in the cholesterol gallstone groups compared to controls.
The intestinal concentration of VIP increased gradually during development. The levels of VIP and VIPR1 (show VIPR1 Antibodies) in liver gradually decreased during development.
PACAP (show ADCYAP1 Antibodies)/PAC1 (show ADCYAP1R1 Antibodies) signaling is important for light regulated behavior, VIP/VPAC2 (show VIPR2 Antibodies) signaling for stable clock function and both signaling pathways may play a role in shaping diurnality versus nocturnality.
The data of this study support a key role for VIP interneurons in cortical circuit development and suggest a possible contribution to pathophysiology in neurodevelopmental disorders
During arousal, VIP cells rapidly and directly inhibit pyramidal neurons; VIP cells also indirectly excite these pyramidal neurons via parallel disinhibition.
This study found that VIP-/- mice show a bilateral tactile hypersensitivity after spared nerve injury and a bilateral increase in neuronal activation upon innocuous tactile paw stimulation.
Several studies have shown that VIP(+) cells are sensitive to neuromodulation and increase their firing during locomotion, whisking, and pupil dilation and are involved in spatially specific top-down modulation, reminiscent of the effects of top-down attention, and also that attention enhances spatial resolution. Our findings provide a bridge between these studies by establishing the inhibitory circuitry that regulates th
majority of GnRH neurons in male and female mice express functional VIP receptors; effects of VIP on GnRH neurons do not alter across the estrous cycle
Backup mechanisms maintain PACAP (show ADCYAP1 Antibodies)/VIP-induced arterial vasodilation in PACAP (show ADCYAP1 Antibodies)-deficient mice.
in cortical VIP neurons, experience-dependent gene transcription regulates visual acuity by activating the expression of IGF1 (show IGF1 Antibodies), thus promoting the inhibition of disinhibitory neurons and affecting inhibition onto cortical pyramidal neurons
Chronic loss of VIP in mice leads to a disruption of certain but not all immunological compartments.
The protein encoded by this gene belongs to the glucagon family. It stimulates myocardial contractility, causes vasodilation, increases glycogenolysis, lowers arterial blood pressure and relaxes the smooth muscle of trachea, stomach and gall bladder. Alternative splicing occurs at this locus and two transcript variants encoding distinct isoforms have been identified.
, vasoactive intestinal peptide
, histidine-isoleucine/vasoactive intestinal polypeptide
, vasoactive intestinal polypeptide type I
, vasoactive intestinal polypeptide
, vasoactive instestinal peptide
, PHI, peptide histidine isoleucine