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Human HLAG ELISA Kit for Sandwich ELISA - ABIN414849
Wu, Wu, Luo, Tang, Yang, Xie, Liu, Zhou, Guan, Yuan: Lack of association between HLA-G 14-bp polymorphism and systemic lupus erythematosus in a Han Chinese population. in Lupus 2009
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Serum levels of soluble HLA-G were similar in CMV- and CMV+ subjects but levels in culture supernatants were significantly higher in cells from CMV+ than from CMV- subjects stimulated with CMV antigens. The HLA-G ligand KIR2DL4 (show KIR2DL4 ELISA Kits) was mainly expressed on NK cells and CD56 (show NCAM1 ELISA Kits)+ T cells with no differences between CMV+ and CMV- subjects.
our study is the first to suggest that HLA-G expression may influence the clinical outcome of chronic myeloid leukemia (show BCL11A ELISA Kits)
SV40 immune-inhibitory direct effect and the presence of high levels of the immune-inhibitory HLA-G molecules could co-operate in impairing B lymphocyte (show AKAP17A ELISA Kits) activation towards SV40 specific peptides.
HLA-G 3'UTR (show UTS2R ELISA Kits) polymorphisms associated with a greater magnitude of HLA-G production were associated with differentiated thyroid tumours and with variables implicated in poor prognosis.
The inhibitory properties of bronchial endothelial cellss are dysregulated in stable lung transplant recipients via TGF-beta (show TGFB1 ELISA Kits), IL-10 (show IL10 ELISA Kits) and HLA-G signaling pathway.
HLA-G expression was upregulated in chondrocyte-differentiated mesenchymal stem cells under hypoxia context and could be boosted in allogenic settings.
this review provides new insights into the mechanisms controlling the expression and function of HLA-G at the maternal-fetal interface, and discuss their relevance for fetal tolerance
we propose a massively parallel sequencing (NGS) with a bioinformatics strategy to evaluate the entire HLA-G regulatory and coding segments
3'UTR (show UTS2R ELISA Kits) typing may be a predictor of the genetic predisposition of an individual to express different levels of HLA-G.
The results of this study suggest that HLA-G expression may serve as a potential biomarker for predicting aggressive tumor grades of gliomas and for histological subtype of low-grade gliomas.
HLA-G belongs to the HLA class I heavy chain paralogues. This class I molecule is a heterodimer consisting of a heavy chain and a light chain (beta-2 microglobulin). The heavy chain is anchored in the membrane. HLA-G is expressed on fetal derived placental cells. The heavy chain is approximately 45 kDa and its gene contains 8 exons. Exon one encodes the leader peptide, exons 2 and 3 encode the alpha1 and alpha2 domain, which both bind the peptide, exon 4 encodes the alpha3 domain, exon 5 encodes the transmembrane region, and exon 6 encodes the cytoplasmic tail.
HLA G antigen
, HLA class I histocompatibility antigen, alpha chain G
, HLA class I molecule
, HLA-G histocompatibility antigen, class I, G
, MHC class I antigen G
, b2 microglobulin
, B-F minor
, MHC class I antigen alpha chain
, MHC class I glycoprotein
, major histocompatibility complex, class I, G