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anti-Human RAB11B Antibodies:
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Human Polyclonal RAB11B Primary Antibody for WB - ABIN522685
Oehlke, Martin, Osterberg, Roussa: Rab11b and its effector Rip11 regulate the acidosis-induced traffic of V-ATPase in salivary ducts. in Journal of cellular physiology 2010
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Recurrent de novo mutations disturbing the GTP (show AK3 Antibodies)/GDP binding pocket of RAB11B cause intellectual disability and a distinctive brain phenotype.
High RAB11B expression is associated with pancreatic cancer.
Rab11A (show RAB11A Antibodies) and Rab11B differentially regulate intracellular trafficking of PAR1 (show MARK2 Antibodies) through distinct endosomal sorting mechanisms
we determined a crystal structure of the PKG (show PRKG1 Antibodies) II LZ-Rab11b complex. The PKG (show PRKG1 Antibodies) II LZ domain presents a mostly nonpolar surface onto which Rab11b docks, through van (show TNIP1 Antibodies) der (show GDF3 Antibodies) Waals interactions
Upon Rab11b depletion, FGFR4 (show FGFR4 Antibodies) is trapped in the pericentriolar recycling compartment.
the predominant mechanism of melanin transfer is keratinocyte-induced exocytosis, mediated by Rab11b through remodeling of the melanosome membrane, followed by subsequent endocytosis by keratinocytes
Data show that the cAMP/PKA/CREB (show CREB1 Antibodies) signaling pathway initiates acidosis-induced V-ATPase (show ATP6V1H Antibodies) trafficking in salivary ducts via regulation of Rab11b expression.
These findings reveal a novel role for Rab11b in limiting, rather than promoting, the plasma membrane expression of Cav1.2 (show CACNA1C Antibodies) L-type Ca2 (show CA2 Antibodies)+ channels.
These data introduce Rab11b as a crucial regulator and Rip11 (show RAB11FIP5 Antibodies) as mediator of acidosis-induced V-ATPase (show ATP6V1H Antibodies) traffic in duct cells of submandibular gland.
propose a model where Rab11B specifically transports vesicles derived from the Golgi to the immature Inner Membrane Complex of the growing daughter parasites.
these data show an interaction between the smallGTPase Rab11b and the BK channel (show KCNMA1 Antibodies), which suggests that BK is shuttledthrough a slow recycling endocytic pathway.
These data indicate that Rab11b, and to a lesser extent Rab11a (show RAB11A Antibodies), is involved in establishing the constitutive and cAMP-stimulated Na(+) transport in mpkCCD cells.
These data indicate that Rab11b is necessary for the trafficking of cGK-II (show PRKG2 Antibodies) and that the cGMP/cGK-II (show PRKG2 Antibodies) signaling pathway is closely related to Rab11b recycling pathway.
Data show that in huntingtin-null embryonic stem cells, the levels of Rab11 on membranes and nucleotide exchange activity on Rab11 are significantly reduced compared with normal embryonic stem cells.
Study shows that both Rab11 (show RAB11A Antibodies) and its effector Rip11 (show RAB11FIP5 Antibodies) participate in insulin (show INS Antibodies) granule exocytosis and that Rip11 (show RAB11FIP5 Antibodies), as a substrate of PKA, regulates the potentiation of exocytosis by cAMP in pancreatic beta-cells.
The Ras superfamily of small GTP-binding proteins, which includes the Ras (see MIM 190020), Ral (see MIM 179550), Rho (see MIM 165390), Rap (see MIM 179520), and Rab (see MIM 179508) families, is involved in controlling a diverse set of essential cellular functions. The Rab family, including RAB11B, appears to play a critical role in regulating exocytotic and endocytotic pathways (summary by Zhu et al., 1994
ras-related protein Rab-11B
, GTP-binding protein YPT3
, RAB11B, member of RAS oncogene family